435 related articles for article (PubMed ID: 10775125)
1. Nitric oxide produced via neuronal NOS may impair vasodilatation in septic rat skeletal muscle.
Gocan NC; Scott JA; Tyml K
Am J Physiol Heart Circ Physiol; 2000 May; 278(5):H1480-9. PubMed ID: 10775125
[TBL] [Abstract][Full Text] [Related]
2. Deletion of neuronal NOS prevents impaired vasodilation in septic mouse skeletal muscle.
Lidington D; Li F; Tyml K
Cardiovasc Res; 2007 Apr; 74(1):151-8. PubMed ID: 17258180
[TBL] [Abstract][Full Text] [Related]
3. Capillary and arteriolar responses to local vasodilators are impaired in a rat model of sepsis.
Tyml K; Yu J; McCormack DG
J Appl Physiol (1985); 1998 Mar; 84(3):837-44. PubMed ID: 9480941
[TBL] [Abstract][Full Text] [Related]
4. Contribution of nitric oxide synthase isoforms to cholinergic vasodilation in murine retinal arterioles.
Gericke A; Goloborodko E; Sniatecki JJ; Steege A; Wojnowski L; Pfeiffer N
Exp Eye Res; 2013 Apr; 109():60-6. PubMed ID: 23434456
[TBL] [Abstract][Full Text] [Related]
5. Ascorbate protects against impaired arteriolar constriction in sepsis by inhibiting inducible nitric oxide synthase expression.
Wu F; Wilson JX; Tyml K
Free Radic Biol Med; 2004 Oct; 37(8):1282-9. PubMed ID: 15451067
[TBL] [Abstract][Full Text] [Related]
6. Reduced arteriolar conducted vasoconstriction in septic mouse cremaster muscle is mediated by nNOS-derived NO.
McKinnon RL; Lidington D; Bolon M; Ouellette Y; Kidder GM; Tyml K
Cardiovasc Res; 2006 Jan; 69(1):236-44. PubMed ID: 16226732
[TBL] [Abstract][Full Text] [Related]
7. Selective iNOS inhibition prevents hypotension in septic rats while preserving endothelium-dependent vasodilation.
Strunk V; Hahnenkamp K; Schneuing M; Fischer LG; Rich GF
Anesth Analg; 2001 Mar; 92(3):681-7. PubMed ID: 11226101
[TBL] [Abstract][Full Text] [Related]
8. Kupffer cells protect liver from ischemia-reperfusion injury by an inducible nitric oxide synthase-dependent mechanism.
Hsu CM; Wang JS; Liu CH; Chen LW
Shock; 2002 Apr; 17(4):280-5. PubMed ID: 11954827
[TBL] [Abstract][Full Text] [Related]
9. Atorvastatin restores the impaired vascular endothelium-dependent relaxations mediated by nitric oxide and endothelium-derived hyperpolarizing factors but not hypotension in sepsis.
Subramani J; Kathirvel K; Leo MD; Kuntamallappanavar G; Uttam Singh T; Mishra SK
J Cardiovasc Pharmacol; 2009 Dec; 54(6):526-34. PubMed ID: 19755915
[TBL] [Abstract][Full Text] [Related]
10. Nascent EDHF-mediated cerebral vasodilation in ovariectomized rats is not induced by eNOS dysfunction.
Xu HL; Santizo RA; Baughman VL; Pelligrino DA
Am J Physiol Heart Circ Physiol; 2003 Nov; 285(5):H2045-53. PubMed ID: 12869371
[TBL] [Abstract][Full Text] [Related]
11. ACh dilates pial arterioles in endothelial and neuronal NOS knockout mice by NO-dependent mechanisms.
Meng W; Ma J; Ayata C; Hara H; Huang PL; Fishman MC; Moskowitz MA
Am J Physiol; 1996 Sep; 271(3 Pt 2):H1145-50. PubMed ID: 8853353
[TBL] [Abstract][Full Text] [Related]
12. Neuronal nitric oxide synthase-derived hydrogen peroxide is a major endothelium-dependent relaxing factor.
Capettini LS; Cortes SF; Gomes MA; Silva GA; Pesquero JL; Lopes MJ; Teixeira MM; Lemos VS
Am J Physiol Heart Circ Physiol; 2008 Dec; 295(6):H2503-11. PubMed ID: 18952716
[TBL] [Abstract][Full Text] [Related]
13. Nitric oxide synthase inhibition reverses arteriolar hyporesponsiveness to endothelin-1 in septic rats.
Hollenberg SM; Piotrowski MJ; Parrillo JE
Am J Physiol; 1997 Mar; 272(3 Pt 2):R969-74. PubMed ID: 9087662
[TBL] [Abstract][Full Text] [Related]
14. Role of nitric oxide synthase isoforms for ophthalmic artery reactivity in mice.
Laspas P; Goloborodko E; Sniatecki JJ; Kordasz ML; Manicam C; Wojnowski L; Li H; Patzak A; Pfeiffer N; Gericke A
Exp Eye Res; 2014 Oct; 127():1-8. PubMed ID: 25017185
[TBL] [Abstract][Full Text] [Related]
15. Effect of aminoguanidine on plasma nitric oxide by-products and blood flow during chronic peritoneal sepsis.
Alden KJ; Motew SJ; Sharma AC; Ferguson JL
Shock; 1998 Apr; 9(4):289-95. PubMed ID: 9565258
[TBL] [Abstract][Full Text] [Related]
16. Endotoxin releases a substance from the aorta that dilates an isolated arteriole by up-regulating INOS.
Viol AW; Prewitt RL; Doviak M; Britt LD
J Surg Res; 2005 Aug; 127(2):106-11. PubMed ID: 15921695
[TBL] [Abstract][Full Text] [Related]
17. Evaluating the role of inducible nitric oxide synthase using a novel and selective inducible nitric oxide synthase inhibitor in septic lung injury produced by cecal ligation and puncture.
Okamoto I; Abe M; Shibata K; Shimizu N; Sakata N; Katsuragi T; Tanaka K
Am J Respir Crit Care Med; 2000 Aug; 162(2 Pt 1):716-22. PubMed ID: 10934111
[TBL] [Abstract][Full Text] [Related]
18. Sepsis increases NOS-2 activity and decreases non-NOS-mediated acetylcholine-induced dilation in rat aorta.
Beach PK; Spain DA; Kawabe T; Harris PD; Garrison RN
J Surg Res; 2001 Mar; 96(1):17-22. PubMed ID: 11180991
[TBL] [Abstract][Full Text] [Related]
19. Hindlimb unweighting alters endothelium-dependent vasodilation and ecNOS expression in soleus arterioles.
Schrage WG; Woodman CR; Laughlin MH
J Appl Physiol (1985); 2000 Oct; 89(4):1483-90. PubMed ID: 11007586
[TBL] [Abstract][Full Text] [Related]
20. Effects of ageing and exercise training on endothelium-dependent vasodilatation and structure of rat skeletal muscle arterioles.
Spier SA; Delp MD; Meininger CJ; Donato AJ; Ramsey MW; Muller-Delp JM
J Physiol; 2004 May; 556(Pt 3):947-58. PubMed ID: 15004211
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]