These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

190 related articles for article (PubMed ID: 11583580)

  • 1. Survival-factor-induced phosphorylation of Bad results in its dissociation from Bcl-x(L) but not Bcl-2.
    Hirai I; Wang HG
    Biochem J; 2001 Oct; 359(Pt 2):345-52. PubMed ID: 11583580
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L).
    Zha J; Harada H; Yang E; Jockel J; Korsmeyer SJ
    Cell; 1996 Nov; 87(4):619-28. PubMed ID: 8929531
    [TBL] [Abstract][Full Text] [Related]  

  • 3. BAD Ser-155 phosphorylation regulates BAD/Bcl-XL interaction and cell survival.
    Tan Y; Demeter MR; Ruan H; Comb MJ
    J Biol Chem; 2000 Aug; 275(33):25865-9. PubMed ID: 10837486
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Growth factors inactivate the cell death promoter BAD by phosphorylation of its BH3 domain on Ser155.
    Zhou XM; Liu Y; Payne G; Lutz RJ; Chittenden T
    J Biol Chem; 2000 Aug; 275(32):25046-51. PubMed ID: 10837473
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Regulation of BAD by cAMP-dependent protein kinase is mediated via phosphorylation of a novel site, Ser155.
    Lizcano JM; Morrice N; Cohen P
    Biochem J; 2000 Jul; 349(Pt 2):547-57. PubMed ID: 10880354
    [TBL] [Abstract][Full Text] [Related]  

  • 6. p21-activated kinase 1 phosphorylates the death agonist bad and protects cells from apoptosis.
    Schürmann A; Mooney AF; Sanders LC; Sells MA; Wang HG; Reed JC; Bokoch GM
    Mol Cell Biol; 2000 Jan; 20(2):453-61. PubMed ID: 10611223
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Ca2+-induced apoptosis through calcineurin dephosphorylation of BAD.
    Wang HG; Pathan N; Ethell IM; Krajewski S; Yamaguchi Y; Shibasaki F; McKeon F; Bobo T; Franke TF; Reed JC
    Science; 1999 Apr; 284(5412):339-43. PubMed ID: 10195903
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Distinct domains of Bcl-XL are involved in Bax and Bad antagonism and in apoptosis inhibition.
    Zhou H; Hou Q; Chai Y; Hsu YT
    Exp Cell Res; 2005 Oct; 309(2):316-28. PubMed ID: 16061221
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Protein phosphatase 2A activates the proapoptotic function of BAD in interleukin- 3-dependent lymphoid cells by a mechanism requiring 14-3-3 dissociation.
    Chiang CW; Harris G; Ellig C; Masters SC; Subramanian R; Shenolikar S; Wadzinski BE; Yang E
    Blood; 2001 Mar; 97(5):1289-97. PubMed ID: 11222372
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Identification of a novel phosphorylation site, Ser-170, as a regulator of bad pro-apoptotic activity.
    Dramsi S; Scheid MP; Maiti A; Hojabrpour P; Chen X; Schubert K; Goodlett DR; Aebersold R; Duronio V
    J Biol Chem; 2002 Feb; 277(8):6399-405. PubMed ID: 11717309
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Bcl-x(L) sequesters its C-terminal membrane anchor in soluble, cytosolic homodimers.
    Jeong SY; Gaume B; Lee YJ; Hsu YT; Ryu SW; Yoon SH; Youle RJ
    EMBO J; 2004 May; 23(10):2146-55. PubMed ID: 15131699
    [TBL] [Abstract][Full Text] [Related]  

  • 12. 14-3-3 proteins and survival kinases cooperate to inactivate BAD by BH3 domain phosphorylation.
    Datta SR; Katsov A; Hu L; Petros A; Fesik SW; Yaffe MB; Greenberg ME
    Mol Cell; 2000 Jul; 6(1):41-51. PubMed ID: 10949026
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Hepatocyte growth factor promotes renal epithelial cell survival by dual mechanisms.
    Liu Y
    Am J Physiol; 1999 Oct; 277(4):F624-33. PubMed ID: 10516287
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Interleukin-7 inactivates the pro-apoptotic protein Bad promoting T cell survival.
    Li WQ; Jiang Q; Khaled AR; Keller JR; Durum SK
    J Biol Chem; 2004 Jul; 279(28):29160-6. PubMed ID: 15123689
    [TBL] [Abstract][Full Text] [Related]  

  • 15. BAD detects coincidence of G2/M phase and growth factor deprivation to regulate apoptosis.
    Hashimoto A; Hirose K; Iino M
    J Biol Chem; 2005 Jul; 280(28):26225-32. PubMed ID: 15901741
    [TBL] [Abstract][Full Text] [Related]  

  • 16. JNK suppresses apoptosis via phosphorylation of the proapoptotic Bcl-2 family protein BAD.
    Yu C; Minemoto Y; Zhang J; Liu J; Tang F; Bui TN; Xiang J; Lin A
    Mol Cell; 2004 Feb; 13(3):329-40. PubMed ID: 14967141
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Caspase cleavage enhances the apoptosis-inducing effects of BAD.
    Condorelli F; Salomoni P; Cotteret S; Cesi V; Srinivasula SM; Alnemri ES; Calabretta B
    Mol Cell Biol; 2001 May; 21(9):3025-36. PubMed ID: 11287608
    [TBL] [Abstract][Full Text] [Related]  

  • 18. High Bcr-Abl expression prevents the translocation of Bax and Bad to the mitochondrion.
    Keeshan K; Cotter TG; McKenna SL
    Leukemia; 2002 Sep; 16(9):1725-34. PubMed ID: 12200687
    [TBL] [Abstract][Full Text] [Related]  

  • 19. p21-activated Kinase 1 (Pak1)-dependent phosphorylation of Raf-1 regulates its mitochondrial localization, phosphorylation of BAD, and Bcl-2 association.
    Jin S; Zhuo Y; Guo W; Field J
    J Biol Chem; 2005 Jul; 280(26):24698-705. PubMed ID: 15849194
    [TBL] [Abstract][Full Text] [Related]  

  • 20. BAD/BCL-[X(L)] heterodimerization leads to bypass of G0/G1 arrest.
    Chattopadhyay A; Chiang CW; Yang E
    Oncogene; 2001 Jul; 20(33):4507-18. PubMed ID: 11494146
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.