BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

84 related articles for article (PubMed ID: 10547001)

  • 1. Evidence from studies with N-ethyl-maleimide and 12-O-tetradecanoylphorbol-13-acetate that AP-1 and CREB are involved in the glucocorticoid activation of TRH gene expression in hypothalamic cultures.
    Luo LG; Wang S; Su E; Jackson IM
    Brain Res; 1999 Sep; 841(1-2):189-92. PubMed ID: 10547001
    [TBL] [Abstract][Full Text] [Related]  

  • 2. A rapid interference between glucocorticoids and cAMP-activated signalling in hypothalamic neurones prevents binding of phosphorylated cAMP response element binding protein and glucocorticoid receptor at the CRE-Like and composite GRE sites of thyrotrophin-releasing hormone gene promoter.
    Díaz-Gallardo MY; Cote-Vélez A; Charli JL; Joseph-Bravo P
    J Neuroendocrinol; 2010 Apr; 22(4):282-93. PubMed ID: 20136691
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Glucocorticoids stimulate TRH and c-fos/c-jun gene co-expression in cultured hypothalamic neurons.
    Luo LG; Jackson IM
    Brain Res; 1998 Apr; 791(1-2):56-62. PubMed ID: 9593821
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Glucocorticoids curtail stimuli-induced CREB phosphorylation in TRH neurons through interaction of the glucocorticoid receptor with the catalytic subunit of protein kinase A.
    Sotelo-Rivera I; Cote-Vélez A; Uribe RM; Charli JL; Joseph-Bravo P
    Endocrine; 2017 Mar; 55(3):861-871. PubMed ID: 28063130
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Dexamethasone represses cAMP rapid upregulation of TRH gene transcription: identification of a composite glucocorticoid response element and a cAMP response element in TRH promoter.
    Cote-Vélez A; Pérez-Martínez L; Díaz-Gallardo MY; Pérez-Monter C; Carreón-Rodríguez A; Charli JL; Joseph-Bravo P
    J Mol Endocrinol; 2005 Feb; 34(1):177-97. PubMed ID: 15691887
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Dexamethasone rapidly regulates TRH mRNA levels in hypothalamic cell cultures: interaction with the cAMP pathway.
    Pérez-Martínez L; Carreón-Rodríguez A; González-Alzati ME; Morales C; Charli JL; Joseph-Bravo P
    Neuroendocrinology; 1998 Nov; 68(5):345-54. PubMed ID: 9822802
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Advantage of double labeled in situ hybridization for detecting the effects of glucocorticoids on the mRNAs of protooncogenes and neural peptides (TRH) in cultured hypothalamic neurons.
    Luo LG; Jackson IM
    Brain Res Brain Res Protoc; 1999 Jul; 4(2):201-8. PubMed ID: 10446415
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Inhibition of phorbol ester-induced monocytic differentiation by dexamethasone is associated with down-regulation of c-fos and c-jun (AP-1).
    Hass R; Brach M; Kharbanda S; Giese G; Traub P; Kufe D
    J Cell Physiol; 1991 Oct; 149(1):125-31. PubMed ID: 1939341
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Antisense oligomers of cfos and cjun block glucocorticoid stimulation of thyrotropin-releasing hormone (TRH) gene expression in cultured anterior pituitary cells.
    Luo LG; Jackson IM
    Peptides; 1998; 19(8):1295-302. PubMed ID: 9809642
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Family members CREB and CREM control thyrotropin-releasing hormone (TRH) expression in the hypothalamus.
    Chiappini F; Ramadoss P; Vella KR; Cunha LL; Ye FD; Stuart RC; Nillni EA; Hollenberg AN
    Mol Cell Endocrinol; 2013 Jan; 365(1):84-94. PubMed ID: 23000398
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Abnormal glucocorticoid receptor-activator protein 1 interaction in steroid-resistant asthma.
    Adcock IM; Lane SJ; Brown CR; Lee TH; Barnes PJ
    J Exp Med; 1995 Dec; 182(6):1951-8. PubMed ID: 7500041
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Phorbol ester or cAMP enhance thyrotropin-releasing hormone mRNA in primary cultures of hypothalamic cells.
    Uribe RM; Pérez-Martínez L; de Lourdes Covarrubias M; Gómez O; Covarrubias L; Charli JL; Joseph-Bravo P
    Neurosci Lett; 1995 Dec; 201(1):41-4. PubMed ID: 8830308
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Thyrotropin-releasing hormone and c-fos/c-jun genes are colocalized in rat anterior pituitary cells: stimulation of transcription by glucocorticoids.
    Luo L; Jackson IM
    Endocrinology; 1995 Jun; 136(6):2705-10. PubMed ID: 7750495
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Major role of 3',5'-cyclic adenosine monophosphate-dependent protein kinase A pathway in corticotropin-releasing factor gene expression in the rat hypothalamus in vivo.
    Itoi K; Horiba N; Tozawa F; Sakai Y; Sakai K; Abe K; Demura H; Suda T
    Endocrinology; 1996 Jun; 137(6):2389-96. PubMed ID: 8641191
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Lithium increases transcription factor binding to AP-1 and cyclic AMP-responsive element in cultured neurons and rat brain.
    Ozaki N; Chuang DM
    J Neurochem; 1997 Dec; 69(6):2336-44. PubMed ID: 9375664
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Synergistic activation of neurotensin/neuromedin N gene expression by c-Jun and glucocorticoids: novel effects of Fos family proteins.
    Harrison RJ; McNeil GP; Dobner PR
    Mol Endocrinol; 1995 Aug; 9(8):981-93. PubMed ID: 7476995
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Central administration of neuropeptide Y reduces alpha-melanocyte-stimulating hormone-induced cyclic adenosine 5'-monophosphate response element binding protein (CREB) phosphorylation in pro-thyrotropin-releasing hormone neurons and increases CREB phosphorylation in corticotropin-releasing hormone neurons in the hypothalamic paraventricular nucleus.
    Sarkar S; Lechan RM
    Endocrinology; 2003 Jan; 144(1):281-91. PubMed ID: 12488356
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Corticotrophin-releasing factor gene transcription is directly activated after deprivation of glucocorticoids in hypothalamic cells.
    Kageyama K; Akimoto K; Suda T
    J Neuroendocrinol; 2010 Sep; 22(9):971-8. PubMed ID: 20626567
    [TBL] [Abstract][Full Text] [Related]  

  • 19. ERK signaling pathway is involved in p15INK4b/p16INK4a expression and HepG2 growth inhibition triggered by TPA and Saikosaponin a.
    Wen-Sheng W
    Oncogene; 2003 Feb; 22(7):955-63. PubMed ID: 12592382
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Successive occupancy by immediate early transcriptional factors of the tyrosine hydroxylase gene TRE and CRE sites in PACAP-stimulated PC12 pheochromocytoma cells.
    Yukimasa N; Isobe K; Nagai H; Takuwa Y; Nakai T
    Neuropeptides; 1999 Dec; 33(6):475-82. PubMed ID: 10657527
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 5.