188 related articles for article (PubMed ID: 10366347)
1. GAD, a single autoantigen for diabetes.
von Boehmer H; Sarukhan A
Science; 1999 May; 284(5417):1135, 1137. PubMed ID: 10366347
[No Abstract] [Full Text] [Related]
2. Control of autoimmune diabetes in NOD mice by GAD expression or suppression in beta cells.
Yoon JW; Yoon CS; Lim HW; Huang QQ; Kang Y; Pyun KH; Hirasawa K; Sherwin RS; Jun HS
Science; 1999 May; 284(5417):1183-7. PubMed ID: 10325232
[TBL] [Abstract][Full Text] [Related]
3. Does GAD have a unique role in triggering IDDM?
Baekkeskov S; Kanaani J; Jaume JC; Kash S
J Autoimmun; 2000 Nov; 15(3):279-86. PubMed ID: 11040068
[No Abstract] [Full Text] [Related]
4. Autoimmune diabetes: is GAD the culprit?
Lopez-Liuchi JV
Eur J Endocrinol; 1999 Nov; 141(5):458-9. PubMed ID: 10576760
[No Abstract] [Full Text] [Related]
5. It's insulin.
Wegmann DR; Eisenbarth GS
J Autoimmun; 2000 Nov; 15(3):286-91. PubMed ID: 11040069
[No Abstract] [Full Text] [Related]
6. Active tolerance induction and prevention of autoimmune diabetes by immunogene therapy using recombinant adenoassociated virus expressing glutamic acid decarboxylase 65 peptide GAD(500-585).
Han G; Li Y; Wang J; Wang R; Chen G; Song L; Xu R; Yu M; Wu X; Qian J; Shen B
J Immunol; 2005 Apr; 174(8):4516-24. PubMed ID: 15814672
[TBL] [Abstract][Full Text] [Related]
7. T-lymphocyte lines specific for glutamic acid decarboxylase (GAD) the 64K beta-cell antigen of IDDM.
Diaz JL; Ways J; Hammonds P
Diabetes; 1992 Jan; 41(1):118-21. PubMed ID: 1727731
[TBL] [Abstract][Full Text] [Related]
8. Normal incidence of diabetes in NOD mice tolerant to glutamic acid decarboxylase.
Jaeckel E; Klein L; Martin-Orozco N; von Boehmer H
J Exp Med; 2003 Jun; 197(12):1635-44. PubMed ID: 12796471
[TBL] [Abstract][Full Text] [Related]
9. Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice.
Cooke A; Mandel TE
J Endocrinol Invest; 1994; 17(7):586-93. PubMed ID: 7829835
[No Abstract] [Full Text] [Related]
10. Differential protection in two transgenic lines of NOD/Lt mice hyperexpressing the autoantigen GAD65 in pancreatic beta-cells.
Bridgett M; Cetkovic-Cvrlje M; O'Rourke R; Shi Y; Narayanswami S; Lambert J; Ramiya V; Baekkeskov S; Leiter EH
Diabetes; 1998 Dec; 47(12):1848-56. PubMed ID: 9836515
[TBL] [Abstract][Full Text] [Related]
11. The glutamate decarboxylase and 38KD autoantigens in type 1 diabetes: aspects of structure and epitope recognition.
Baekkeskov S; Aanstoot HJ; Fu Q; Jaffe M; Kim J; Quan J; Richter W; Shi Y
Autoimmunity; 1993; 15 Suppl():24-6. PubMed ID: 7692998
[No Abstract] [Full Text] [Related]
12. GAD-about BDC2.5: peptides that stimulate BDC2.5 T cells and inhibit IDDM.
Wilson DB
J Autoimmun; 2003 May; 20(3):199-201. PubMed ID: 12753803
[No Abstract] [Full Text] [Related]
13. Role of glutamic acid decarboxylase in the pathogenesis of type 1 diabetes.
Jun HS; Khil LY; Yoon JW
Cell Mol Life Sci; 2002 Nov; 59(11):1892-901. PubMed ID: 12530520
[TBL] [Abstract][Full Text] [Related]
14. Conversations with GAD.
Nepom GT
J Autoimmun; 2003 May; 20(3):195-8. PubMed ID: 12753802
[No Abstract] [Full Text] [Related]
15. Hyperexpression of GAD in the islets may be relevant but is it sufficient to induce autoimmune insulitis?
Pozzilli P; Leslie RD
Diabetologia; 1997 Mar; 40(3):357-61. PubMed ID: 9084978
[No Abstract] [Full Text] [Related]
16. Widespread expression of an autoantigen-GAD65 transgene does not tolerize non-obese diabetic mice and can exacerbate disease.
Geng L; Solimena M; Flavell RA; Sherwin RS; Hayday AC
Proc Natl Acad Sci U S A; 1998 Aug; 95(17):10055-60. PubMed ID: 9707599
[TBL] [Abstract][Full Text] [Related]
17. Diabetes. Spotlight on a neuronal enzyme.
Solimena M; De Camilli P
Nature; 1993 Nov; 366(6450):15-7. PubMed ID: 8232529
[No Abstract] [Full Text] [Related]
18. Characterization of immunodominant peptide determinants of IDDM-associated autoantigens in the NOD mouse.
Zechel MA; Chaturvedi P; Singh B
Res Immunol; 1997 Jun; 148(5):338-48. PubMed ID: 9352599
[No Abstract] [Full Text] [Related]
19. Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice.
Tisch R; Yang XD; Singer SM; Liblau RS; Fugger L; McDevitt HO
Nature; 1993 Nov; 366(6450):72-5. PubMed ID: 8232539
[TBL] [Abstract][Full Text] [Related]
20. How important is GAD in the etiology of spontaneous disease in human and murine type 1 diabetes?
Nepom G; Quinn A; Sercarz E; Wilson DB
J Autoimmun; 2003 May; 20(3):193-4. PubMed ID: 12753801
[No Abstract] [Full Text] [Related]
[Next] [New Search]