273 related articles for article (PubMed ID: 11169525)
1. Abnormal vitamin B6 metabolism in alkaline phosphatase knock-out mice causes multiple abnormalities, but not the impaired bone mineralization.
Narisawa S; Wennberg C; Millán JL
J Pathol; 2001 Jan; 193(1):125-33. PubMed ID: 11169525
[TBL] [Abstract][Full Text] [Related]
2. Inactivation of two mouse alkaline phosphatase genes and establishment of a model of infantile hypophosphatasia.
Narisawa S; Fröhlander N; Millán JL
Dev Dyn; 1997 Mar; 208(3):432-46. PubMed ID: 9056646
[TBL] [Abstract][Full Text] [Related]
3. Functional characterization of osteoblasts and osteoclasts from alkaline phosphatase knockout mice.
Wennberg C; Hessle L; Lundberg P; Mauro S; Narisawa S; Lerner UH; Millán JL
J Bone Miner Res; 2000 Oct; 15(10):1879-88. PubMed ID: 11028439
[TBL] [Abstract][Full Text] [Related]
4. Mice lacking tissue non-specific alkaline phosphatase die from seizures due to defective metabolism of vitamin B-6.
Waymire KG; Mahuren JD; Jaje JM; Guilarte TR; Coburn SP; MacGregor GR
Nat Genet; 1995 Sep; 11(1):45-51. PubMed ID: 7550313
[TBL] [Abstract][Full Text] [Related]
5. Genetically Modified Mice for Studying TNAP Function.
Narisawa S
Subcell Biochem; 2015; 76():45-57. PubMed ID: 26219706
[TBL] [Abstract][Full Text] [Related]
6. The mechanism of mineralization and the role of alkaline phosphatase in health and disease.
Orimo H
J Nippon Med Sch; 2010 Feb; 77(1):4-12. PubMed ID: 20154452
[TBL] [Abstract][Full Text] [Related]
7. Vitamin B6 related epilepsy during childhood.
Wang HS; Kuo MF
Chang Gung Med J; 2007; 30(5):396-401. PubMed ID: 18062169
[TBL] [Abstract][Full Text] [Related]
8. Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene.
Baumgartner-Sigl S; Haberlandt E; Mumm S; Scholl-Bürgi S; Sergi C; Ryan L; Ericson KL; Whyte MP; Högler W
Bone; 2007 Jun; 40(6):1655-61. PubMed ID: 17395561
[TBL] [Abstract][Full Text] [Related]
9. Perinatal hypophosphatasia: tissue levels of vitamin B6 are unremarkable despite markedly increased circulating concentrations of pyridoxal-5'-phosphate. Evidence for an ectoenzyme role for tissue-nonspecific alkaline phosphatase.
Whyte MP; Mahuren JD; Fedde KN; Cole FS; McCabe ER; Coburn SP
J Clin Invest; 1988 Apr; 81(4):1234-9. PubMed ID: 3350970
[TBL] [Abstract][Full Text] [Related]
10. Multisystemic functions of alkaline phosphatases.
Buchet R; Millán JL; Magne D
Methods Mol Biol; 2013; 1053():27-51. PubMed ID: 23860646
[TBL] [Abstract][Full Text] [Related]
11. Kinetic characterization of hypophosphatasia mutations with physiological substrates.
Di Mauro S; Manes T; Hessle L; Kozlenkov A; Pizauro JM; Hoylaerts MF; Millán JL
J Bone Miner Res; 2002 Aug; 17(8):1383-91. PubMed ID: 12162492
[TBL] [Abstract][Full Text] [Related]
12. What Can We Learn About the Neural Functions of TNAP from Studies on Other Organs and Tissues?
Millán JL
Subcell Biochem; 2015; 76():155-66. PubMed ID: 26219711
[TBL] [Abstract][Full Text] [Related]
13. Tissue-nonspecific alkaline phosphatase deficiency causes abnormal craniofacial bone development in the Alpl(-/-) mouse model of infantile hypophosphatasia.
Liu J; Nam HK; Campbell C; Gasque KC; Millán JL; Hatch NE
Bone; 2014 Oct; 67():81-94. PubMed ID: 25014884
[TBL] [Abstract][Full Text] [Related]
14. Neurodevelopmental alterations and seizures developed by mouse model of infantile hypophosphatasia are associated with purinergic signalling deregulation.
Sebastián-Serrano Á; Engel T; de Diego-García L; Olivos-Oré LA; Arribas-Blázquez M; Martínez-Frailes C; Pérez-Díaz C; Millán JL; Artalejo AR; Miras-Portugal MT; Henshall DC; Díaz-Hernández M
Hum Mol Genet; 2016 Oct; 25(19):4143-4156. PubMed ID: 27466191
[TBL] [Abstract][Full Text] [Related]
15. Physiological role of alkaline phosphatase explored in hypophosphatasia.
Whyte MP
Ann N Y Acad Sci; 2010 Mar; 1192():190-200. PubMed ID: 20392236
[TBL] [Abstract][Full Text] [Related]
16. Orientation of mineral crystallites and mineral density during skeletal development in mice deficient in tissue nonspecific alkaline phosphatase.
Tesch W; Vandenbos T; Roschgr P; Fratzl-Zelman N; Klaushofer K; Beertsen W; Fratzl P
J Bone Miner Res; 2003 Jan; 18(1):117-25. PubMed ID: 12510812
[TBL] [Abstract][Full Text] [Related]
17. Bone mineralization-dependent craniosynostosis and craniofacial shape abnormalities in the mouse model of infantile hypophosphatasia.
Durussel J; Liu J; Campbell C; Nam HK; Hatch NE
Dev Dyn; 2016 Feb; 245(2):175-82. PubMed ID: 26605996
[TBL] [Abstract][Full Text] [Related]
18. Enzyme replacement for craniofacial skeletal defects and craniosynostosis in murine hypophosphatasia.
Liu J; Campbell C; Nam HK; Caron A; Yadav MC; Millán JL; Hatch NE
Bone; 2015 Sep; 78():203-11. PubMed ID: 25959417
[TBL] [Abstract][Full Text] [Related]
19. Improvement of the skeletal and dental hypophosphatasia phenotype in Alpl-/- mice by administration of soluble (non-targeted) chimeric alkaline phosphatase.
Gasque KC; Foster BL; Kuss P; Yadav MC; Liu J; Kiffer-Moreira T; van Elsas A; Hatch N; Somerman MJ; Millán JL
Bone; 2015 Mar; 72():137-47. PubMed ID: 25433339
[TBL] [Abstract][Full Text] [Related]
20. Tissue-nonspecific alkaline phosphatase and plasma cell membrane glycoprotein-1 are central antagonistic regulators of bone mineralization.
Hessle L; Johnson KA; Anderson HC; Narisawa S; Sali A; Goding JW; Terkeltaub R; Millan JL
Proc Natl Acad Sci U S A; 2002 Jul; 99(14):9445-9. PubMed ID: 12082181
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
