BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

207 related articles for article (PubMed ID: 10086344)

  • 1. Mutations of c-kit JM domain are found in a minority of human gastrointestinal stromal tumors.
    Moskaluk CA; Tian Q; Marshall CR; Rumpel CA; Franquemont DW; Frierson HF
    Oncogene; 1999 Mar; 18(10):1897-902. PubMed ID: 10086344
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Gastrointestinal stromal tumors with internal tandem duplications in 3' end of KIT juxtamembrane domain occur predominantly in stomach and generally seem to have a favorable course.
    Lasota J; Dansonka-Mieszkowska A; Stachura T; Schneider-Stock R; Kallajoki M; Steigen SE; Sarlomo-Rikala M; Boltze C; Kordek R; Roessner A; Stachura J; Miettinen M
    Mod Pathol; 2003 Dec; 16(12):1257-64. PubMed ID: 14681327
    [TBL] [Abstract][Full Text] [Related]  

  • 3. c-kit mutations in gastrointestinal stromal tumors occur preferentially in the spindle rather than in the epithelioid cell variant.
    Wardelmann E; Neidt I; Bierhoff E; Speidel N; Manegold C; Fischer HP; Pfeifer U; Pietsch T
    Mod Pathol; 2002 Feb; 15(2):125-36. PubMed ID: 11850541
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Effect of c-kit mutation on prognosis of gastrointestinal stromal tumors.
    Taniguchi M; Nishida T; Hirota S; Isozaki K; Ito T; Nomura T; Matsuda H; Kitamura Y
    Cancer Res; 1999 Sep; 59(17):4297-300. PubMed ID: 10485475
    [TBL] [Abstract][Full Text] [Related]  

  • 5. [Analysis of c-kit gene mutations in gastrointestinal stromal tumors].
    Hou YY; Sun MH; Tan YS; Wei YK; Lu XY; Zhang TM; Du X; Wang J; Zhu XZ
    Zhonghua Zhong Liu Za Zhi; 2004 Feb; 26(2):89-92. PubMed ID: 15059325
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Gastrointestinal stromal tumors (GISTs): definition, occurrence, pathology, differential diagnosis and molecular genetics.
    Miettinen M; Lasota J
    Pol J Pathol; 2003; 54(1):3-24. PubMed ID: 12817876
    [TBL] [Abstract][Full Text] [Related]  

  • 7. KIT activation is a ubiquitous feature of gastrointestinal stromal tumors.
    Rubin BP; Singer S; Tsao C; Duensing A; Lux ML; Ruiz R; Hibbard MK; Chen CJ; Xiao S; Tuveson DA; Demetri GD; Fletcher CD; Fletcher JA
    Cancer Res; 2001 Nov; 61(22):8118-21. PubMed ID: 11719439
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Gastrointestinal stromal tumours.
    Miettinen M; Sarlomo-Rikala M; Lasota J
    Ann Chir Gynaecol; 1998; 87(4):278-81. PubMed ID: 9891765
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Gastrointestinal stromal tumours (GISTs) negative for KIT (CD117 antigen) immunoreactivity.
    Debiec-Rychter M; Wasag B; Stul M; De Wever I; Van Oosterom A; Hagemeijer A; Sciot R
    J Pathol; 2004 Apr; 202(4):430-8. PubMed ID: 15095270
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Gain-of-function mutation at the extracellular domain of KIT in gastrointestinal stromal tumours.
    Hirota S; Nishida T; Isozaki K; Taniguchi M; Nakamura J; Okazaki T; Kitamura Y
    J Pathol; 2001 Apr; 193(4):505-10. PubMed ID: 11276010
    [TBL] [Abstract][Full Text] [Related]  

  • 11. KIT exon 11 deletion-inversions represent complex mutations in gastrointestinal stromal tumors.
    Lasota J; Miettinen M
    Cancer Genet Cytogenet; 2007 May; 175(1):69-72. PubMed ID: 17498562
    [TBL] [Abstract][Full Text] [Related]  

  • 12. A missense mutation in KIT kinase domain 1 correlates with imatinib resistance in gastrointestinal stromal tumors.
    Chen LL; Trent JC; Wu EF; Fuller GN; Ramdas L; Zhang W; Raymond AK; Prieto VG; Oyedeji CO; Hunt KK; Pollock RE; Feig BW; Hayes KJ; Choi H; Macapinlac HA; Hittelman W; Velasco MA; Patel S; Burgess MA; Benjamin RS; Frazier ML
    Cancer Res; 2004 Sep; 64(17):5913-9. PubMed ID: 15342366
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Gene expression in gastrointestinal stromal tumors is distinguished by KIT genotype and anatomic site.
    Antonescu CR; Viale A; Sarran L; Tschernyavsky SJ; Gonen M; Segal NH; Maki RG; Socci ND; DeMatteo RP; Besmer P
    Clin Cancer Res; 2004 May; 10(10):3282-90. PubMed ID: 15161681
    [TBL] [Abstract][Full Text] [Related]  

  • 14. [Therapeutic targets in gastrointestinal stromal tumors].
    Wardelmann E; Schildhaus HU; Merkelbach-Bruse S; Büttner R
    Verh Dtsch Ges Pathol; 2006; 90():73-9. PubMed ID: 17867582
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Gastrointestinal stromal tumors with KIT mutations exhibit a remarkably homogeneous gene expression profile.
    Allander SV; Nupponen NN; Ringnér M; Hostetter G; Maher GW; Goldberger N; Chen Y; Carpten J; Elkahloun AG; Meltzer PS
    Cancer Res; 2001 Dec; 61(24):8624-8. PubMed ID: 11751374
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Clinicopathological features of gastric stromal tumors.
    Nishida T; Nakamura J; Taniguchi M; Hirota S; Ito T; Kitamura Y; Matsuda H
    J Exp Clin Cancer Res; 2000 Dec; 19(4):417-25. PubMed ID: 11277317
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Detection of c-kit-activating mutations in gastrointestinal stromal tumors by high-resolution amplicon melting analysis.
    Willmore C; Holden JA; Zhou L; Tripp S; Wittwer CT; Layfield LJ
    Am J Clin Pathol; 2004 Aug; 122(2):206-16. PubMed ID: 15323137
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Mutations in exon 11 of the c-kit gene in a myogenic tumor and a neurogenic tumor as well as in gastrointestinal stromal tumors. Utility of c-kit mutation as a prognostic biomarker for gastrointestinal mesenchymal tumor.
    Yasuoka R; Sakakura C; Shimomura K; Fujita Y; Nakanishi M; Aragane H; Hagiwara A; Bamba M; Abe T; Yamagishi H
    Dig Surg; 2003; 20(3):183-91. PubMed ID: 12759497
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Absence of c-kit gene mutations in gastrointestinal stromal tumours from neurofibromatosis type 1 patients.
    Kinoshita K; Hirota S; Isozaki K; Ohashi A; Nishida T; Kitamura Y; Shinomura Y; Matsuzawa Y
    J Pathol; 2004 Jan; 202(1):80-5. PubMed ID: 14694524
    [TBL] [Abstract][Full Text] [Related]  

  • 20. [Gastrointestinal stromal tumors carrying PDGFRalpha mutations occur preferentially in the stomach and exhibit an epithelioid or mixed phenotype].
    Wardelmann E; Pauls K; Merkelbach-Bruse S; Hrychyk A; Losen I; Hohenberger P; Büttner R; Pietsch T
    Verh Dtsch Ges Pathol; 2004; 88():174-83. PubMed ID: 16892550
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.