These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

156 related articles for article (PubMed ID: 10677533)

  • 1. Generation of in vivo activating factors in the ischemic intestine by pancreatic enzymes.
    Mitsuoka H; Kistler EB; Schmid-Schonbein GW
    Proc Natl Acad Sci U S A; 2000 Feb; 97(4):1772-7. PubMed ID: 10677533
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Pancreatic proteases and inflammatory mediators in peritoneal fluid during splanchnic arterial occlusion and reperfusion.
    Ishimaru K; Mitsuoka H; Unno N; Inuzuka K; Nakamura S; Schmid-Schönbein GW
    Shock; 2004 Nov; 22(5):467-71. PubMed ID: 15489640
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Protease inhibition in the intestinal lumen: attenuation of systemic inflammation and early indicators of multiple organ failure in shock.
    Mitsuoka H; Kistler EB; Schmid-Schönbein GW
    Shock; 2002 Mar; 17(3):205-9. PubMed ID: 11900339
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Pancreatic protease inhibition during shock attenuates cell activation and peripheral inflammation.
    Fitzal F; DeLano FA; Young C; Rosario HS; Schmid-Schönbein GW
    J Vasc Res; 2002; 39(4):320-9. PubMed ID: 12187122
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Mechanisms for blockade of in vivo activator production in the ischemic intestine and multi-organ failure.
    Mitsuoka H; Schmid-Schönbein GW
    Shock; 2000 Nov; 14(5):522-7. PubMed ID: 11092684
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Pancreatic digestive enzyme blockade in the intestine increases survival after experimental shock.
    DeLano FA; Hoyt DB; Schmid-Schönbein GW
    Sci Transl Med; 2013 Jan; 5(169):169ra11. PubMed ID: 23345609
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Pancreatic enzymes generate cytotoxic mediators in the intestine.
    Penn AH; Hugli TE; Schmid-Schönbein GW
    Shock; 2007 Mar; 27(3):296-304. PubMed ID: 17304111
    [TBL] [Abstract][Full Text] [Related]  

  • 8. The pancreas as a source of cardiovascular cell activating factors.
    Kistler EB; Hugli TE; Schmid-Schönbein GW
    Microcirculation; 2000 Jun; 7(3):183-92. PubMed ID: 10901497
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Improvement in early symptoms of shock by delayed intestinal protease inhibition.
    Fitzal F; DeLano FA; Young C; Schmid-Schönbein GW
    Arch Surg; 2004 Sep; 139(9):1008-16. PubMed ID: 15381622
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Impaired small-bowel barrier integrity in the presence of lumenal pancreatic digestive enzymes leads to circulatory shock.
    Kistler EB; Alsaigh T; Chang M; Schmid-Schönbein GW
    Shock; 2012 Aug; 38(3):262-7. PubMed ID: 22576000
    [TBL] [Abstract][Full Text] [Related]  

  • 11. [Role of pancreatic enzymes in gut injury secondary to trauma/hemorrhagic shock in rats].
    Shi HP; Wen Y; Zhang C; Xue Q; Lei ST
    Di Yi Jun Yi Da Xue Xue Bao; 2004 Oct; 24(10):1137-9. PubMed ID: 15485785
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Plasma activation during splanchnic arterial occlusion shock.
    Kistler EB; Lefer AM; Hugli TE; Schmid-Schönbein GW
    Shock; 2000 Jul; 14(1):30-4. PubMed ID: 10909890
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Protease activity increases in plasma, peritoneal fluid, and vital organs after hemorrhagic shock in rats.
    Altshuler AE; Penn AH; Yang JA; Kim GR; Schmid-Schönbein GW
    PLoS One; 2012; 7(3):e32672. PubMed ID: 22479334
    [TBL] [Abstract][Full Text] [Related]  

  • 14. The intestine as source of cytotoxic mediators in shock: free fatty acids and degradation of lipid-binding proteins.
    Penn AH; Schmid-Schönbein GW
    Am J Physiol Heart Circ Physiol; 2008 Apr; 294(4):H1779-92. PubMed ID: 18263716
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Pancreatic digestive enzymes are potent generators of mediators for leukocyte activation and mortality.
    Waldo SW; Rosario HS; Penn AH; Schmid-Schönbein GW
    Shock; 2003 Aug; 20(2):138-43. PubMed ID: 12865657
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Pancreatic digestive enzyme blockade in the small intestine prevents insulin resistance in hemorrhagic shock.
    DeLano FA; Schmid-Schönbein GW
    Shock; 2014 Jan; 41(1):55-61. PubMed ID: 24088998
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Autodigestion by migrated trypsin is a major factor in small intestinal ischemia-reperfusion injury.
    Verhaegh R; Petrat F; Brencher L; Kirsch M; de Groot H
    J Surg Res; 2017 Nov; 219():266-278. PubMed ID: 29078893
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Breakdown of mucin as barrier to digestive enzymes in the ischemic rat small intestine.
    Chang M; Alsaigh T; Kistler EB; Schmid-Schönbein GW
    PLoS One; 2012; 7(6):e40087. PubMed ID: 22768227
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Disruption of the mucosal barrier during gut ischemia allows entry of digestive enzymes into the intestinal wall.
    Chang M; Kistler EB; Schmid-Schönbein GW
    Shock; 2012 Mar; 37(3):297-305. PubMed ID: 22089198
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Biomechanical aspects of the auto-digestion theory.
    Schmid-Schönbein GW
    Mol Cell Biomech; 2008 Jun; 5(2):83-95. PubMed ID: 18589497
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 8.