459 related articles for article (PubMed ID: 11585247)
1. Copper chaperone for superoxide dismutase co-aggregates with superoxide dismutase 1 (SOD1) in neuronal Lewy body-like hyaline inclusions: an immunohistochemical study on familial amyotrophic lateral sclerosis with SOD1 gene mutation.
Kato S; Sumi-Akamaru H; Fujimura H; Sakoda S; Kato M; Hirano A; Takikawa M; Ohama E
Acta Neuropathol; 2001 Sep; 102(3):233-8. PubMed ID: 11585247
[TBL] [Abstract][Full Text] [Related]
2. Colocalization of 14-3-3 proteins with SOD1 in Lewy body-like hyaline inclusions in familial amyotrophic lateral sclerosis cases and the animal model.
Okamoto Y; Shirakashi Y; Ihara M; Urushitani M; Oono M; Kawamoto Y; Yamashita H; Shimohama S; Kato S; Hirano A; Tomimoto H; Ito H; Takahashi R
PLoS One; 2011; 6(5):e20427. PubMed ID: 21655264
[TBL] [Abstract][Full Text] [Related]
3. Histological evidence of redox system breakdown caused by superoxide dismutase 1 (SOD1) aggregation is common to SOD1-mutated motor neurons in humans and animal models.
Kato S; Saeki Y; Aoki M; Nagai M; Ishigaki A; Itoyama Y; Kato M; Asayama K; Awaya A; Hirano A; Ohama E
Acta Neuropathol; 2004 Feb; 107(2):149-58. PubMed ID: 14648077
[TBL] [Abstract][Full Text] [Related]
4. Familial amyotrophic lateral sclerosis with His46Arg mutation in Cu/Zn superoxide dismutase presenting characteristic clinical features and Lewy body-like hyaline inclusions.
Ohi T; Nabeshima K; Kato S; Yazawa S; Takechi S
J Neurol Sci; 2004 Oct; 225(1-2):19-25. PubMed ID: 15465081
[TBL] [Abstract][Full Text] [Related]
5. Recent advances in research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutations: neuronal Lewy body-like hyaline inclusions and astrocytic hyaline inclusions.
Kato S; Saito M; Hirano A; Ohama E
Histol Histopathol; 1999 Jul; 14(3):973-89. PubMed ID: 10425565
[TBL] [Abstract][Full Text] [Related]
6. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
Watanabe M; Dykes-Hoberg M; Culotta VC; Price DL; Wong PC; Rothstein JD
Neurobiol Dis; 2001 Dec; 8(6):933-41. PubMed ID: 11741389
[TBL] [Abstract][Full Text] [Related]
7. Astrocytic hyaline inclusions contain advanced glycation endproducts in familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutation: immunohistochemical and immunoelectron microscopical analyses.
Kato S; Horiuchi S; Nakashima K; Hirano A; Shibata N; Nakano I; Saito M; Kato M; Asayama K; Ohama E
Acta Neuropathol; 1999 Mar; 97(3):260-6. PubMed ID: 10090673
[TBL] [Abstract][Full Text] [Related]
8. Inverse correlation between the formation of mitochondria-derived vacuoles and Lewy-body-like hyaline inclusions in G93A superoxide-dismutase-transgenic mice.
Sumi H; Nagano S; Fujimura H; Kato S; Sakoda S
Acta Neuropathol; 2006 Jul; 112(1):52-63. PubMed ID: 16642310
[TBL] [Abstract][Full Text] [Related]
9. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
Tan CF; Eguchi H; Tagawa A; Onodera O; Iwasaki T; Tsujino A; Nishizawa M; Kakita A; Takahashi H
Acta Neuropathol; 2007 May; 113(5):535-42. PubMed ID: 17333220
[TBL] [Abstract][Full Text] [Related]
10. Familial amyotrophic lateral sclerosis with Cys111Tyr mutation in Cu/Zn superoxide dismutase showing widespread Lewy body-like hyaline inclusions.
Suzuki M; Yasui K; Ishikawai H; Nomura M; Watanabe T; Mikami H; Yamano T; Ono S
J Neurol Sci; 2011 Jan; 300(1-2):182-4. PubMed ID: 20888599
[TBL] [Abstract][Full Text] [Related]
11. 14-3-3 proteins in Lewy body-like hyaline inclusions in a patient with familial amyotrophic lateral sclerosis with a two-base pair deletion in the Cu/Zn superoxide dismutase (SOD1) gene.
