These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
468 related articles for article (PubMed ID: 10850453)
1. Mitogenic conversion of transforming growth factor-beta1 effect by oncogenic Ha-Ras-induced activation of the mitogen-activated protein kinase signaling pathway in human prostate cancer. Park BJ; Park JI; Byun DS; Park JH; Chi SG Cancer Res; 2000 Jun; 60(11):3031-8. PubMed ID: 10850453 [TBL] [Abstract][Full Text] [Related]
2. Transforming growth factor-beta1 activates interleukin-6 expression in prostate cancer cells through the synergistic collaboration of the Smad2, p38-NF-kappaB, JNK, and Ras signaling pathways. Park JI; Lee MG; Cho K; Park BJ; Chae KS; Byun DS; Ryu BK; Park YK; Chi SG Oncogene; 2003 Jul; 22(28):4314-32. PubMed ID: 12853969 [TBL] [Abstract][Full Text] [Related]
3. Requirement of Ras/MAPK pathway activation by transforming growth factor beta for transforming growth factor beta 1 production in a Smad-dependent pathway. Yue J; Mulder KM J Biol Chem; 2000 Oct; 275(40):30765-73. PubMed ID: 10843986 [TBL] [Abstract][Full Text] [Related]
4. Transforming growth factor-beta1-dependent activation of Smad2/3 and up-regulation of PAI-1 expression is negatively regulated by Src in SKOV-3 human ovarian cancer cells. Wakahara K; Kobayashi H; Yagyu T; Matsuzaki H; Kondo T; Kurita N; Sekino H; Inagaki K; Suzuki M; Kanayama N; Terao T J Cell Biochem; 2004 Oct; 93(3):437-53. PubMed ID: 15372629 [TBL] [Abstract][Full Text] [Related]
5. Transforming growth factor beta 1 stimulates expression of the Epstein-Barr virus BZLF1 immediate-early gene product ZEBRA by an indirect mechanism which requires the MAPK kinase pathway. Fahmi H; Cochet C; Hmama Z; Opolon P; Joab I J Virol; 2000 Jul; 74(13):5810-8. PubMed ID: 10846060 [TBL] [Abstract][Full Text] [Related]
6. The transforming growth factor-beta/SMAD signaling pathway is present and functional in human mesangial cells. Poncelet AC; de Caestecker MP; Schnaper HW Kidney Int; 1999 Oct; 56(4):1354-65. PubMed ID: 10504488 [TBL] [Abstract][Full Text] [Related]
8. Insulin-like growth factor-I inhibits transcriptional responses of transforming growth factor-beta by phosphatidylinositol 3-kinase/Akt-dependent suppression of the activation of Smad3 but not Smad2. Song K; Cornelius SC; Reiss M; Danielpour D J Biol Chem; 2003 Oct; 278(40):38342-51. PubMed ID: 12876289 [TBL] [Abstract][Full Text] [Related]
9. Transforming growth factor-beta1 (TGF-beta)-induced apoptosis of prostate cancer cells involves Smad7-dependent activation of p38 by TGF-beta-activated kinase 1 and mitogen-activated protein kinase kinase 3. Edlund S; Bu S; Schuster N; Aspenström P; Heuchel R; Heldin NE; ten Dijke P; Heldin CH; Landström M Mol Biol Cell; 2003 Feb; 14(2):529-44. PubMed ID: 12589052 [TBL] [Abstract][Full Text] [Related]
10. Cross-talk between the p42/p44 MAP kinase and Smad pathways in transforming growth factor beta 1-induced furin gene transactivation. Blanchette F; Rivard N; Rudd P; Grondin F; Attisano L; Dubois CM J Biol Chem; 2001 Sep; 276(36):33986-94. PubMed ID: 11448947 [TBL] [Abstract][Full Text] [Related]
