1470 related articles for article (PubMed ID: 10579212)
1. Prior D1 dopamine receptor stimulation is required to prime D2-mediated striatal Fos expression in 6-hydroxydopamine-lesioned rats.
Pollack AE; Yates TM
Neuroscience; 1999; 94(2):505-14. PubMed ID: 10579212
[TBL] [Abstract][Full Text] [Related]
2. Apomorphine priming alters the response of striatal outflow pathways to D2 agonist stimulation in 6-hydroxydopamine-lesioned rats.
Pollack AE; Turgeon SM; Fink JS
Neuroscience; 1997 Jul; 79(1):79-93. PubMed ID: 9178866
[TBL] [Abstract][Full Text] [Related]
3. D1 priming enhances both D1- and D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats.
Pollack AE; Thomas LI
Pharmacol Biochem Behav; 2010 Jan; 94(3):346-51. PubMed ID: 19800912
[TBL] [Abstract][Full Text] [Related]
4. Priming of a D1 dopamine receptor behavioural response is dissociated from striatal immediate-early gene activity.
Paul ML; Currie RW; Robertson HA
Neuroscience; 1995 May; 66(2):347-59. PubMed ID: 7477877
[TBL] [Abstract][Full Text] [Related]
5. Time dependence and role of N-methyl-D-aspartate glutamate receptors in the priming of D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats.
Pollack AE; Strauss JB
Brain Res; 1999 May; 827(1-2):160-8. PubMed ID: 10320705
[TBL] [Abstract][Full Text] [Related]
6. Motor stimulant effects of caffeine in 6-hydroxydopamine-lesioned rats are dependent on previous stimulation of dopamine receptors: a different role of D1 and D2 receptors.
Fenu S; Morelli M
Eur J Neurosci; 1998 May; 10(5):1878-84. PubMed ID: 9751157
[TBL] [Abstract][Full Text] [Related]
7. Role of NMDA and AMPA glutamate receptors in the induction and the expression of dopamine-mediated sensitization in 6-hydroxydopamine-lesioned rats.
Pollack AE; St Martin JL; MacPherson AT
Synapse; 2005 Apr; 56(1):45-53. PubMed ID: 15700284
[TBL] [Abstract][Full Text] [Related]
8. Repeated D1 dopamine receptor agonist administration prevents the development of both D1 and D2 striatal receptor supersensitivity following denervation.
Hu XT; White FJ
Synapse; 1992 Mar; 10(3):206-16. PubMed ID: 1532677
[TBL] [Abstract][Full Text] [Related]
9. Synergistic interaction between an adenosine antagonist and a D1 dopamine agonist on rotational behavior and striatal c-Fos induction in 6-hydroxydopamine-lesioned rats.
Pollack AE; Fink JS
Brain Res; 1996 Dec; 743(1-2):124-30. PubMed ID: 9017239
[TBL] [Abstract][Full Text] [Related]
10. D1-like and D2-like dopamine receptors synergistically activate rotation and c-fos expression in the dopamine-depleted striatum in a rat model of Parkinson's disease.
Paul ML; Graybiel AM; David JC; Robertson HA
J Neurosci; 1992 Oct; 12(10):3729-42. PubMed ID: 1357113
[TBL] [Abstract][Full Text] [Related]
11. Behavioral responsitivity to dopamine receptor agonists after extensive striatal dopamine lesions during development.
Neal-Beliveau BS; Joyce JN
Dev Psychobiol; 1998 May; 32(4):313-26. PubMed ID: 9589220
[TBL] [Abstract][Full Text] [Related]
12. Role of a 35 kDa fos-related antigen (FRA) in the long-term induction of striatal dynorphin expression in the 6-hydroxydopamine lesioned rat.
Bronstein DM; Ye H; Pennypacker KR; Hudson PM; Hong JS
Brain Res Mol Brain Res; 1994 May; 23(3):191-203. PubMed ID: 7914658
[TBL] [Abstract][Full Text] [Related]
13. Abnormal involuntary movement (AIM) expression following D2 dopamine agonist challenge is determined by the nature of prior dopamine receptor stimulation (priming) in 6-hydroxydopamine lesioned rats.
Drake JD; Kibuuka LN; Dimitrov KD; Pollack AE
Pharmacol Biochem Behav; 2013 Apr; 105():26-33. PubMed ID: 23369985
[TBL] [Abstract][Full Text] [Related]
14. Effects of intrasubthalamic injection of dopamine receptor agonists on subthalamic neurons in normal and 6-hydroxydopamine-lesioned rats: an electrophysiological and c-Fos study.
Hassani OK; Féger J
Neuroscience; 1999; 92(2):533-43. PubMed ID: 10408602
[TBL] [Abstract][Full Text] [Related]
15. Striatal Fos expression is indicative of dopamine D1/D2 synergism and receptor supersensitivity.
LaHoste GJ; Yu J; Marshall JF
Proc Natl Acad Sci U S A; 1993 Aug; 90(16):7451-5. PubMed ID: 8102797
[TBL] [Abstract][Full Text] [Related]
16. Ontogenetic quinpirole treatments fail to prime for D2 agonist-enhancement of locomotor activity in 6-hydroxydopamine-lesioned rats.
Brus R; Kostrzewa RM; Nowak P; Perry KW; Kostrzewa JP
Neurotox Res; 2003; 5(5):329-38. PubMed ID: 14715452
[TBL] [Abstract][Full Text] [Related]
17. Dopamine supersensitivity and D1/D2 synergism are unrelated to changes in striatal receptor density.
LaHoste GJ; Marshall JF
Synapse; 1992 Sep; 12(1):14-26. PubMed ID: 1357762
[TBL] [Abstract][Full Text] [Related]
18. The atypical dopamine D1 receptor agonist SKF 83959 induces striatal Fos expression in rats.
Wirtshafter D; Osborn CV
Eur J Pharmacol; 2005 Dec; 528(1-3):88-94. PubMed ID: 16324697
[TBL] [Abstract][Full Text] [Related]
19. Autoradiographic study of striatal dopamine re-uptake sites and dopamine D1 and D2 receptors in a 6-hydroxydopamine and quinolinic acid double-lesion rat model of striatonigral degeneration (multiple system atrophy) and effects of embryonic ventral mesencephalic, striatal or co-grafts.
Puschban Z; Scherfler C; Granata R; Laboyrie P; Quinn NP; Jenner P; Poewe W; Wenning GK
Neuroscience; 2000; 95(2):377-88. PubMed ID: 10658617
[TBL] [Abstract][Full Text] [Related]
20. NMDA glutamate receptor stimulation is required for the expression of D2 dopamine mediated responses in apomorphine primed 6-hydroxydopamine lesioned rats.
Pollack AE; Haisley EC
Brain Res; 2001 Apr; 897(1-2):213-6. PubMed ID: 11282380
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]