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2. The interaction of p53 with replication protein A mediates suppression of homologous recombination. Romanova LY; Willers H; Blagosklonny MV; Powell SN Oncogene; 2004 Dec; 23(56):9025-33. PubMed ID: 15489903 [TBL] [Abstract][Full Text] [Related]
3. Suppression of the p53- or pRB-mediated G1 checkpoint is required for E2F-induced S-phase entry. Lomazzi M; Moroni MC; Jensen MR; Frittoli E; Helin K Nat Genet; 2002 Jun; 31(2):190-4. PubMed ID: 11992123 [TBL] [Abstract][Full Text] [Related]
4. Cell cycle control by p53 in normal (3T3) and chemically transformed (Meth A) mouse cells. I. Regulation of p53 expression. Steinmeyer K; Maacke H; Deppert W Oncogene; 1990 Nov; 5(11):1691-9. PubMed ID: 2267135 [TBL] [Abstract][Full Text] [Related]
6. Homologous recombination induced by replication inhibition, is stimulated by expression of mutant p53. Saintigny Y; Lopez BS Oncogene; 2002 Jan; 21(3):488-92. PubMed ID: 11821962 [TBL] [Abstract][Full Text] [Related]
7. Maintenance of genomic integrity by p53: complementary roles for activated and non-activated p53. Albrechtsen N; Dornreiter I; Grosse F; Kim E; Wiesmüller L; Deppert W Oncogene; 1999 Dec; 18(53):7706-17. PubMed ID: 10618711 [TBL] [Abstract][Full Text] [Related]
8. Prevention of mammalian DNA reduplication, following the release from the mitotic spindle checkpoint, requires p53 protein, but not p53-mediated transcriptional activity. Notterman D; Young S; Wainger B; Levine AJ Oncogene; 1998 Nov; 17(21):2743-51. PubMed ID: 9840938 [TBL] [Abstract][Full Text] [Related]
10. p53 modulates base excision repair activity in a cell cycle-specific manner after genotoxic stress. Offer H; Zurer I; Banfalvi G; Reha'k M; Falcovitz A; Milyavsky M; Goldfinger N; Rotter V Cancer Res; 2001 Jan; 61(1):88-96. PubMed ID: 11196204 [TBL] [Abstract][Full Text] [Related]
11. p53/p21(CIP1) cooperate in enforcing rapamycin-induced G(1) arrest and determine the cellular response to rapamycin. Huang S; Liu LN; Hosoi H; Dilling MB; Shikata T; Houghton PJ Cancer Res; 2001 Apr; 61(8):3373-81. PubMed ID: 11309295 [TBL] [Abstract][Full Text] [Related]
12. A transcriptional activation function of p53 is dispensable for and inhibitory of its apoptotic function. Kokontis JM; Wagner AJ; O'Leary M; Liao S; Hay N Oncogene; 2001 Feb; 20(6):659-68. PubMed ID: 11313999 [TBL] [Abstract][Full Text] [Related]
13. A novel human p53 isoform is an essential element of the ATR-intra-S phase checkpoint. Rohaly G; Chemnitz J; Dehde S; Nunez AM; Heukeshoven J; Deppert W; Dornreiter I Cell; 2005 Jul; 122(1):21-32. PubMed ID: 16009130 [TBL] [Abstract][Full Text] [Related]
14. Cell cycle control of p53 in normal (3T3) and chemically transformed (Meth A) mouse cells. II. Requirement for cell cycle progression. Deppert W; Buschhausen-Denker G; Patschinsky T; Steinmeyer K Oncogene; 1990 Nov; 5(11):1701-6. PubMed ID: 2267136 [TBL] [Abstract][Full Text] [Related]
15. Suppression of replication fork progression in low-dose-specific p53-dependent S-phase DNA damage checkpoint. Shimura T; Toyoshima M; Adiga SK; Kunoh T; Nagai H; Shimizu N; Inoue M; Niwa O Oncogene; 2006 Sep; 25(44):5921-32. PubMed ID: 16682953 [TBL] [Abstract][Full Text] [Related]
16. Inactivation of p53 results in high rates of homologous recombination. Mekeel KL; Tang W; Kachnic LA; Luo CM; DeFrank JS; Powell SN Oncogene; 1997 Apr; 14(15):1847-57. PubMed ID: 9150391 [TBL] [Abstract][Full Text] [Related]
17. Loss of normal G1 checkpoint control is an early step in carcinogenesis, independent of p53 status. Syljuåsen RG; Krolewski B; Little JB Cancer Res; 1999 Mar; 59(5):1008-14. PubMed ID: 10070956 [TBL] [Abstract][Full Text] [Related]
18. Association of p53 and MSH2 with recombinative repair complexes during S phase. Zink D; Mayr C; Janz C; Wiesmüller L Oncogene; 2002 Jul; 21(31):4788-800. PubMed ID: 12101417 [TBL] [Abstract][Full Text] [Related]
20. Activation of the p53-dependent G1 checkpoint response in mouse embryo fibroblasts depends on the specific DNA damage inducer. Attardi LD; de Vries A; Jacks T Oncogene; 2004 Jan; 23(4):973-80. PubMed ID: 14749764 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]