BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

110 related articles for article (PubMed ID: 11451745)

  • 1. LDL-activated p38 in endothelial cells is mediated by Ras.
    Zhu Y; Liao H; Wang N; Ma KS; Verna LK; Shyy JY; Chien S; Stemerman MB
    Arterioscler Thromb Vasc Biol; 2001 Jul; 21(7):1159-64. PubMed ID: 11451745
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Tumor necrosis factor alpha-induced E-selectin expression is activated by the nuclear factor-kappaB and c-JUN N-terminal kinase/p38 mitogen-activated protein kinase pathways.
    Read MA; Whitley MZ; Gupta S; Pierce JW; Best J; Davis RJ; Collins T
    J Biol Chem; 1997 Jan; 272(5):2753-61. PubMed ID: 9006914
    [TBL] [Abstract][Full Text] [Related]  

  • 3. p38 Mitogen-activated protein kinase regulation of JB6 Cl41 cell transformation promoted by epidermal growth factor.
    He Z; Cho YY; Liu G; Ma WY; Bode AM; Dong Z
    J Biol Chem; 2003 Jul; 278(29):26435-42. PubMed ID: 12748197
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Retinoic acid inhibits induction of c-Jun protein by ultraviolet radiation that occurs subsequent to activation of mitogen-activated protein kinase pathways in human skin in vivo.
    Fisher GJ; Talwar HS; Lin J; Lin P; McPhillips F; Wang Z; Li X; Wan Y; Kang S; Voorhees JJ
    J Clin Invest; 1998 Mar; 101(6):1432-40. PubMed ID: 9502786
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Active, phosphorylation-dependent MAP kinases, MAPK/ERK, SAPK/JNK and p38, and specific transcription factor substrates are differentially expressed following systemic administration of kainic acid to the adult rat.
    Ferrer I; Blanco R; Carmona M; Puig B; Domínguez I; Viñals F
    Acta Neuropathol; 2002 Apr; 103(4):391-407. PubMed ID: 11904760
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Low-density lipoprotein activates Jun N-terminal kinase (JNK) in human endothelial cells.
    Zhu Y; Liao HL; Wang N; Friedli O; Verna L; Stemerman MB
    Biochim Biophys Acta; 1999 Jan; 1436(3):557-64. PubMed ID: 9989285
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Inhibition of the mitogen activated protein kinase, p38 alpha, prevents proinflammatory cytokine induction by human adherent mononuclear leukocytes in response to lipid loading.
    Feng Y; Schreiner GF; Chakravarty S; Liu DY; Joly AH
    Atherosclerosis; 2001 Oct; 158(2):331-8. PubMed ID: 11583711
    [TBL] [Abstract][Full Text] [Related]  

  • 8. p38/RK is essential for stress-induced nuclear responses: JNK/SAPKs and c-Jun/ATF-2 phosphorylation are insufficient.
    Hazzalin CA; Cano E; Cuenda A; Barratt MJ; Cohen P; Mahadevan LC
    Curr Biol; 1996 Aug; 6(8):1028-31. PubMed ID: 8805335
    [TBL] [Abstract][Full Text] [Related]  

  • 9. TNF initiates E-selectin transcription in human endothelial cells through parallel TRAF-NF-kappa B and TRAF-RAC/CDC42-JNK-c-Jun/ATF2 pathways.
    Min W; Pober JS
    J Immunol; 1997 Oct; 159(7):3508-18. PubMed ID: 9317150
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Helicobacter pylori VacA activates the p38/activating transcription factor 2-mediated signal pathway in AZ-521 cells.
    Nakayama M; Kimura M; Wada A; Yahiro K; Ogushi K; Niidome T; Fujikawa A; Shirasaka D; Aoyama N; Kurazono H; Noda M; Moss J; Hirayama T
    J Biol Chem; 2004 Feb; 279(8):7024-8. PubMed ID: 14630932
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Anti-apoptotic effect of quercetin: intervention in the JNK- and ERK-mediated apoptotic pathways.
    Ishikawa Y; Kitamura M
    Kidney Int; 2000 Sep; 58(3):1078-87. PubMed ID: 10972672
    [TBL] [Abstract][Full Text] [Related]  

  • 12. AP-1 and ATF-2 are constitutively activated via the JNK pathway in Theileria parva-transformed T-cells.
    Botteron C; Dobbelaere D
    Biochem Biophys Res Commun; 1998 May; 246(2):418-21. PubMed ID: 9610375
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Activation of cardiac c-Jun NH(2)-terminal kinases and p38-mitogen-activated protein kinases with abrupt changes in hemodynamic load.
    Fischer TA; Ludwig S; Flory E; Gambaryan S; Singh K; Finn P; Pfeffer MA; Kelly RA; Pfeffer JM
    Hypertension; 2001 May; 37(5):1222-8. PubMed ID: 11358932
    [TBL] [Abstract][Full Text] [Related]  

  • 14. High density lipoprotein-induced angiogenesis requires the activation of Ras/MAP kinase in human coronary artery endothelial cells.
    Miura S; Fujino M; Matsuo Y; Kawamura A; Tanigawa H; Nishikawa H; Saku K
    Arterioscler Thromb Vasc Biol; 2003 May; 23(5):802-8. PubMed ID: 12637339
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Activation of c-Jun N-terminal kinase (JNK) by widely used specific p38 MAPK inhibitors SB202190 and SB203580: a MLK-3-MKK7-dependent mechanism.
    Muniyappa H; Das KC
    Cell Signal; 2008 Apr; 20(4):675-83. PubMed ID: 18222647
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Role of mitogen-activated protein kinases in pulmonary endothelial cells exposed to cyclic strain.
    Kito H; Chen EL; Wang X; Ikeda M; Azuma N; Nakajima N; Gahtan V; Sumpio BE
    J Appl Physiol (1985); 2000 Dec; 89(6):2391-400. PubMed ID: 11090594
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Effects of transcription factor activator protein-1 on interleukin-8 expression and enteritis in response to Clostridium difficile toxin A.
    Lee JY; Park HR; Oh YK; Kim YJ; Youn J; Han JS; Kim JM
    J Mol Med (Berl); 2007 Dec; 85(12):1393-404. PubMed ID: 17639289
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Raf-independent deregulation of p38 and JNK mitogen-activated protein kinases are critical for Ras transformation.
    Pruitt K; Pruitt WM; Bilter GK; Westwick JK; Der CJ
    J Biol Chem; 2002 Aug; 277(35):31808-17. PubMed ID: 12082106
    [TBL] [Abstract][Full Text] [Related]  

  • 19. The role of JNK and p38 MAPK activities in UVA-induced signaling pathways leading to AP-1 activation and c-Fos expression.
    Silvers AL; Bachelor MA; Bowden GT
    Neoplasia; 2003; 5(4):319-29. PubMed ID: 14511403
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Differential expression of active, phosphorylation-dependent MAP kinases, MAPK/ERK, SAPK/JNK and p38, and specific transcription factor substrates following quinolinic acid excitotoxicity in the rat.
    Ferrer I; Blanco R; Carmona M
    Brain Res Mol Brain Res; 2001 Oct; 94(1-2):48-58. PubMed ID: 11597764
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 6.