129 related articles for article (PubMed ID: 10434310)
1. CAG-polyglutamine-repeat mutations: independence from gene context.
Ordway JM; Cearley JA; Detloff PJ
Philos Trans R Soc Lond B Biol Sci; 1999 Jun; 354(1386):1083-8. PubMed ID: 10434310
[TBL] [Abstract][Full Text] [Related]
2. Hprt(CAG)146 mice: age of onset of behavioral abnormalities, time course of neuronal intranuclear inclusion accumulation, neurotransmitter marker alterations, mitochondrial function markers, and susceptibility to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine.
Tallaksen-Greene SJ; Ordway JM; Crouse AB; Jackson WS; Detloff PJ; Albin RL
J Comp Neurol; 2003 Oct; 465(2):205-19. PubMed ID: 12949782
[TBL] [Abstract][Full Text] [Related]
3. Ectopically expressed CAG repeats cause intranuclear inclusions and a progressive late onset neurological phenotype in the mouse.
Ordway JM; Tallaksen-Greene S; Gutekunst CA; Bernstein EM; Cearley JA; Wiener HW; Dure LS; Lindsey R; Hersch SM; Jope RS; Albin RL; Detloff PJ
Cell; 1997 Dec; 91(6):753-63. PubMed ID: 9413985
[TBL] [Abstract][Full Text] [Related]
4. Familial frontotemporal dementia with neuronal intranuclear inclusions is not a polyglutamine expansion disease.
Mackenzie IR; Butland SL; Devon RS; Dwosh E; Feldman H; Lindholm C; Neal SJ; Ouellette BF; Leavitt BR
BMC Neurol; 2006 Aug; 6():32. PubMed ID: 16945149
[TBL] [Abstract][Full Text] [Related]
5. [Molecular biology of polyglutamine diseases].
Owecki M; Kozubski W
Postepy Hig Med Dosw; 2002; 56(6):779-88. PubMed ID: 12661407
[TBL] [Abstract][Full Text] [Related]
6. Embryonic stem cells expressing expanded CAG repeats undergo aberrant neuronal differentiation and have persistent Oct-4 and REST/NRSF expression.
Lorincz MT; Detloff PJ; Albin RL; O'Shea KS
Mol Cell Neurosci; 2004 May; 26(1):135-43. PubMed ID: 15121185
[TBL] [Abstract][Full Text] [Related]
7. CAG repeat disorder models and human neuropathology: similarities and differences.
Yamada M; Sato T; Tsuji S; Takahashi H
Acta Neuropathol; 2008 Jan; 115(1):71-86. PubMed ID: 17786457
[TBL] [Abstract][Full Text] [Related]
8. DRPLA transgenic mouse substrains carrying single copy of full-length mutant human DRPLA gene with variable sizes of expanded CAG repeats exhibit CAG repeat length- and age-dependent changes in behavioral abnormalities and gene expression profiles.
Suzuki K; Zhou J; Sato T; Takao K; Miyagawa T; Oyake M; Yamada M; Takahashi H; Takahashi Y; Goto J; Tsuji S
Neurobiol Dis; 2012 May; 46(2):336-50. PubMed ID: 22342974
[TBL] [Abstract][Full Text] [Related]
9. Trinucleotide repeat expansion in neurological disease.
La Spada AR; Paulson HL; Fischbeck KH
Ann Neurol; 1994 Dec; 36(6):814-22. PubMed ID: 7998766
[TBL] [Abstract][Full Text] [Related]
10. cAMP-response element-binding protein and heat-shock protein 70 additively suppress polyglutamine-mediated toxicity in Drosophila.
Iijima-Ando K; Wu P; Drier EA; Iijima K; Yin JC
Proc Natl Acad Sci U S A; 2005 Jul; 102(29):10261-6. PubMed ID: 16009936
[TBL] [Abstract][Full Text] [Related]
11. Allele-selective suppression of mutant genes in polyglutamine diseases.
Liu CR; Cheng TH
J Neurogenet; 2015; 29(2-3):41-9. PubMed ID: 26174158
[TBL] [Abstract][Full Text] [Related]
12. Selectable system for monitoring the instability of CTG/CAG triplet repeats in mammalian cells.
Gorbunova V; Seluanov A; Dion V; Sandor Z; Meservy JL; Wilson JH
Mol Cell Biol; 2003 Jul; 23(13):4485-93. PubMed ID: 12808091
[TBL] [Abstract][Full Text] [Related]
13. Comparative toxicity of polyglutamine, polyalanine and polyleucine tracts in Drosophila models of expanded repeat disease.
van Eyk CL; McLeod CJ; O'Keefe LV; Richards RI
Hum Mol Genet; 2012 Feb; 21(3):536-47. PubMed ID: 22021427
[TBL] [Abstract][Full Text] [Related]
14. The pathogenic agent in Drosophila models of 'polyglutamine' diseases.
McLeod CJ; O'Keefe LV; Richards RI
Hum Mol Genet; 2005 Apr; 14(8):1041-8. PubMed ID: 15757976
[TBL] [Abstract][Full Text] [Related]
15. Identification of six CAG repeat domains into the human chromosomic region 12q24.1.
Aguiar J
Biochem Biophys Res Commun; 2000 Jun; 272(2):400-2. PubMed ID: 10833426
[TBL] [Abstract][Full Text] [Related]
16. Effects of CAG repeat length, HTT protein length and protein context on cerebral metabolism measured using magnetic resonance spectroscopy in transgenic mouse models of Huntington's disease.
Jenkins BG; Andreassen OA; Dedeoglu A; Leavitt B; Hayden M; Borchelt D; Ross CA; Ferrante RJ; Beal MF
J Neurochem; 2005 Oct; 95(2):553-62. PubMed ID: 16135087
[TBL] [Abstract][Full Text] [Related]
17. Roles of trinucleotide-repeat RNA in neurological disease and degeneration.
Li LB; Bonini NM
Trends Neurosci; 2010 Jun; 33(6):292-8. PubMed ID: 20398949
[TBL] [Abstract][Full Text] [Related]
18. Bidirectional expression of CUG and CAG expansion transcripts and intranuclear polyglutamine inclusions in spinocerebellar ataxia type 8.
Moseley ML; Zu T; Ikeda Y; Gao W; Mosemiller AK; Daughters RS; Chen G; Weatherspoon MR; Clark HB; Ebner TJ; Day JW; Ranum LP
Nat Genet; 2006 Jul; 38(7):758-69. PubMed ID: 16804541
[TBL] [Abstract][Full Text] [Related]
19. A SCA7 CAG/CTG repeat expansion is stable in Drosophila melanogaster despite modulation of genomic context and gene dosage.
Jackson SM; Whitworth AJ; Greene JC; Libby RT; Baccam SL; Pallanck LJ; La Spada AR
Gene; 2005 Feb; 347(1):35-41. PubMed ID: 15715978
[TBL] [Abstract][Full Text] [Related]
20. A role for selection in regulating the evolutionary emergence of disease-causing and other coding CAG repeats in humans and mice.
Hancock JM; Worthey EA; Santibáñez-Koref MF
Mol Biol Evol; 2001 Jun; 18(6):1014-23. PubMed ID: 11371590
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