11. Blockade of Smad4 in transformed keratinocytes containing a Ras oncogene leads to hyperactivation of the Ras-dependent Erk signalling pathway associated with progression to undifferentiated carcinomas. Iglesias M; Frontelo P; Gamallo C; Quintanilla M Oncogene; 2000 Aug; 19(36):4134-45. PubMed ID: 10962574 [TBL] [Abstract][Full Text] [Related]
12. Smad4/DPC4-dependent regulation of biglycan gene expression by transforming growth factor-beta in pancreatic tumor cells. Chen WB; Lenschow W; Tiede K; Fischer JW; Kalthoff H; Ungefroren H J Biol Chem; 2002 Sep; 277(39):36118-28. PubMed ID: 12140283 [TBL] [Abstract][Full Text] [Related]
13. Suppression of transforming growth factor-beta-induced apoptosis through a phosphatidylinositol 3-kinase/Akt-dependent pathway. Chen RH; Su YH; Chuang RL; Chang TY Oncogene; 1998 Oct; 17(15):1959-68. PubMed ID: 9788439 [TBL] [Abstract][Full Text] [Related]
14. Transcriptional cross-talk between Smad, ERK1/2, and p38 mitogen-activated protein kinase pathways regulates transforming growth factor-beta-induced aggrecan gene expression in chondrogenic ATDC5 cells. Watanabe H; de Caestecker MP; Yamada Y J Biol Chem; 2001 Apr; 276(17):14466-73. PubMed ID: 11278290 [TBL] [Abstract][Full Text] [Related]
15. Grape seed extract inhibits EGF-induced and constitutively active mitogenic signaling but activates JNK in human prostate carcinoma DU145 cells: possible role in antiproliferation and apoptosis. Tyagi A; Agarwal R; Agarwal C Oncogene; 2003 Mar; 22(9):1302-16. PubMed ID: 12618755 [TBL] [Abstract][Full Text] [Related]
16. Beta-hydroxybutyrate-induced growth inhibition and collagen production in HK-2 cells are dependent on TGF-beta and Smad3. Guh JY; Chuang TD; Chen HC; Hung WC; Lai YH; Shin SJ; Chuang LY Kidney Int; 2003 Dec; 64(6):2041-51. PubMed ID: 14633126 [TBL] [Abstract][Full Text] [Related]
17. TGF-beta activated Smad signalling leads to a Smad3-mediated down-regulation of DSPP in an odontoblast cell line. He WX; Niu ZY; Zhao SL; Jin WL; Gao J; Smith AJ Arch Oral Biol; 2004 Nov; 49(11):911-8. PubMed ID: 15353247 [TBL] [Abstract][Full Text] [Related]
18. Potentiation of Smad transactivation by Jun proteins during a combined treatment with epidermal growth factor and transforming growth factor-beta in rat hepatocytes. role of phosphatidylinositol 3-kinase-induced AP-1 activation. Peron P; Rahmani M; Zagar Y; Durand-Schneider AM; Lardeux B; Bernuau D J Biol Chem; 2001 Mar; 276(13):10524-31. PubMed ID: 11134003 [TBL] [Abstract][Full Text] [Related]
19. Pathway- and expression level-dependent effects of oncogenic N-Ras: p27(Kip1) mislocalization by the Ral-GEF pathway and Erk-mediated interference with Smad signaling. Kfir S; Ehrlich M; Goldshmid A; Liu X; Kloog Y; Henis YI Mol Cell Biol; 2005 Sep; 25(18):8239-50. PubMed ID: 16135812 [TBL] [Abstract][Full Text] [Related]
20. Separate roles for H-Ras and Rac in signaling by transforming growth factor (TGF)-beta. H-Ras is essential for activation of MAP kinase, partially required for transcriptional activation by TGF-beta, but not required for signaling of growth suppression by TGF-beta. Yamamoto H; Atsuchi N; Tanaka H; Ogawa W; Abe M; Takeshita A; Ueno H Eur J Biochem; 1999 Aug; 264(1):110-9. PubMed ID: 10447679 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]