These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

149 related articles for article (PubMed ID: 11067919)

  • 1. Mechanisms of tolerance induction by a gene-transferred peptide-IgG fusion protein expressed in B lineage cells.
    El-Amine M; Melo M; Kang Y; Nguyen H; Qian J; Scott DW
    J Immunol; 2000 Nov; 165(10):5631-6. PubMed ID: 11067919
    [TBL] [Abstract][Full Text] [Related]  

  • 2. In vivo induction of tolerance by an Ig peptide is not affected by the deletion of FcR or a mutated IgG Fc fragment.
    el-Amine M; Hinshaw JA; Scott DW
    Int Immunol; 2002 Jul; 14(7):761-6. PubMed ID: 12096035
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Genetically transferred central and peripheral immune tolerance via retroviral-mediated expression of immunogenic epitopes in hematopoietic progenitors or peripheral B lymphocytes.
    Zambidis ET; Kurup A; Scott DW
    Mol Med; 1997 Mar; 3(3):212-24. PubMed ID: 9100227
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Tolerance induction via a B-cell delivered gene therapy-based protocol: optimization and role of the Ig scaffold.
    Lei TC; Su Y; Scott DW
    Cell Immunol; 2005 May; 235(1):12-20. PubMed ID: 16098495
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Induction of hyporesponsiveness to intact foreign protein via retroviral-mediated gene expression: the IgG scaffold is important for induction and maintenance of immune hyporesponsiveness.
    Kang Y; Melo M; Deng E; Tisch R; El-Amine M; Scott DW
    Proc Natl Acad Sci U S A; 1999 Jul; 96(15):8609-14. PubMed ID: 10411923
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Both resting and activated B lymphocytes expressing engineered peptide-Ig molecules serve as highly efficient tolerogenic vehicles in immunocompetent adult recipients.
    Zambidis ET; Barth RK; Scott DW
    J Immunol; 1997 Mar; 158(5):2174-82. PubMed ID: 9036963
    [TBL] [Abstract][Full Text] [Related]  

  • 7. B cells induce tolerance by presenting endogenous peptide-IgG on MHC class II molecules via an IFN-gamma-inducible lysosomal thiol reductase-dependent pathway.
    Su Y; Carey G; Maric M; Scott DW
    J Immunol; 2008 Jul; 181(2):1153-60. PubMed ID: 18606668
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Mechanisms of gene therapy for tolerance: B7 signaling is required for peptide-IgG gene-transferred tolerance induction.
    Litzinger MT; Su Y; Lei TC; Soukhareva N; Scott DW
    J Immunol; 2005 Jul; 175(2):780-7. PubMed ID: 16002674
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Retroviral delivery of GAD-IgG fusion construct induces tolerance and modulates diabetes: a role for CD4+ regulatory T cells and TGF-beta?
    Song L; Wang J; Wang R; Yu M; Sun Y; Han G; Li Y; Qian J; Scott DW; Kang Y; Soukhareva N; Shen B
    Gene Ther; 2004 Oct; 11(20):1487-96. PubMed ID: 15343360
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Gene therapy for immunologic tolerance: using bone marrow-derived cells to treat autoimmunity and hemophilia.
    Scott DW
    Curr Stem Cell Res Ther; 2011 Mar; 6(1):38-43. PubMed ID: 20955157
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Epitope-specific tolerance induction with an engineered immunoglobulin.
    Zambidis ET; Scott DW
    Proc Natl Acad Sci U S A; 1996 May; 93(10):5019-24. PubMed ID: 8643522
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Induction of tolerance to factor VIII inhibitors by gene therapy with immunodominant A2 and C2 domains presented by B cells as Ig fusion proteins.
    Lei TC; Scott DW
    Blood; 2005 Jun; 105(12):4865-70. PubMed ID: 15769892
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Gene transfer of Ig-fusion proteins into B cells prevents and treats autoimmune diseases.
    Melo ME; Qian J; El-Amine M; Agarwal RK; Soukhareva N; Kang Y; Scott DW
    J Immunol; 2002 May; 168(9):4788-95. PubMed ID: 11971030
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Feasibility of CTLA4Ig gene delivery and expression in vivo using retrovirally transduced myeloid dendritic cells that induce alloantigen-specific T cell anergy in vitro.
    Takayama T; Morelli AE; Robbins PD; Tahara H; Thomson AW
    Gene Ther; 2000 Aug; 7(15):1265-73. PubMed ID: 10918497
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Blockade of T cell activation using a surface-linked single-chain antibody to CTLA-4 (CD152).
    Griffin MD; Hong DK; Holman PO; Lee KM; Whitters MJ; O'Herrin SM; Fallarino F; Collins M; Segal DM; Gajewski TF; Kranz DM; Bluestone JA
    J Immunol; 2000 May; 164(9):4433-42. PubMed ID: 10779742
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Roles of cytotoxic T-lymphocyte-associated antigen-4 in the inductive phase of oral tolerance.
    Chen Y; Ma Y; Chen Y
    Immunology; 2002 Feb; 105(2):171-80. PubMed ID: 11872092
    [TBL] [Abstract][Full Text] [Related]  

  • 17. In vivo induction of type 1-like regulatory T cells using genetically modified B cells confers long-term IL-10-dependent antigen-specific unresponsiveness.
    Ahangarani RR; Janssens W; VanderElst L; Carlier V; VandenDriessche T; Chuah M; Weynand B; Vanoirbeek JA; Jacquemin M; Saint-Remy JM
    J Immunol; 2009 Dec; 183(12):8232-43. PubMed ID: 20007587
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Defining the requirements for peptide recognition in gene therapy-induced T cell tolerance.
    Bagley J; Wu Y; Sachs DH; Iacomini J
    J Immunol; 2000 Nov; 165(9):4842-7. PubMed ID: 11046007
    [TBL] [Abstract][Full Text] [Related]  

  • 19. B7 interactions with CD28 and CTLA-4 control tolerance or induction of mucosal inflammation in chronic experimental colitis.
    Liu Z; Geboes K; Hellings P; Maerten P; Heremans H; Vandenberghe P; Boon L; van Kooten P; Rutgeerts P; Ceuppens JL
    J Immunol; 2001 Aug; 167(3):1830-8. PubMed ID: 11466409
    [TBL] [Abstract][Full Text] [Related]  

  • 20. B cell function in mice transgenic for mCTLA4-H gamma 1: lack of germinal centers correlated with poor affinity maturation and class switching despite normal priming of CD4+ T cells.
    Lane P; Burdet C; Hubele S; Scheidegger D; Müller U; McConnell F; Kosco-Vilbois M
    J Exp Med; 1994 Mar; 179(3):819-30. PubMed ID: 7509361
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 8.