398 related articles for article (PubMed ID: 11257107)
21. [Preparation of the gene targeted knockout mice for human premature aging diseases, Werner syndrome, and Rothmund-Thomson syndrome caused by the mutation of DNA helicases].
Ichikawa K; Noda T; Furuichi Y
Nihon Yakurigaku Zasshi; 2002 Apr; 119(4):219-26. PubMed ID: 11979727
[TBL] [Abstract][Full Text] [Related]
22. Human RECQ helicases: roles in DNA metabolism, mutagenesis and cancer biology.
Monnat RJ
Semin Cancer Biol; 2010 Oct; 20(5):329-39. PubMed ID: 20934517
[TBL] [Abstract][Full Text] [Related]
23. Rising from the RecQ-age: the role of human RecQ helicases in genome maintenance.
Bohr VA
Trends Biochem Sci; 2008 Dec; 33(12):609-20. PubMed ID: 18926708
[TBL] [Abstract][Full Text] [Related]
24. Rothmund-Thomson syndrome and RECQL4 defect: splitting and lumping.
Larizza L; Magnani I; Roversi G
Cancer Lett; 2006 Jan; 232(1):107-20. PubMed ID: 16271439
[TBL] [Abstract][Full Text] [Related]
25. Molecular genetics of RecQ helicase disorders.
Hanada K; Hickson ID
Cell Mol Life Sci; 2007 Sep; 64(17):2306-22. PubMed ID: 17571213
[TBL] [Abstract][Full Text] [Related]
26. Homologous recombination is responsible for cell death in the absence of the Sgs1 and Srs2 helicases.
Gangloff S; Soustelle C; Fabre F
Nat Genet; 2000 Jun; 25(2):192-4. PubMed ID: 10835635
[TBL] [Abstract][Full Text] [Related]
27. Localization of the Bloom syndrome helicase to punctate nuclear structures and the nuclear matrix and regulation during the cell cycle: comparison with the Werner's syndrome helicase.
Gharibyan V; Youssoufian H
Mol Carcinog; 1999 Dec; 26(4):261-73. PubMed ID: 10569803
[TBL] [Abstract][Full Text] [Related]
28. Werner's syndrome protein (WRN) migrates Holliday junctions and co-localizes with RPA upon replication arrest.
Constantinou A; Tarsounas M; Karow JK; Brosh RM; Bohr VA; Hickson ID; West SC
EMBO Rep; 2000 Jul; 1(1):80-4. PubMed ID: 11256630
[TBL] [Abstract][Full Text] [Related]
29. Analysis of the DNA unwinding activity of RecQ family helicases.
Bachrati CZ; Hickson ID
Methods Enzymol; 2006; 409():86-100. PubMed ID: 16793396
[TBL] [Abstract][Full Text] [Related]
30. The Bloom's syndrome helicase: keeping cancer at bay.
Mohaghegh P; Hickson ID
Biologist (London); 2003 Feb; 50(1):29-33. PubMed ID: 12586954
[TBL] [Abstract][Full Text] [Related]
31. [Functional analysis of yeast homologue gene associated with human DNA helicase causative syndromes].
Miyajima A
Kokuritsu Iyakuhin Shokuhin Eisei Kenkyusho Hokoku; 2002; (120):53-74. PubMed ID: 12638184
[TBL] [Abstract][Full Text] [Related]
32. Competition between the DNA unwinding and strand pairing activities of the Werner and Bloom syndrome proteins.
Machwe A; Lozada EM; Xiao L; Orren DK
BMC Mol Biol; 2006 Jan; 7():1. PubMed ID: 16412221
[TBL] [Abstract][Full Text] [Related]
33. Molecular defect of RAPADILINO syndrome expands the phenotype spectrum of RECQL diseases.
Siitonen HA; Kopra O; Kääriäinen H; Haravuori H; Winter RM; Säämänen AM; Peltonen L; Kestilä M
Hum Mol Genet; 2003 Nov; 12(21):2837-44. PubMed ID: 12952869
[TBL] [Abstract][Full Text] [Related]
34. WRN, the protein deficient in Werner syndrome, plays a critical structural role in optimizing DNA repair.
Chen L; Huang S; Lee L; Davalos A; Schiestl RH; Campisi J; Oshima J
Aging Cell; 2003 Aug; 2(4):191-9. PubMed ID: 12934712
[TBL] [Abstract][Full Text] [Related]
35. Werner and Bloom helicases are involved in DNA repair in a complementary fashion.
Imamura O; Fujita K; Itoh C; Takeda S; Furuichi Y; Matsumoto T
Oncogene; 2002 Jan; 21(6):954-63. PubMed ID: 11840341
[TBL] [Abstract][Full Text] [Related]
36. Bloom's syndrome gene suppresses premature ageing caused by Sgs1 deficiency in yeast.
Heo SJ; Tatebayashi K; Ohsugi I; Shimamoto A; Furuichi Y; Ikeda H
Genes Cells; 1999 Nov; 4(11):619-25. PubMed ID: 10620009
[TBL] [Abstract][Full Text] [Related]
37. The Werner syndrome gene: the molecular basis of RecQ helicase-deficiency diseases.
Shen JC; Loeb LA
Trends Genet; 2000 May; 16(5):213-20. PubMed ID: 10782115
[TBL] [Abstract][Full Text] [Related]
38. Inhibition of the Bloom's and Werner's syndrome helicases by G-quadruplex interacting ligands.
Li JL; Harrison RJ; Reszka AP; Brosh RM; Bohr VA; Neidle S; Hickson ID
Biochemistry; 2001 Dec; 40(50):15194-202. PubMed ID: 11735402
[TBL] [Abstract][Full Text] [Related]
39. Possible anti-recombinogenic role of Bloom's syndrome helicase in double-strand break processing.
Onclercq-Delic R; Calsou P; Delteil C; Salles B; Papadopoulo D; Amor-Guéret M
Nucleic Acids Res; 2003 Nov; 31(21):6272-82. PubMed ID: 14576316
[TBL] [Abstract][Full Text] [Related]
40. The N-terminal region of Sgs1, which interacts with Top3, is required for complementation of MMS sensitivity and suppression of hyper-recombination in sgs1 disruptants.
Ui A; Satoh Y; Onoda F; Miyajima A; Seki M; Enomoto T
Mol Genet Genomics; 2001 Jul; 265(5):837-50. PubMed ID: 11523801
[TBL] [Abstract][Full Text] [Related]
[Previous] [Next] [New Search]
