These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

165 related articles for article (PubMed ID: 12011096)

  • 1. NCS-1 inhibits insulin-stimulated GLUT4 translocation in 3T3L1 adipocytes through a phosphatidylinositol 4-kinase-dependent pathway.
    Mora S; Durham PL; Smith JR; Russo AF; Jeromin A; Pessin JE
    J Biol Chem; 2002 Jul; 277(30):27494-500. PubMed ID: 12011096
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Protein kinase B phosphorylation of PIKfyve regulates the trafficking of GLUT4 vesicles.
    Berwick DC; Dell GC; Welsh GI; Heesom KJ; Hers I; Fletcher LM; Cooke FT; Tavaré JM
    J Cell Sci; 2004 Dec; 117(Pt 25):5985-93. PubMed ID: 15546921
    [TBL] [Abstract][Full Text] [Related]  

  • 3. The role of Ca2+ in insulin-stimulated glucose transport in 3T3-L1 cells.
    Whitehead JP; Molero JC; Clark S; Martin S; Meneilly G; James DE
    J Biol Chem; 2001 Jul; 276(30):27816-24. PubMed ID: 11375387
    [TBL] [Abstract][Full Text] [Related]  

  • 4. An inhibitor of p38 mitogen-activated protein kinase prevents insulin-stimulated glucose transport but not glucose transporter translocation in 3T3-L1 adipocytes and L6 myotubes.
    Sweeney G; Somwar R; Ramlal T; Volchuk A; Ueyama A; Klip A
    J Biol Chem; 1999 Apr; 274(15):10071-8. PubMed ID: 10187787
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Protein kinase B stimulates the translocation of GLUT4 but not GLUT1 or transferrin receptors in 3T3-L1 adipocytes by a pathway involving SNAP-23, synaptobrevin-2, and/or cellubrevin.
    Foran PG; Fletcher LM; Oatey PB; Mohammed N; Dolly JO; Tavaré JM
    J Biol Chem; 1999 Oct; 274(40):28087-95. PubMed ID: 10497159
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Calmodulin antagonists inhibit insulin-stimulated GLUT4 (glucose transporter 4) translocation by preventing the formation of phosphatidylinositol 3,4,5-trisphosphate in 3T3L1 adipocytes.
    Yang C; Watson RT; Elmendorf JS; Sacks DB; Pessin JE
    Mol Endocrinol; 2000 Feb; 14(2):317-26. PubMed ID: 10674403
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Osmotic shock stimulates GLUT4 translocation in 3T3L1 adipocytes by a novel tyrosine kinase pathway.
    Chen D; Elmendorf JS; Olson AL; Li X; Earp HS; Pessin JE
    J Biol Chem; 1997 Oct; 272(43):27401-10. PubMed ID: 9341192
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Functional consequence of targeting protein kinase B/Akt to GLUT4 vesicles.
    Ducluzeau PH; Fletcher LM; Welsh GI; Tavaré JM
    J Cell Sci; 2002 Jul; 115(Pt 14):2857-66. PubMed ID: 12082147
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Separation of insulin signaling into distinct GLUT4 translocation and activation steps.
    Funaki M; Randhawa P; Janmey PA
    Mol Cell Biol; 2004 Sep; 24(17):7567-77. PubMed ID: 15314166
    [TBL] [Abstract][Full Text] [Related]  

  • 10. A role for protein kinase Bbeta/Akt2 in insulin-stimulated GLUT4 translocation in adipocytes.
    Hill MM; Clark SF; Tucker DF; Birnbaum MJ; James DE; Macaulay SL
    Mol Cell Biol; 1999 Nov; 19(11):7771-81. PubMed ID: 10523666
    [TBL] [Abstract][Full Text] [Related]  

  • 11. The trimeric GTP-binding protein (G(q)/G(11)) alpha subunit is required for insulin-stimulated GLUT4 translocation in 3T3L1 adipocytes.
    Kanzaki M; Watson RT; Artemyev NO; Pessin JE
    J Biol Chem; 2000 Mar; 275(10):7167-75. PubMed ID: 10702285
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Activation of the mammalian target of rapamycin pathway acutely inhibits insulin signaling to Akt and glucose transport in 3T3-L1 and human adipocytes.
    Tremblay F; Gagnon A; Veilleux A; Sorisky A; Marette A
    Endocrinology; 2005 Mar; 146(3):1328-37. PubMed ID: 15576463
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Regulation of insulin-stimulated glucose transporter GLUT4 translocation and Akt kinase activity by ceramide.
    Summers SA; Garza LA; Zhou H; Birnbaum MJ
    Mol Cell Biol; 1998 Sep; 18(9):5457-64. PubMed ID: 9710629
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Essential role of insulin receptor substrate-2 in insulin stimulation of Glut4 translocation and glucose uptake in brown adipocytes.
    Fasshauer M; Klein J; Ueki K; Kriauciunas KM; Benito M; White MF; Kahn CR
    J Biol Chem; 2000 Aug; 275(33):25494-501. PubMed ID: 10829031
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Insulin-stimulated GLUT4 translocation requires the CAP-dependent activation of TC10.
    Chiang SH; Baumann CA; Kanzaki M; Thurmond DC; Watson RT; Neudauer CL; Macara IG; Pessin JE; Saltiel AR
    Nature; 2001 Apr; 410(6831):944-8. PubMed ID: 11309621
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Akt activation is required at a late stage of insulin-induced GLUT4 translocation to the plasma membrane.
    van Dam EM; Govers R; James DE
    Mol Endocrinol; 2005 Apr; 19(4):1067-77. PubMed ID: 15650020
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Akt mediates insulin induction of glucose uptake and up-regulation of GLUT4 gene expression in brown adipocytes.
    Hernandez R; Teruel T; Lorenzo M
    FEBS Lett; 2001 Apr; 494(3):225-31. PubMed ID: 11311245
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Oxidative stress disrupts insulin-induced cellular redistribution of insulin receptor substrate-1 and phosphatidylinositol 3-kinase in 3T3-L1 adipocytes. A putative cellular mechanism for impaired protein kinase B activation and GLUT4 translocation.
    Tirosh A; Potashnik R; Bashan N; Rudich A
    J Biol Chem; 1999 Apr; 274(15):10595-602. PubMed ID: 10187855
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Chronic endothelin-1 treatment leads to heterologous desensitization of insulin signaling in 3T3-L1 adipocytes.
    Ishibashi KI; Imamura T; Sharma PM; Huang J; Ugi S; Olefsky JM
    J Clin Invest; 2001 May; 107(9):1193-202. PubMed ID: 11342583
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Osmotic shock inhibits insulin signaling by maintaining Akt/protein kinase B in an inactive dephosphorylated state.
    Chen D; Fucini RV; Olson AL; Hemmings BA; Pessin JE
    Mol Cell Biol; 1999 Jul; 19(7):4684-94. PubMed ID: 10373517
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.