206 related articles for article (PubMed ID: 11717399)
1. Switch junction sequences in PMS2-deficient mice reveal a microhomology-mediated mechanism of Ig class switch recombination.
Ehrenstein MR; Rada C; Jones AM; Milstein C; Neuberger MS
Proc Natl Acad Sci U S A; 2001 Dec; 98(25):14553-8. PubMed ID: 11717399
[TBL] [Abstract][Full Text] [Related]
2. Role for mismatch repair proteins Msh2, Mlh1, and Pms2 in immunoglobulin class switching shown by sequence analysis of recombination junctions.
Schrader CE; Vardo J; Stavnezer J
J Exp Med; 2002 Feb; 195(3):367-73. PubMed ID: 11828012
[TBL] [Abstract][Full Text] [Related]
3. Mlh1 can function in antibody class switch recombination independently of Msh2.
Schrader CE; Vardo J; Stavnezer J
J Exp Med; 2003 May; 197(10):1377-83. PubMed ID: 12743174
[TBL] [Abstract][Full Text] [Related]
4. Deficiency in Msh2 affects the efficiency and local sequence specificity of immunoglobulin class-switch recombination: parallels with somatic hypermutation.
Ehrenstein MR; Neuberger MS
EMBO J; 1999 Jun; 18(12):3484-90. PubMed ID: 10369687
[TBL] [Abstract][Full Text] [Related]
5. Human PMS2 deficiency is associated with impaired immunoglobulin class switch recombination.
Péron S; Metin A; Gardès P; Alyanakian MA; Sheridan E; Kratz CP; Fischer A; Durandy A
J Exp Med; 2008 Oct; 205(11):2465-72. PubMed ID: 18824584
[TBL] [Abstract][Full Text] [Related]
6. Evidence for the lack of mismatch-repair directed antirecombination during mouse meiosis.
Qin J; Baker S; Te Riele H; Liskay RM; Arnheim N
J Hered; 2002; 93(3):201-5. PubMed ID: 12195036
[TBL] [Abstract][Full Text] [Related]
7. Mutagenesis in PMS2- and MSH2-deficient mice indicates differential protection from transversions and frameshifts.
Andrew SE; Xu XS; Baross-Francis A; Narayanan L; Milhausen K; Liskay RM; Jirik FR; Glazer PM
Carcinogenesis; 2000 Jul; 21(7):1291-5. PubMed ID: 10874005
[TBL] [Abstract][Full Text] [Related]
8. Contributions by MutL homologues Mlh3 and Pms2 to DNA mismatch repair and tumor suppression in the mouse.
Chen PC; Dudley S; Hagen W; Dizon D; Paxton L; Reichow D; Yoon SR; Yang K; Arnheim N; Liskay RM; Lipkin SM
Cancer Res; 2005 Oct; 65(19):8662-70. PubMed ID: 16204034
[TBL] [Abstract][Full Text] [Related]
9. Molecular characterization of the spectrum of genomic deletions in the mismatch repair genes MSH2, MLH1, MSH6, and PMS2 responsible for hereditary nonpolyposis colorectal cancer (HNPCC).
van der Klift H; Wijnen J; Wagner A; Verkuilen P; Tops C; Otway R; Kohonen-Corish M; Vasen H; Oliani C; Barana D; Moller P; Delozier-Blanchet C; Hutter P; Foulkes W; Lynch H; Burn J; Möslein G; Fodde R
Genes Chromosomes Cancer; 2005 Oct; 44(2):123-38. PubMed ID: 15942939
[TBL] [Abstract][Full Text] [Related]
10. Msh2 ATPase activity is essential for somatic hypermutation at a-T basepairs and for efficient class switch recombination.
Martin A; Li Z; Lin DP; Bardwell PD; Iglesias-Ussel MD; Edelmann W; Scharff MD
J Exp Med; 2003 Oct; 198(8):1171-8. PubMed ID: 14568978
[TBL] [Abstract][Full Text] [Related]
11. Alterations in PMS2, MSH2 and MLH1 expression in human prostate cancer.
Chen Y; Wang J; Fraig MM; Henderson K; Bissada NK; Watson DK; Schweinfest CW
Int J Oncol; 2003 May; 22(5):1033-43. PubMed ID: 12684669
[TBL] [Abstract][Full Text] [Related]
12. Differing patterns of genetic instability in mice deficient in the mismatch repair genes Pms2, Mlh1, Msh2, Msh3 and Msh6.
Hegan DC; Narayanan L; Jirik FR; Edelmann W; Liskay RM; Glazer PM
Carcinogenesis; 2006 Dec; 27(12):2402-8. PubMed ID: 16728433
[TBL] [Abstract][Full Text] [Related]
13. Mismatch repair proteins MSH2, MLH1, and EXO1 are important for class-switch recombination events occurring in B cells that lack nonhomologous end joining.
Eccleston J; Yan C; Yuan K; Alt FW; Selsing E
J Immunol; 2011 Feb; 186(4):2336-43. PubMed ID: 21242524
[TBL] [Abstract][Full Text] [Related]
14. Reduced isotype switching in splenic B cells from mice deficient in mismatch repair enzymes.
Schrader CE; Edelmann W; Kucherlapati R; Stavnezer J
J Exp Med; 1999 Aug; 190(3):323-30. PubMed ID: 10430621
[TBL] [Abstract][Full Text] [Related]
15. Pms2 is a genetic enhancer of trinucleotide CAG.CTG repeat somatic mosaicism: implications for the mechanism of triplet repeat expansion.
Gomes-Pereira M; Fortune MT; Ingram L; McAbney JP; Monckton DG
Hum Mol Genet; 2004 Aug; 13(16):1815-25. PubMed ID: 15198993
[TBL] [Abstract][Full Text] [Related]
16. Class switch recombination efficiency and junction microhomology patterns in Msh2-, Mlh1-, and Exo1-deficient mice depend on the presence of mu switch region tandem repeats.
Eccleston J; Schrader CE; Yuan K; Stavnezer J; Selsing E
J Immunol; 2009 Jul; 183(2):1222-8. PubMed ID: 19553545
[TBL] [Abstract][Full Text] [Related]
17. Mismatch repair gene PMS2: disease-causing germline mutations are frequent in patients whose tumors stain negative for PMS2 protein, but paralogous genes obscure mutation detection and interpretation.
Nakagawa H; Lockman JC; Frankel WL; Hampel H; Steenblock K; Burgart LJ; Thibodeau SN; de la Chapelle A
Cancer Res; 2004 Jul; 64(14):4721-7. PubMed ID: 15256438
[TBL] [Abstract][Full Text] [Related]
18. Altered spectra of hypermutation in antibodies from mice deficient for the DNA mismatch repair protein PMS2.
Winter DB; Phung QH; Umar A; Baker SM; Tarone RE; Tanaka K; Liskay RM; Kunkel TA; Bohr VA; Gearhart PJ
Proc Natl Acad Sci U S A; 1998 Jun; 95(12):6953-8. PubMed ID: 9618520
[TBL] [Abstract][Full Text] [Related]
19. Polymorphisms in a pseudogene highly homologous to PMS2.
Chadwick RB; Meek JE; Prior TW; Peltomaki P; de La Chapelle A
Hum Mutat; 2000 Dec; 16(6):530. PubMed ID: 11102987
[TBL] [Abstract][Full Text] [Related]
20. Shifts in targeting of class switch recombination sites in mice that lack mu switch region tandem repeats or Msh2.
Min IM; Rothlein LR; Schrader CE; Stavnezer J; Selsing E
J Exp Med; 2005 Jun; 201(12):1885-90. PubMed ID: 15955838
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
