These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
197 related articles for article (PubMed ID: 11356621)
1. Neutrophils are primary source of O2 radicals during reperfusion after prolonged myocardial ischemia. Duilio C; Ambrosio G; Kuppusamy P; DiPaula A; Becker LC; Zweier JL Am J Physiol Heart Circ Physiol; 2001 Jun; 280(6):H2649-57. PubMed ID: 11356621 [TBL] [Abstract][Full Text] [Related]
2. Factors modifying protective effect of anti-CD18 antibodies on myocardial reperfusion injury in dogs. Perez RG; Arai M; Richardson C; DiPaula A; Siu C; Matsumoto N; Hildreth JE; Mariscalco MM; Smith CW; Becker LC Am J Physiol; 1996 Jan; 270(1 Pt 2):H53-64. PubMed ID: 8769734 [TBL] [Abstract][Full Text] [Related]
3. An anti-CD18 antibody limits infarct size and preserves left ventricular function in dogs with ischemia and 48-hour reperfusion. Arai M; Lefer DJ; So T; DiPaula A; Aversano T; Becker LC J Am Coll Cardiol; 1996 Apr; 27(5):1278-85. PubMed ID: 8609356 [TBL] [Abstract][Full Text] [Related]
4. Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone. Bolli R; Patel BS; Jeroudi MO; Lai EK; McCay PB J Clin Invest; 1988 Aug; 82(2):476-85. PubMed ID: 2841353 [TBL] [Abstract][Full Text] [Related]
5. Effect of the platelet-activating factor antagonist RP 59227 (Tulopafant) on myocardial ischemia/reperfusion injury and neutrophil function. Auchampach JA; Pieper GM; Cavero I; Gross GJ Basic Res Cardiol; 1998 Oct; 93(5):361-71. PubMed ID: 9833148 [TBL] [Abstract][Full Text] [Related]
6. Evidence that mitochondrial respiration is a source of potentially toxic oxygen free radicals in intact rabbit hearts subjected to ischemia and reflow. Ambrosio G; Zweier JL; Duilio C; Kuppusamy P; Santoro G; Elia PP; Tritto I; Cirillo P; Condorelli M; Chiariello M J Biol Chem; 1993 Sep; 268(25):18532-41. PubMed ID: 8395507 [TBL] [Abstract][Full Text] [Related]
7. Ascorbyl free radical as a real-time marker of free radical generation in briefly ischemic and reperfused hearts. An electron paramagnetic resonance study. Sharma MK; Buettner GR; Spencer KT; Kerber RE Circ Res; 1994 Apr; 74(4):650-8. PubMed ID: 8137501 [TBL] [Abstract][Full Text] [Related]
8. Use of spin traps in intact animals undergoing myocardial ischemia/reperfusion: a new approach to assessing the role of oxygen radicals in myocardial "stunning". Bolli R; McCay PB Free Radic Res Commun; 1990; 9(3-6):169-80. PubMed ID: 2167254 [TBL] [Abstract][Full Text] [Related]
9. Transcardiac alteration of neutrophil function relates to myocardial ischaemia/reperfusion injury. Hoshida S; Kuzuya T; Fuji H; Oe H; Hori M; Kamada T; Tada M Cardiovasc Res; 1993 Mar; 27(3):377-83. PubMed ID: 8490937 [TBL] [Abstract][Full Text] [Related]
10. The nitric oxide donor S-nitroso-N-acetylpenicillamine (SNAP) increases free radical generation and degrades left ventricular function after myocardial ischemia-reperfusion. Zhang Y; Davies LR; Martin SM; Coddington WJ; Miller FJ; Buettner GR; Kerber RE Resuscitation; 2003 Dec; 59(3):345-52. PubMed ID: 14659604 [TBL] [Abstract][Full Text] [Related]
11. Role of neutrophils in radical production during ischemia and reperfusion of the rat brain: effect of neutrophil depletion on extracellular ascorbyl radical formation. Matsuo Y; Kihara T; Ikeda M; Ninomiya M; Onodera H; Kogure K J Cereb Blood Flow Metab; 1995 Nov; 15(6):941-7. PubMed ID: 7593354 [TBL] [Abstract][Full Text] [Related]
12. The relationship between oxygen radical generation and impairment of myocardial energy metabolism following post-ischemic reperfusion. Ambrosio G; Zweier JL; Flaherty JT J Mol Cell Cardiol; 1991 Dec; 23(12):1359-74. PubMed ID: 1811055 [TBL] [Abstract][Full Text] [Related]
13. Oxygen radicals trigger activation of NF-kappaB and AP-1 and upregulation of ICAM-1 in reperfused canine heart. Fan H; Sun B; Gu Q; Lafond-Walker A; Cao S; Becker LC Am J Physiol Heart Circ Physiol; 2002 May; 282(5):H1778-86. PubMed ID: 11959643 [TBL] [Abstract][Full Text] [Related]
14. Demonstration of free radical generation in the "stunned" myocardium in the conscious dog and identification of major differences between conscious and open-chest dogs. Li XY; McCay PB; Zughaib M; Jeroudi MO; Triana JF; Bolli R J Clin Invest; 1993 Aug; 92(2):1025-41. PubMed ID: 8394382 [TBL] [Abstract][Full Text] [Related]
15. Soluble complement receptor type 1 inhibits the complement pathway and prevents contractile failure in the postischemic heart. Evidence that complement activation is required for neutrophil-mediated reperfusion injury. Shandelya SM; Kuppusamy P; Herskowitz A; Weisfeldt ML; Zweier JL Circulation; 1993 Dec; 88(6):2812-26. PubMed ID: 8252695 [TBL] [Abstract][Full Text] [Related]
17. Deleterious effects of oxygen radicals in ischemia/reperfusion. Resolved and unresolved issues. Kloner RA; Przyklenk K; Whittaker P Circulation; 1989 Nov; 80(5):1115-27. PubMed ID: 2553296 [TBL] [Abstract][Full Text] [Related]
18. Superoxide anion production during reperfusion is reduced by an antineutrophil antibody after prolonged cerebral ischemia. Fabian RH; Kent TA Free Radic Biol Med; 1999 Feb; 26(3-4):355-61. PubMed ID: 9895227 [TBL] [Abstract][Full Text] [Related]
19. Inhibitory effect of a hydrophilic alpha-tocopherol analogue, MDL 74,405, on generation of free radicals in stunned myocardium in dogs. Tang XL; McCay PB; Sun JZ; Hartley CJ; Schleman M; Bolli R Free Radic Res; 1995 Apr; 22(4):293-302. PubMed ID: 7633559 [TBL] [Abstract][Full Text] [Related]
20. Oxidative stress during reperfusion of human hearts: potential sources of oxygen free radicals. Curello S; Ceconi C; de Giuli F; Panzali AF; Milanesi B; Calarco M; Pardini A; Marzollo P; Alfieri O; Messineo F Cardiovasc Res; 1995 Jan; 29(1):118-25. PubMed ID: 7895229 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]