Kawamoto Y; Akiguchi I; Fujimura H; Shirakashi Y; Honjo Y; Sakoda S
Acta Neuropathol; 2005 Aug; 110(2):203-4. PubMed ID: 15973542
[No Abstract] [Full Text] [Related]
12. HtrA2/Omi-immunoreactive intraneuronal inclusions in the anterior horn of patients with sporadic and Cu/Zn superoxide dismutase (SOD1) mutant amyotrophic lateral sclerosis.
Kawamoto Y; Ito H; Kobayashi Y; Suzuki Y; Akiguchi I; Fujimura H; Sakoda S; Kusaka H; Hirano A; Takahashi R
Neuropathol Appl Neurobiol; 2010 Jun; 36(4):331-44. PubMed ID: 20202124
[TBL] [Abstract][Full Text] [Related]
13. Advanced glycation endproducts are deposited in neuronal hyaline inclusions: a study on familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation.
Shibata N; Hirano A; Kato S; Nagai R; Horiuchi S; Komori T; Umahara T; Asayama K; Kobayashi M
Acta Neuropathol; 1999 Mar; 97(3):240-6. PubMed ID: 10090670
[TBL] [Abstract][Full Text] [Related]
14. Familial amyotrophic lateral sclerosis with a two base pair deletion in superoxide dismutase 1: gene multisystem degeneration with intracytoplasmic hyaline inclusions in astrocytes.
Kato S; Shimoda M; Watanabe Y; Nakashima K; Takahashi K; Ohama E
J Neuropathol Exp Neurol; 1996 Oct; 55(10):1089-101. PubMed ID: 8858006
[TBL] [Abstract][Full Text] [Related]
15. Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation.
Kato S; Nakashima K; Horiuchi S; Nagai R; Cleveland DW; Liu J; Hirano A; Takikawa M; Kato M; Nakano I; Sakoda S; Asayama K; Ohama E
Neuropathology; 2001 Mar; 21(1):67-81. PubMed ID: 11304045
[TBL] [Abstract][Full Text] [Related]
16. Familial amyotrophic lateral sclerosis with widespread vacuolation and hyaline inclusions.
Sasaki S; Ohsawa Y; Yamane K; Sakuma H; Shibata N; Nakano R; Kikugawa K; Mizutani T; Tsuji S; Iwata M
Neurology; 1998 Sep; 51(3):871-3. PubMed ID: 9748044
[TBL] [Abstract][Full Text] [Related]
17. Advanced glycation endproduct-modified superoxide dismutase-1 (SOD1)-positive inclusions are common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutations and transgenic mice expressing human SOD1 with a G85R mutation.
Kato S; Horiuchi S; Liu J; Cleveland DW; Shibata N; Nakashima K; Nagai R; Hirano A; Takikawa M; Kato M; Nakano I; Ohama E
Acta Neuropathol; 2000 Nov; 100(5):490-505. PubMed ID: 11045671
[TBL] [Abstract][Full Text] [Related]
18. Immunohistochemical study on superoxide dismutases in spinal cords from autopsied patients with amyotrophic lateral sclerosis.
Shibata N; Asayama K; Hirano A; Kobayashi M
Dev Neurosci; 1996; 18(5-6):492-8. PubMed ID: 8940623
[TBL] [Abstract][Full Text] [Related]
19. An in vitro model for Lewy body-like hyaline inclusion/astrocytic hyaline inclusion: induction by ER stress with an ALS-linked SOD1 mutation.
Yamagishi S; Koyama Y; Katayama T; Taniguchi M; Hitomi J; Kato M; Aoki M; Itoyama Y; Kato S; Tohyama M
PLoS One; 2007 Oct; 2(10):e1030. PubMed ID: 17925878
[TBL] [Abstract][Full Text] [Related]
20. Expression of hepatocyte growth factor and c-Met in the anterior horn cells of the spinal cord in the patients with amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS and familial ALS with superoxide dismutase 1 gene mutation.
Kato S; Funakoshi H; Nakamura T; Kato M; Nakano I; Hirano A; Ohama E
Acta Neuropathol; 2003 Aug; 106(2):112-20. PubMed ID: 12707786
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
