198 related articles for article (PubMed ID: 10803461)
1. LMP1 of Epstein-Barr virus suppresses cellular senescence associated with the inhibition of p16INK4a expression.
Yang X; He Z; Xin B; Cao L
Oncogene; 2000 Apr; 19(16):2002-13. PubMed ID: 10803461
[TBL] [Abstract][Full Text] [Related]
2. Epstein-Barr virus LMP1 blocks p16INK4a-RB pathway by promoting nuclear export of E2F4/5.
Ohtani N; Brennan P; Gaubatz S; Sanij E; Hertzog P; Wolvetang E; Ghysdael J; Rowe M; Hara E
J Cell Biol; 2003 Jul; 162(2):173-83. PubMed ID: 12860972
[TBL] [Abstract][Full Text] [Related]
3. Murine fibroblasts lacking p21 undergo senescence and are resistant to transformation by oncogenic Ras.
Pantoja C; Serrano M
Oncogene; 1999 Sep; 18(35):4974-82. PubMed ID: 10490832
[TBL] [Abstract][Full Text] [Related]
4. A functional screen identifies hDRIL1 as an oncogene that rescues RAS-induced senescence.
Peeper DS; Shvarts A; Brummelkamp T; Douma S; Koh EY; Daley GQ; Bernards R
Nat Cell Biol; 2002 Feb; 4(2):148-53. PubMed ID: 11812999
[TBL] [Abstract][Full Text] [Related]
5. Intrinsic cooperation between p16INK4a and p21Waf1/Cip1 in the onset of cellular senescence and tumor suppression in vivo.
Takeuchi S; Takahashi A; Motoi N; Yoshimoto S; Tajima T; Yamakoshi K; Hirao A; Yanagi S; Fukami K; Ishikawa Y; Sone S; Hara E; Ohtani N
Cancer Res; 2010 Nov; 70(22):9381-90. PubMed ID: 21062974
[TBL] [Abstract][Full Text] [Related]
6. A novel dominant-negative mutant form of Epstein-Barr virus latent membrane protein-1 (LMP1) selectively and differentially impairs LMP1 and TNF signaling pathways.
Adriaenssens E; Mougel A; Goormachtigh G; Loing E; Fafeur V; Auriault C; Coll J
Oncogene; 2004 Apr; 23(15):2681-93. PubMed ID: 14767477
[TBL] [Abstract][Full Text] [Related]
7. H-, K- and N-Ras inhibit myeloid leukemia cell proliferation by a p21WAF1-dependent mechanism.
Delgado MD; Vaqué JP; Arozarena I; López-Ilasaca MA; Martínez C; Crespo P; León J
Oncogene; 2000 Feb; 19(6):783-90. PubMed ID: 10698496
[TBL] [Abstract][Full Text] [Related]
8. Cellular senescence induced by p53-ras cooperation is independent of p21waf1 in murine embryo fibroblasts.
Castro ME; del Valle Guijarro M; Moneo V; Carnero A
J Cell Biochem; 2004 Jun; 92(3):514-24. PubMed ID: 15156563
[TBL] [Abstract][Full Text] [Related]
9. Expression of the Leo1-like domain of replicative senescence down-regulated Leo1-like (RDL) protein promotes senescence of 2BS fibroblasts.
Zhao L; Tong T; Zhang Z
FASEB J; 2005 Apr; 19(6):521-32. PubMed ID: 15791002
[TBL] [Abstract][Full Text] [Related]
10. TRADD domain of Epstein-Barr virus transforming protein LMP1 is essential for inducing immortalization and suppressing senescence of primary rodent fibroblasts.
Xin B; He Z; Yang X; Chan CP; Ng MH; Cao L
J Virol; 2001 Mar; 75(6):3010-5. PubMed ID: 11222727
[TBL] [Abstract][Full Text] [Related]
11. Telomerase activation by Epstein-Barr virus latent membrane protein 1 is associated with c-Myc expression in human nasopharyngeal epithelial cells.
Yang J; Deng X; Deng L; Gu H; Fan W; Cao Y
J Exp Clin Cancer Res; 2004 Sep; 23(3):495-506. PubMed ID: 15595642
[TBL] [Abstract][Full Text] [Related]
12. Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence.
Ohtani N; Zebedee Z; Huot TJ; Stinson JA; Sugimoto M; Ohashi Y; Sharrocks AD; Peters G; Hara E
Nature; 2001 Feb; 409(6823):1067-70. PubMed ID: 11234019
[TBL] [Abstract][Full Text] [Related]
13. Latent membrane protein 1 of Epstein-Barr virus plays an important role in the serum starvation resistance of Epstein-Barr virus-immortalized B lymphocytes.
Park CH; Kim HR; Kim J; Jang SH; Lee KY; Chung GH; Jang YS
J Cell Biochem; 2004 Mar; 91(4):777-85. PubMed ID: 14991769
[TBL] [Abstract][Full Text] [Related]
14. The latent membrane protein 1 of Epstein-Barr virus and loss of the INK4a locus: paradoxes resolve to cooperation in carcinogenesis in vivo.
Macdiarmid J; Stevenson D; Campbell DH; Wilson JB
Carcinogenesis; 2003 Jul; 24(7):1209-18. PubMed ID: 12807717
[TBL] [Abstract][Full Text] [Related]
15. Expression of catalytically active telomerase does not prevent premature senescence caused by overexpression of oncogenic Ha-Ras in normal human fibroblasts.
Wei S; Wei S; Sedivy JM
Cancer Res; 1999 Apr; 59(7):1539-43. PubMed ID: 10197626
[TBL] [Abstract][Full Text] [Related]
16. Role of the alternative INK4A proteins in human keratinocyte senescence: evidence for the specific inactivation of p16INK4A upon immortalization.
Munro J; Stott FJ; Vousden KH; Peters G; Parkinson EK
Cancer Res; 1999 Jun; 59(11):2516-21. PubMed ID: 10363964
[TBL] [Abstract][Full Text] [Related]
17. Histone deacetylase inhibitors induce a senescence-like state in human cells by a p16-dependent mechanism that is independent of a mitotic clock.
Munro J; Barr NI; Ireland H; Morrison V; Parkinson EK
Exp Cell Res; 2004 May; 295(2):525-38. PubMed ID: 15093749
[TBL] [Abstract][Full Text] [Related]
18. Epstein-Barr virus latent membrane protein 1 (LMP1) upregulates Id1 expression in nasopharyngeal epithelial cells.
Li HM; Zhuang ZH; Wang Q; Pang JC; Wang XH; Wong HL; Feng HC; Jin DY; Ling MT; Wong YC; Eliopoulos AG; Young LS; Huang DP; Tsao SW
Oncogene; 2004 May; 23(25):4488-94. PubMed ID: 15064751
[TBL] [Abstract][Full Text] [Related]
19. Modulation of the expression of p16INK4a and p14ARF by hnRNP A1 and A2 RNA binding proteins: implications for cellular senescence.
Zhu D; Xu G; Ghandhi S; Hubbard K
J Cell Physiol; 2002 Oct; 193(1):19-25. PubMed ID: 12209876
[TBL] [Abstract][Full Text] [Related]
20. A senescence rescue screen identifies BCL6 as an inhibitor of anti-proliferative p19(ARF)-p53 signaling.
Shvarts A; Brummelkamp TR; Scheeren F; Koh E; Daley GQ; Spits H; Bernards R
Genes Dev; 2002 Mar; 16(6):681-6. PubMed ID: 11914273
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
