BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

835 related articles for article (PubMed ID: 11138010)

  • 1. SGS1, the Saccharomyces cerevisiae homologue of BLM and WRN, suppresses genome instability and homeologous recombination.
    Myung K; Datta A; Chen C; Kolodner RD
    Nat Genet; 2001 Jan; 27(1):113-6. PubMed ID: 11138010
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Functions of RecQ family helicases: possible involvement of Bloom's and Werner's syndrome gene products in guarding genome integrity during DNA replication.
    Enomoto T
    J Biochem; 2001 Apr; 129(4):501-7. PubMed ID: 11275547
    [TBL] [Abstract][Full Text] [Related]  

  • 3. [Functional analysis of yeast homologue gene associated with human DNA helicase causative syndromes].
    Miyajima A
    Kokuritsu Iyakuhin Shokuhin Eisei Kenkyusho Hokoku; 2002; (120):53-74. PubMed ID: 12638184
    [TBL] [Abstract][Full Text] [Related]  

  • 4. SGS1, a homologue of the Bloom's and Werner's syndrome genes, is required for maintenance of genome stability in Saccharomyces cerevisiae.
    Watt PM; Hickson ID; Borts RH; Louis EJ
    Genetics; 1996 Nov; 144(3):935-45. PubMed ID: 8913739
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Homologous recombination is responsible for cell death in the absence of the Sgs1 and Srs2 helicases.
    Gangloff S; Soustelle C; Fabre F
    Nat Genet; 2000 Jun; 25(2):192-4. PubMed ID: 10835635
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Bloom's and Werner's syndrome genes suppress hyperrecombination in yeast sgs1 mutant: implication for genomic instability in human diseases.
    Yamagata K; Kato J; Shimamoto A; Goto M; Furuichi Y; Ikeda H
    Proc Natl Acad Sci U S A; 1998 Jul; 95(15):8733-8. PubMed ID: 9671747
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Bloom's syndrome gene suppresses premature ageing caused by Sgs1 deficiency in yeast.
    Heo SJ; Tatebayashi K; Ohsugi I; Shimamoto A; Furuichi Y; Ikeda H
    Genes Cells; 1999 Nov; 4(11):619-25. PubMed ID: 10620009
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Control of translocations between highly diverged genes by Sgs1, the Saccharomyces cerevisiae homolog of the Bloom's syndrome protein.
    Schmidt KH; Wu J; Kolodner RD
    Mol Cell Biol; 2006 Jul; 26(14):5406-20. PubMed ID: 16809776
    [TBL] [Abstract][Full Text] [Related]  

  • 9. WRN helicase defective in the premature aging disorder Werner syndrome genetically interacts with topoisomerase 3 and restores the top3 slow growth phenotype of sgs1 top3.
    Aggarwal M; Brosh RM
    Aging (Albany NY); 2009 Feb; 1(2):219-33. PubMed ID: 20157511
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The N-terminal region of Sgs1, which interacts with Top3, is required for complementation of MMS sensitivity and suppression of hyper-recombination in sgs1 disruptants.
    Ui A; Satoh Y; Onoda F; Miyajima A; Seki M; Enomoto T
    Mol Genet Genomics; 2001 Jul; 265(5):837-50. PubMed ID: 11523801
    [TBL] [Abstract][Full Text] [Related]  

  • 11. A radical solution to death.
    Klein HL
    Nat Genet; 2000 Jun; 25(2):132-4. PubMed ID: 10835619
    [No Abstract]   [Full Text] [Related]  

  • 12. Recombination-mediated lengthening of terminal telomeric repeats requires the Sgs1 DNA helicase.
    Cohen H; Sinclair DA
    Proc Natl Acad Sci U S A; 2001 Mar; 98(6):3174-9. PubMed ID: 11248051
    [TBL] [Abstract][Full Text] [Related]  

  • 13. A yeast gene, MGS1, encoding a DNA-dependent AAA(+) ATPase is required to maintain genome stability.
    Hishida T; Iwasaki H; Ohno T; Morishita T; Shinagawa H
    Proc Natl Acad Sci U S A; 2001 Jul; 98(15):8283-9. PubMed ID: 11459965
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Elevated incidence of loss of heterozygosity (LOH) in an sgs1 mutant of Saccharomyces cerevisiae: roles of yeast RecQ helicase in suppression of aneuploidy, interchromosomal rearrangement, and the simultaneous incidence of both events during mitotic growth.
    Ajima J; Umezu K; Maki H
    Mutat Res; 2002 Jul; 504(1-2):157-72. PubMed ID: 12106656
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The three-dimensional structure of the HRDC domain and implications for the Werner and Bloom syndrome proteins.
    Liu Z; Macias MJ; Bottomley MJ; Stier G; Linge JP; Nilges M; Bork P; Sattler M
    Structure; 1999 Dec; 7(12):1557-66. PubMed ID: 10647186
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Sgs1 truncations induce genome rearrangements but suppress detrimental effects of BLM overexpression in Saccharomyces cerevisiae.
    Mirzaei H; Syed S; Kennedy J; Schmidt KH
    J Mol Biol; 2011 Jan; 405(4):877-91. PubMed ID: 21111748
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Competition between the DNA unwinding and strand pairing activities of the Werner and Bloom syndrome proteins.
    Machwe A; Lozada EM; Xiao L; Orren DK
    BMC Mol Biol; 2006 Jan; 7():1. PubMed ID: 16412221
    [TBL] [Abstract][Full Text] [Related]  

  • 18. rqh1+, a fission yeast gene related to the Bloom's and Werner's syndrome genes, is required for reversible S phase arrest.
    Stewart E; Chapman CR; Al-Khodairy F; Carr AM; Enoch T
    EMBO J; 1997 May; 16(10):2682-92. PubMed ID: 9184215
    [TBL] [Abstract][Full Text] [Related]  

  • 19. DNA helicases, genomic instability, and human genetic disease.
    van Brabant AJ; Stan R; Ellis NA
    Annu Rev Genomics Hum Genet; 2000; 1():409-59. PubMed ID: 11701636
    [TBL] [Abstract][Full Text] [Related]  

  • 20. A recQ family DNA helicase gene from Aspergillus nidulans.
    Appleyard MV; McPheat WL; Stark MJ
    DNA Seq; 2000; 11(3-4):315-9. PubMed ID: 11092746
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 42.