These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
3. Skewed differentiation of thymocytes toward CD8 T cells by 2,3,7,8-tetrachlorodibenzo- p-dioxin requires activation of the extracellular signal-related kinase pathway. Tsukumo S; Iwata M; Tohyama C; Nohara K Arch Toxicol; 2002 Jun; 76(5-6):335-43. PubMed ID: 12107651 [TBL] [Abstract][Full Text] [Related]
4. Differential effects of diethylstilbestrol and 2,3,7,8-tetrachlorodibenzo-p-dioxin on thymocyte differentiation, proliferation, and apoptosis in bcl-2 transgenic mouse fetal thymus organ culture. Lai ZW; Fiore NC; Hahn PJ; Gasiewicz TA; Silverstone AE Toxicol Appl Pharmacol; 2000 Oct; 168(1):15-24. PubMed ID: 11000096 [TBL] [Abstract][Full Text] [Related]
5. Alterations in the developing immune system of the F344 rat after perinatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin I. [correction of II]. Effects on the fetus and the neonate. Gehrs BC; Smialowicz RJ Toxicology; 1997 Oct; 122(3):219-28. PubMed ID: 9328222 [TBL] [Abstract][Full Text] [Related]
6. 2,3,7,8-Tetrachlorodibenzo-p-dioxin and diethylstilbestrol affect thymocytes at different stages of development in fetal thymus organ culture. Lai ZW; Fiore NC; Gasiewicz TA; Silverstone AE Toxicol Appl Pharmacol; 1998 Apr; 149(2):167-77. PubMed ID: 9571985 [TBL] [Abstract][Full Text] [Related]
7. Flow cytometric analysis of lymphocyte subpopulations in the spleen and thymus of mice exposed to an acute immunosuppressive dose of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Kerkvliet NI; Brauner JA Environ Res; 1990 Aug; 52(2):146-54. PubMed ID: 2118453 [TBL] [Abstract][Full Text] [Related]
8. Perinatal thymocyte antigen expression and postnatal immune development altered by gestational exposure to tetrachlorodibenzo-p-dioxin (TCDD). Holladay SD; Lindstrom P; Blaylock BL; Comment CE; Germolec DR; Heindell JJ; Luster MI Teratology; 1991 Oct; 44(4):385-93. PubMed ID: 1683717 [TBL] [Abstract][Full Text] [Related]
9. Thymocyte development in Ah-receptor-deficient mice is refractory to TCDD-inducible changes. Hundeiker C; Pineau T; Cassar G; Betensky RA; Gleichmann E; Esser C Int J Immunopharmacol; 1999 Dec; 21(12):841-59. PubMed ID: 10606004 [TBL] [Abstract][Full Text] [Related]
10. The effects of perinatal exposure to low doses of 2,3,7,8-tetrachlorodibenzo-p-dioxin on immune organs in rats. Nohara K; Fujimaki H; Tsukumo S; Ushio H; Miyabara Y; Kijima M; Tohyama C; Yonemoto J Toxicology; 2000 Nov; 154(1-3):123-33. PubMed ID: 11118676 [TBL] [Abstract][Full Text] [Related]
11. Targeted deletion of the aryl hydrocarbon receptor in dendritic cells prevents thymic atrophy in response to dioxin. Beamer CA; Kreitinger JM; Cole SL; Shepherd DM Arch Toxicol; 2019 Feb; 93(2):355-368. PubMed ID: 30499018 [TBL] [Abstract][Full Text] [Related]
12. Thymic stroma exposed to arylhydrocarbon receptor-binding xenobiotics fails to support proliferation of early thymocytes but induces differentiation. Kremer J; Gleichmann E; Esser C J Immunol; 1994 Sep; 153(6):2778-86. PubMed ID: 8077682 [TBL] [Abstract][Full Text] [Related]
13. 2,3,7,8-tetrachlorodibenzo-p-dioxin enhances negative selection of T cells in the thymus but allows autoreactive T cells to escape deletion and migrate to the periphery. Fisher MT; Nagarkatti M; Nagarkatti PS Mol Pharmacol; 2005 Jan; 67(1):327-35. PubMed ID: 15492116 [TBL] [Abstract][Full Text] [Related]
14. Functional activation of arylhydrocarbon receptor (AhR) in primary T cells by 2,3,7,8-tetrachlorodibenzo-p-dioxin. Doi H; Baba T; Tohyama C; Nohara K Chemosphere; 2003 Jul; 52(4):655-62. PubMed ID: 12738279 [TBL] [Abstract][Full Text] [Related]
15. Evidence for induction of apoptosis in T cells from murine fetal thymus following perinatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Camacho IA; Nagarkatti M; Nagarkatti PS Toxicol Sci; 2004 Mar; 78(1):96-106. PubMed ID: 14718643 [TBL] [Abstract][Full Text] [Related]
16. Thymic alterations induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin are strictly dependent on aryl hydrocarbon receptor activation in hemopoietic cells. Staples JE; Murante FG; Fiore NC; Gasiewicz TA; Silverstone AE J Immunol; 1998 Apr; 160(8):3844-54. PubMed ID: 9558089 [TBL] [Abstract][Full Text] [Related]
17. Lymphodepletion of the thymus cortex in rats after single oral intubation of 2,3,7,8-tetrachlorodibenzo-p-dioxin. De Heer C; Schuurman HJ; Vos JG; Van Loveren H Chemosphere; 1994; 29(9-11):2295-9. PubMed ID: 7850377 [TBL] [Abstract][Full Text] [Related]
18. Role of Fas apoptosis and MHC genes in 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced immunotoxicity of T cells. Rhile MJ; Nagarkatti M; Nagarkatti PS Toxicology; 1996 Jun; 110(1-3):153-67. PubMed ID: 8658555 [TBL] [Abstract][Full Text] [Related]
19. Cytokine gene expression during ontogeny in murine thymus on activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxin. Lai ZW; Hundeiker C; Gleichmann E; Esser C Mol Pharmacol; 1997 Jul; 52(1):30-7. PubMed ID: 9224809 [TBL] [Abstract][Full Text] [Related]
20. Dexamethasone, beta-estradiol, and 2,3,7,8-tetrachlorodibenzo-p-dioxin elicit thymic atrophy through different cellular targets. Silverstone AE; Frazier DE; Fiore NC; Soults JA; Gasiewicz TA Toxicol Appl Pharmacol; 1994 Jun; 126(2):248-59. PubMed ID: 7516097 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]