These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

213 related articles for article (PubMed ID: 11399766)

  • 1. Functional interaction of p53 and BLM DNA helicase in apoptosis.
    Wang XW; Tseng A; Ellis NA; Spillare EA; Linke SP; Robles AI; Seker H; Yang Q; Hu P; Beresten S; Bemmels NA; Garfield S; Harris CC
    J Biol Chem; 2001 Aug; 276(35):32948-55. PubMed ID: 11399766
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Localization of the Bloom syndrome helicase to punctate nuclear structures and the nuclear matrix and regulation during the cell cycle: comparison with the Werner's syndrome helicase.
    Gharibyan V; Youssoufian H
    Mol Carcinog; 1999 Dec; 26(4):261-73. PubMed ID: 10569803
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Regulation and localization of the Bloom syndrome protein in response to DNA damage.
    Bischof O; Kim SH; Irving J; Beresten S; Ellis NA; Campisi J
    J Cell Biol; 2001 Apr; 153(2):367-80. PubMed ID: 11309417
    [TBL] [Abstract][Full Text] [Related]  

  • 4. The processing of Holliday junctions by BLM and WRN helicases is regulated by p53.
    Yang Q; Zhang R; Wang XW; Spillare EA; Linke SP; Subramanian D; Griffith JD; Li JL; Hickson ID; Shen JC; Loeb LA; Mazur SJ; Appella E; Brosh RM; Karmakar P; Bohr VA; Harris CC
    J Biol Chem; 2002 Aug; 277(35):31980-7. PubMed ID: 12080066
    [TBL] [Abstract][Full Text] [Related]  

  • 5. ATM-dependent phosphorylation and accumulation of endogenous BLM protein in response to ionizing radiation.
    Ababou M; Dutertre S; Lécluse Y; Onclercq R; Chatton B; Amor-Guéret M
    Oncogene; 2000 Dec; 19(52):5955-63. PubMed ID: 11146546
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Redundancy of DNA helicases in p53-mediated apoptosis.
    Spillare EA; Wang XW; von Kobbe C; Bohr VA; Hickson ID; Harris CC
    Oncogene; 2006 Mar; 25(14):2119-23. PubMed ID: 16288211
    [TBL] [Abstract][Full Text] [Related]  

  • 7. BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination.
    Sengupta S; Linke SP; Pedeux R; Yang Q; Farnsworth J; Garfield SH; Valerie K; Shay JW; Ellis NA; Wasylyk B; Harris CC
    EMBO J; 2003 Mar; 22(5):1210-22. PubMed ID: 12606585
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Evidence for BLM and Topoisomerase IIIalpha interaction in genomic stability.
    Hu P; Beresten SF; van Brabant AJ; Ye TZ; Pandolfi PP; Johnson FB; Guarente L; Ellis NA
    Hum Mol Genet; 2001 Jun; 10(12):1287-98. PubMed ID: 11406610
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Telomere and ribosomal DNA repeats are chromosomal targets of the bloom syndrome DNA helicase.
    Schawalder J; Paric E; Neff NF
    BMC Cell Biol; 2003 Oct; 4():15. PubMed ID: 14577841
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Bloom syndrome cells undergo p53-dependent apoptosis and delayed assembly of BRCA1 and NBS1 repair complexes at stalled replication forks.
    Davalos AR; Campisi J
    J Cell Biol; 2003 Sep; 162(7):1197-209. PubMed ID: 14517203
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Competition between the DNA unwinding and strand pairing activities of the Werner and Bloom syndrome proteins.
    Machwe A; Lozada EM; Xiao L; Orren DK
    BMC Mol Biol; 2006 Jan; 7():1. PubMed ID: 16412221
    [TBL] [Abstract][Full Text] [Related]  

  • 12. The Bloom syndrome protein interacts and cooperates with p53 in regulation of transcription and cell growth control.
    Garkavtsev IV; Kley N; Grigorian IA; Gudkov AV
    Oncogene; 2001 Dec; 20(57):8276-80. PubMed ID: 11781842
    [TBL] [Abstract][Full Text] [Related]  

  • 13. p53-mediated apoptosis is attenuated in Werner syndrome cells.
    Spillare EA; Robles AI; Wang XW; Shen JC; Yu CE; Schellenberg GD; Harris CC
    Genes Dev; 1999 Jun; 13(11):1355-60. PubMed ID: 10364153
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Structural basis of Bloom syndrome (BS) causing mutations in the BLM helicase domain.
    Rong SB; Väliaho J; Vihinen M
    Mol Med; 2000 Mar; 6(3):155-64. PubMed ID: 10965492
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Phosphorylation of the Bloom's syndrome helicase and its role in recovery from S-phase arrest.
    Davies SL; North PS; Dart A; Lakin ND; Hickson ID
    Mol Cell Biol; 2004 Feb; 24(3):1279-91. PubMed ID: 14729972
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Colocalization, physical, and functional interaction between Werner and Bloom syndrome proteins.
    von Kobbe C; Karmakar P; Dawut L; Opresko P; Zeng X; Brosh RM; Hickson ID; Bohr VA
    J Biol Chem; 2002 Jun; 277(24):22035-44. PubMed ID: 11919194
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Potential role for the BLM helicase in recombinational repair via a conserved interaction with RAD51.
    Wu L; Davies SL; Levitt NC; Hickson ID
    J Biol Chem; 2001 Jun; 276(22):19375-81. PubMed ID: 11278509
    [TBL] [Abstract][Full Text] [Related]  

  • 18. A role for PML and the nuclear body in genomic stability.
    Zhong S; Hu P; Ye TZ; Stan R; Ellis NA; Pandolfi PP
    Oncogene; 1999 Dec; 18(56):7941-7. PubMed ID: 10637504
    [TBL] [Abstract][Full Text] [Related]  

  • 19. BLM (the causative gene of Bloom syndrome) protein translocation into the nucleus by a nuclear localization signal.
    Kaneko H; Orii KO; Matsui E; Shimozawa N; Fukao T; Matsumoto T; Shimamoto A; Furuichi Y; Hayakawa S; Kasahara K; Kondo N
    Biochem Biophys Res Commun; 1997 Nov; 240(2):348-53. PubMed ID: 9388480
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Werner and Bloom helicases are involved in DNA repair in a complementary fashion.
    Imamura O; Fujita K; Itoh C; Takeda S; Furuichi Y; Matsumoto T
    Oncogene; 2002 Jan; 21(6):954-63. PubMed ID: 11840341
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.