BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

179 related articles for article (PubMed ID: 11704863)

  • 1. Enhanced sensitivity to apoptosis in Ras-transformed thyroid cells.
    Cheng G; Meinkoth JL
    Oncogene; 2001 Nov; 20(50):7334-41. PubMed ID: 11704863
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Oncogenic Ras, but not (V600E)B-RAF, protects from cholesterol depletion-induced apoptosis through the PI3K/AKT pathway in colorectal cancer cells.
    Calleros L; Sánchez-Hernández I; Baquero P; Toro MJ; Chiloeches A
    Carcinogenesis; 2009 Oct; 30(10):1670-7. PubMed ID: 19700418
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Essential role of Raf in Ras transformation and deregulation of matrix metalloproteinase expression in ovarian epithelial cells.
    Ulkü AS; Schäfer R; Der CJ
    Mol Cancer Res; 2003 Dec; 1(14):1077-88. PubMed ID: 14707291
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Regulation of myeloid cell growth by distinct effectors of Ras.
    Matsuguchi T; Kraft AS
    Oncogene; 1998 Nov; 17(21):2701-9. PubMed ID: 9840934
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Differential sensitivity of normal and H-ras oncogene-transformed rat kidney epithelial cells to okadaic acid-induced apoptosis.
    Davis MA; Chang SH; Trump BF
    Toxicol Appl Pharmacol; 1996 Nov; 141(1):93-101. PubMed ID: 8917680
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Modulation of phospholipase D by Ras proteins mediated by its effectors Ral-GDS, PI3K and Raf-1.
    Lucas L; Penalva V; Ramírez de Molina A; Del Peso L; Lacal JC
    Int J Oncol; 2002 Sep; 21(3):477-85. PubMed ID: 12168089
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Regulation of choline kinase activity by Ras proteins involves Ral-GDS and PI3K.
    Ramírez de Molina A; Penalva V; Lucas L; Lacal JC
    Oncogene; 2002 Jan; 21(6):937-46. PubMed ID: 11840339
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Role of cAMP, PKA and Rap1A in thyroid follicular cell survival.
    Saavedra AP; Tsygankova OM; Prendergast GV; Dworet JH; Cheng G; Meinkoth JL
    Oncogene; 2002 Jan; 21(5):778-88. PubMed ID: 11850806
    [TBL] [Abstract][Full Text] [Related]  

  • 9. A modest reduction in c-myc expression has minimal effects on cell growth and apoptosis but dramatically reduces susceptibility to Ras and Raf transformation.
    Bazarov AV; Adachi S; Li SF; Mateyak MK; Wei S; Sedivy JM
    Cancer Res; 2001 Feb; 61(3):1178-86. PubMed ID: 11221849
    [TBL] [Abstract][Full Text] [Related]  

  • 10. PI-3-kinase is an essential anti-apoptotic effector in the proliferative response of primary human epithelial cells to mutant RAS.
    Gire V; Marshall C; Wynford-Thomas D
    Oncogene; 2000 May; 19(19):2269-76. PubMed ID: 10822377
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Ras mediates radioresistance through both phosphatidylinositol 3-kinase-dependent and Raf-dependent but mitogen-activated protein kinase/extracellular signal-regulated kinase kinase-independent signaling pathways.
    Grana TM; Rusyn EV; Zhou H; Sartor CI; Cox AD
    Cancer Res; 2002 Jul; 62(14):4142-50. PubMed ID: 12124353
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Analysis of Ras transformation of human thyroid epithelial cells.
    Poghosyan Z; Wynford-Thomas D
    Methods Enzymol; 2006; 407():648-60. PubMed ID: 16757359
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Induction of p202, a modulator of apoptosis, during oncogenic transformation of NIH 3T3 cells by activated H-Ras (Q61L) contributes to cell survival.
    Xin H; Geng Y; Pramanik R; Choubey D
    J Cell Biochem; 2003 Jan; 88(1):191-204. PubMed ID: 12461788
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Mutant RAS selectively promotes sensitivity of myeloid leukemia cells to apoptosis by a protein kinase C-dependent process.
    Gallagher AP; Burnett AK; Bowen DT; Darley RL
    Cancer Res; 1998 May; 58(9):2029-35. PubMed ID: 9581849
    [TBL] [Abstract][Full Text] [Related]  

  • 15. NF-kappa B is required for H-ras oncogene induced abnormal cell proliferation and tumorigenesis.
    Jo H; Zhang R; Zhang H; McKinsey TA; Shao J; Beauchamp RD; Ballard DW; Liang P
    Oncogene; 2000 Feb; 19(7):841-9. PubMed ID: 10702792
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Ras signaling through PI3K confers hormone-independent proliferation that is compatible with differentiation.
    Cass LA; Meinkoth JL
    Oncogene; 2000 Feb; 19(7):924-32. PubMed ID: 10702801
    [TBL] [Abstract][Full Text] [Related]  

  • 17. TGF-beta1 (transforming growth factor-beta1)-mediated adhesion of gastric carcinoma cells involves a decrease in Ras/ERKs (extracellular-signal-regulated kinases) cascade activity dependent on c-Src activity.
    Kim HP; Lee MS; Yu J; Park JA; Jong HS; Kim TY; Lee JW; Bang YJ
    Biochem J; 2004 Apr; 379(Pt 1):141-50. PubMed ID: 14720123
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Multiple aspects of the phenotype of mammary epithelial cells transformed by expression of activated M-Ras depend on an autocrine mechanism mediated by hepatocyte growth factor/scatter factor.
    Zhang KX; Ward KR; Schrader JW
    Mol Cancer Res; 2004 Apr; 2(4):242-55. PubMed ID: 15140946
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter.
    Kukushkin AN; Abramova MV; Svetlikova SB; Darieva ZA; Pospelova TV; Pospelov VA
    Oncogene; 2002 Jan; 21(5):719-30. PubMed ID: 11850800
    [TBL] [Abstract][Full Text] [Related]  

  • 20. [Mutations of growth factor receptor Flt3 in acute myeloid leukemia: transformation of myeloid cells by Ras-dependent and Ras-independent mechanisms].
    Müller-Tidow C; Steur C; Mizuki M; Schwäble J; Brandts C; Berdel WE; Serve H
    Dtsch Med Wochenschr; 2002 Oct; 127(42):2195-200. PubMed ID: 12397548
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.