519 related articles for article (PubMed ID: 11095247)
1. Src homology 2 domain substitution modulates the kinase and transforming activities of the Fes protein-tyrosine kinase.
Rogers JA; Cheng HY; Smithgall TE
Cell Growth Differ; 2000 Nov; 11(11):581-92. PubMed ID: 11095247
[TBL] [Abstract][Full Text] [Related]
2. Regulation of the human c-fes protein tyrosine kinase (p93c-fes) by its src homology 2 domain and major autophosphorylation site (Tyr-713).
Hjermstad SJ; Peters KL; Briggs SD; Glazer RI; Smithgall TE
Oncogene; 1993 Aug; 8(8):2283-92. PubMed ID: 7687763
[TBL] [Abstract][Full Text] [Related]
3. Regulation of c-Fes tyrosine kinase activity by coiled-coil and SH2 domains: analysis with Saccharomyces cerevisiae.
Takashima Y; Delfino FJ; Engen JR; Superti-Furga G; Smithgall TE
Biochemistry; 2003 Apr; 42(12):3567-74. PubMed ID: 12653561
[TBL] [Abstract][Full Text] [Related]
4. The Csk homologous kinase, Chk, binds tyrosine phosphorylated paxillin in human blastic T cells.
Grgurevich S; Mikhael A; McVicar DW
Biochem Biophys Res Commun; 1999 Mar; 256(3):668-75. PubMed ID: 10080957
[TBL] [Abstract][Full Text] [Related]
5. Reactivation of host-dependent src kinase activity by co-expression with a heterologous tyrosine kinase.
Liebl EC; England LJ; Martin GS
Virology; 1993 Jul; 195(1):265-7. PubMed ID: 8317103
[TBL] [Abstract][Full Text] [Related]
6. Phosphorylation of GAP and GAP-associated proteins by transforming and mitogenic tyrosine kinases.
Ellis C; Moran M; McCormick F; Pawson T
Nature; 1990 Jan; 343(6256):377-81. PubMed ID: 1689011
[TBL] [Abstract][Full Text] [Related]
7. Activation of the focal adhesion kinase signaling pathway by structural alterations in the carboxyl-terminal region of c-Crk II.
Zvara A; Fajardo JE; Escalante M; Cotton G; Muir T; Kirsch KH; Birge RB
Oncogene; 2001 Feb; 20(8):951-61. PubMed ID: 11314030
[TBL] [Abstract][Full Text] [Related]
8. SH3-dependent stimulation of Src-family kinase autophosphorylation without tail release from the SH2 domain in vivo.
Lerner EC; Smithgall TE
Nat Struct Biol; 2002 May; 9(5):365-9. PubMed ID: 11976726
[TBL] [Abstract][Full Text] [Related]
9. Most of the substrates of oncogenic viral tyrosine protein kinases can be phosphorylated by cellular tyrosine protein kinases in normal cells.
Kamps MP; Sefton BM
Oncogene Res; 1988 Sep; 3(2):105-15. PubMed ID: 2465525
[TBL] [Abstract][Full Text] [Related]
10. Reduced phosphotyrosine binding by the v-Src SH2 domain is compatible with wild-type transformation.
Tian M; Martin GS
Oncogene; 1996 Feb; 12(4):727-34. PubMed ID: 8632894
[TBL] [Abstract][Full Text] [Related]
11. Phosphorylation of the ras GTPase-activating protein (GAP) by the p93c-fes protein-tyrosine kinase in vitro and formation of GAP-fes complexes via an SH2 domain-dependent mechanism.
Hjermstad SJ; Briggs SD; Smithgall TE
Biochemistry; 1993 Oct; 32(39):10519-25. PubMed ID: 7691175
[TBL] [Abstract][Full Text] [Related]
12. Intracellular targeting of pp60src expression: localization of v-src to adhesion plaques is sufficient to transform chicken embryo fibroblasts.
Liebl EC; Martin GS
Oncogene; 1992 Dec; 7(12):2417-28. PubMed ID: 1334249
[TBL] [Abstract][Full Text] [Related]
13. The human c-Fes tyrosine kinase binds tubulin and microtubules through separate domains and promotes microtubule assembly.
Laurent CE; Delfino FJ; Cheng HY; Smithgall TE
Mol Cell Biol; 2004 Nov; 24(21):9351-8. PubMed ID: 15485904
[TBL] [Abstract][Full Text] [Related]
14. Multiple SH2-mediated interactions in v-src-transformed cells.
Koch CA; Moran MF; Anderson D; Liu XQ; Mbamalu G; Pawson T
Mol Cell Biol; 1992 Mar; 12(3):1366-74. PubMed ID: 1545818
[TBL] [Abstract][Full Text] [Related]
15. Regulation of c-Fes tyrosine kinase and biological activities by N-terminal coiled-coil oligomerization domains.
Cheng H; Rogers JA; Dunham NA; Smithgall TE
Mol Cell Biol; 1999 Dec; 19(12):8335-43. PubMed ID: 10567558
[TBL] [Abstract][Full Text] [Related]
16. Phosphorylation and regulatory effects of the carboxy terminus of a Drosophila src homolog.
Kussick SJ; Cooper JA
Oncogene; 1992 Aug; 7(8):1577-86. PubMed ID: 1630818
[TBL] [Abstract][Full Text] [Related]
17. Autophosphorylation of KDR in the kinase domain is required for maximal VEGF-stimulated kinase activity and receptor internalization.
Dougher M; Terman BI
Oncogene; 1999 Feb; 18(8):1619-27. PubMed ID: 10102632
[TBL] [Abstract][Full Text] [Related]
18. Phosphorylation at Tyr-838 in the kinase domain of EphA8 modulates Fyn binding to the Tyr-615 site by enhancing tyrosine kinase activity.
Choi S; Park S
Oncogene; 1999 Sep; 18(39):5413-22. PubMed ID: 10498895
[TBL] [Abstract][Full Text] [Related]
19. Single point mutations in the SH2 domain impair the transforming potential of vav and fail to activate proto-vav.
Katzav S
Oncogene; 1993 Jul; 8(7):1757-63. PubMed ID: 8510922
[TBL] [Abstract][Full Text] [Related]
20. The c-Fes protein-tyrosine kinase suppresses cytokine-independent outgrowth of myeloid leukemia cells induced by Bcr-Abl.
Lionberger JM; Smithgall TE
Cancer Res; 2000 Feb; 60(4):1097-103. PubMed ID: 10706130
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
