413 related articles for article (PubMed ID: 10196277)
1. Antiretroviral cytolytic T-lymphocyte nonresponsiveness: FasL/Fas-mediated inhibition of CD4(+) and CD8(+) antiviral T cells by viral antigen-positive veto cells.
Rich RF; Green WR
J Virol; 1999 May; 73(5):3826-34. PubMed ID: 10196277
[TBL] [Abstract][Full Text] [Related]
2. Characterization of the Fas ligand/Fas-dependent apoptosis of antiretroviral, class I MHC tetramer-defined, CD8+ CTL by in vivo retrovirus-infected cells.
Rich RF; Green WR
J Immunol; 2002 Mar; 168(6):2751-8. PubMed ID: 11884442
[TBL] [Abstract][Full Text] [Related]
3. AKR.H-2b lymphocytes inhibit the secondary in vitro cytotoxic T-lymphocyte response of primed responder cells to AKR/Gross murine leukemia virus-induced tumor cell stimulation.
Rich RF; Green WR
J Virol; 1996 Jan; 70(1):402-14. PubMed ID: 8523554
[TBL] [Abstract][Full Text] [Related]
4. Spontaneous in vivo retrovirus-infected T and B cells, but not dendritic cells, mediate antigen-specific Fas ligand/Fas-dependent apoptosis of anti-retroviral CTL.
Rich RF; Cook WJ; Green WR
Virology; 2006 Mar; 346(2):287-300. PubMed ID: 16337984
[TBL] [Abstract][Full Text] [Related]
5. Apoptosis of epitope-specific antiretroviral cytotoxic T lymphocytes via Fas ligand-Fas interactions.
Rich RF; Green WR
Viral Immunol; 2006; 19(3):424-33. PubMed ID: 16987061
[TBL] [Abstract][Full Text] [Related]
6. A shift in the requirement for CD4+ T cells in the generation of AKR/Gross MuLV-specific CTL in AKR.H-2b:Fv-1b mice occurs prior to the onset of age-dependent CTL nonresponsiveness.
Smith PM; Rich RF; Green WR
Cell Immunol; 1997 Feb; 175(2):189-98. PubMed ID: 9023425
[TBL] [Abstract][Full Text] [Related]
7. Impaired generation of anti-AKR/Gross murine leukemia virus cytotoxic T lymphocytes in mice experimentally infected with MuLV.
Coppola MA; Green WR; Rich RF
Viral Immunol; 1996; 9(2):107-19. PubMed ID: 8822627
[TBL] [Abstract][Full Text] [Related]
8. Nonresponsiveness of AKR.H-2b congenic mice for anti-AKR/Gross MuLV CTL responses: involvement of inhibitory cells as defined by adoptive transfer experiments.
Rich RF; Green WR
Cell Immunol; 1995 Jan; 160(1):139-51. PubMed ID: 7842479
[TBL] [Abstract][Full Text] [Related]
9. Cell surface expression of cytotoxic T lymphocyte-defined, AKR/Gross leukemia virus-associated tumor antigens by normal AKR.H-2b splenic B cells.
Green WR
J Immunol; 1983 Dec; 131(6):3078-84. PubMed ID: 6605998
[TBL] [Abstract][Full Text] [Related]
10. Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. II. Negative control by the Fv-1 locus in AKR mice of responder H-2b haplotype.
Green WR
J Immunol; 1984 May; 132(5):2665-71. PubMed ID: 6425410
[TBL] [Abstract][Full Text] [Related]
11. An immunodominant Kb-restricted peptide from the p15E transmembrane protein of endogenous ecotropic murine leukemia virus (MuLV) AKR623 that restores susceptibility of a tumor line to anti-AKR/Gross MuLV cytotoxic T lymphocytes.
White HD; Roeder DA; Green WR
J Virol; 1994 Feb; 68(2):897-904. PubMed ID: 8289392
[TBL] [Abstract][Full Text] [Related]
12. Virally infected hepatocytes are resistant to perforin-dependent CTL effector mechanisms.
Kafrouni MI; Brown GR; Thiele DL
J Immunol; 2001 Aug; 167(3):1566-74. PubMed ID: 11466378
[TBL] [Abstract][Full Text] [Related]
13. Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. I. H-2-encoded dominant gene control.
Green WR
J Immunol; 1984 May; 132(5):2658-64. PubMed ID: 6425409
[TBL] [Abstract][Full Text] [Related]
14. Self-veto mechanism of CD8+ cytotoxic effector T cells. Peptide-induced paralysis affects the peptide-MHC-recognizing cytotoxic T lymphocytes and is independent of Fas/Fas ligand interactions.
Bergenthal A; Hofmann M; Heeg K
Eur J Immunol; 1998 Jun; 28(6):1911-22. PubMed ID: 9645373
[TBL] [Abstract][Full Text] [Related]
15. Expression of CTL-defined, AKR/Gross retrovirus-associated tumor antigens by normal spleen cells: control by Fv-1, H-2, and proviral genes and effect on antiviral CTL generation.
Green WR
J Immunol; 1986 Jan; 136(1):308-12. PubMed ID: 2999246
[TBL] [Abstract][Full Text] [Related]
16. Delayed kinetics of tumor necrosis factor-mediated bystander lysis by peptide-specific CD8+ cytotoxic T lymphocytes.
Smyth MJ; Sedgwick JD
Eur J Immunol; 1998 Dec; 28(12):4162-9. PubMed ID: 9862352
[TBL] [Abstract][Full Text] [Related]
17. Induction of anti-AKR/gross virus cytolytic T lymphocytes in AKR.H-2b:Fv-1b congenic mice: age-dependent conversion to a nonresponder phenotype.
Green WR
J Immunol; 1987 Mar; 138(5):1602-6. PubMed ID: 3027182
[TBL] [Abstract][Full Text] [Related]
18. A single amino acid variation within an immunodominant AKR/Gross MuLV cytotoxic T-lymphocyte epitope leads to a loss in immunogenicity.
Kim V; Green WR
Viral Immunol; 1998; 11(4):197-213. PubMed ID: 10189187
[TBL] [Abstract][Full Text] [Related]
19. CD4-CD8+ T lymphocytes mediate AKR/gross murine leukemia virus nonresponsiveness in moderately aged AKR.H-2b:Fv-1b mice.
Rich RF; Fujii T; Green WR
J Immunol; 1992 May; 148(9):2961-7. PubMed ID: 1349325
[TBL] [Abstract][Full Text] [Related]
20. Major and minor Kb-restricted epitopes encoded by the endogenous ecotropic murine leukemia virus AKR623 that are recognized by anti-AKR/Gross MuLV CTL.
White HD; Roeder DA; Lam T; Green WR
Viral Immunol; 1994; 7(2):51-9. PubMed ID: 7848510
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]