These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

101 related articles for article (PubMed ID: 10493251)

  • 1. The impact of misclassification in case-control studies of gene-environment interactions.
    Rothman N; Garcia-Closas M; Stewart WT; Lubin J
    IARC Sci Publ; 1999; (148):89-96. PubMed ID: 10493251
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Misclassification in case-control studies of gene-environment interactions: assessment of bias and sample size.
    Garcia-Closas M; Rothman N; Lubin J
    Cancer Epidemiol Biomarkers Prev; 1999 Dec; 8(12):1043-50. PubMed ID: 10613335
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Computing differential sample size for case-control studies of gene-environment interaction.
    Efird JT; Hong MK
    Ethn Dis; 2008; 18(2 Suppl 2):S2-25-9. PubMed ID: 18646316
    [TBL] [Abstract][Full Text] [Related]  

  • 4. On the use of population attributable fraction to determine sample size for case-control studies of gene-environment interaction.
    Yang Q; Khoury MJ; Friedman JM; Flanders WD
    Epidemiology; 2003 Mar; 14(2):161-7. PubMed ID: 12606881
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Detection of genotype-environment interaction in case-control studies of birth defects: how big a sample size?
    Khoury MJ; Beaty TH; Hwang SJ
    Teratology; 1995 May; 51(5):336-43. PubMed ID: 7482355
    [TBL] [Abstract][Full Text] [Related]  

  • 6. The effects of misclassification in studies of gene-environment interactions.
    Cheng KF; Lin WJ
    Hum Hered; 2009; 67(2):77-87. PubMed ID: 19077425
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Study design options in evaluating gene-environment interactions: practical considerations for a planned case-control study of pediatric leukemia.
    Goodman M; Dana Flanders W
    Pediatr Blood Cancer; 2007 Apr; 48(4):375-9. PubMed ID: 16874764
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Tests for gene-environment interaction from case-control data: a novel study of type I error, power and designs.
    Mukherjee B; Ahn J; Gruber SB; Rennert G; Moreno V; Chatterjee N
    Genet Epidemiol; 2008 Nov; 32(7):615-26. PubMed ID: 18473390
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Re: "flexible matching strategies to increase power and efficiency to detect and estimate gene-environment interactions in case-control studies".
    Schill W; Wild P
    Am J Epidemiol; 2004 Jun; 159(11):1107-8; author reply 1108. PubMed ID: 15155296
    [No Abstract]   [Full Text] [Related]  

  • 10. [Sample size requirements for association studies on gene-gene interaction in case-control study].
    Wang ZP; Li HQ
    Zhonghua Liu Xing Bing Xue Za Zhi; 2004 Jul; 25(7):623-6. PubMed ID: 15308047
    [TBL] [Abstract][Full Text] [Related]  

  • 11. [The investigation of gene-environment interaction using case-control comparisons].
    Kellen E; Zeegers MP; Vandenbroucke JP; Buntinx F
    Ned Tijdschr Geneeskd; 2005 Apr; 149(18):977-82. PubMed ID: 15903038
    [TBL] [Abstract][Full Text] [Related]  

  • 12. When a case is not a case: effects of phenotype misclassification on power and sample size requirements for the transmission disequilibrium test with affected child trios.
    Buyske S; Yang G; Matise TC; Gordon D
    Hum Hered; 2009; 67(4):287-92. PubMed ID: 19172087
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Accounting for error due to misclassification of exposures in case-control studies of gene-environment interaction.
    Zhang L; Mukherjee B; Ghosh M; Gruber S; Moreno V
    Stat Med; 2008 Jul; 27(15):2756-83. PubMed ID: 17879261
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Exploiting gene-environment independence in family-based case-control studies: increased power for detecting associations, interactions and joint effects.
    Chatterjee N; Kalaylioglu Z; Carroll RJ
    Genet Epidemiol; 2005 Feb; 28(2):138-56. PubMed ID: 15593088
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Design options for case-control studies of gene-environment interactions.
    Cheng C
    Pediatr Blood Cancer; 2007 Apr; 48(4):373-4. PubMed ID: 16917907
    [No Abstract]   [Full Text] [Related]  

  • 16. The impact of genotype misclassification errors on the power to detect a gene-environment interaction using cox proportional hazards modeling.
    Tung L; Gordon D; Finch SJ
    Hum Hered; 2007; 63(2):101-10. PubMed ID: 17283439
    [TBL] [Abstract][Full Text] [Related]  

  • 17. The case-combined-control design was efficient in detecting gene-environment interactions.
    Andrieu N; Goldstein AM
    J Clin Epidemiol; 2004 Jul; 57(7):662-71. PubMed ID: 15358394
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Investigating gene environment interaction in complex diseases: increasing power by selective sampling for environmental exposure.
    Boks MP; Schipper M; Schubart CD; Sommer IE; Kahn RS; Ophoff RA
    Int J Epidemiol; 2007 Dec; 36(6):1363-9. PubMed ID: 17971387
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Gene-environment interactions in human diseases.
    Hunter DJ
    Nat Rev Genet; 2005 Apr; 6(4):287-98. PubMed ID: 15803198
    [TBL] [Abstract][Full Text] [Related]  

  • 20. A maximum likelihood method for studying gene-environment interactions under conditional independence of genotype and exposure.
    Cheng KF
    Stat Med; 2006 Sep; 25(18):3093-109. PubMed ID: 16463309
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 6.