148 related articles for article (PubMed ID: 12016140)
1. Morphology and morphometric investigation of hepatocellular preneoplastic lesions and neoplasms in connexin32-deficient mice.
Evert M; Ott T; Temme A; Willecke K; Dombrowski F
Carcinogenesis; 2002 May; 23(5):697-703. PubMed ID: 12016140
[TBL] [Abstract][Full Text] [Related]
2. High incidence of spontaneous and chemically induced liver tumors in mice deficient for connexin32.
Temme A; Buchmann A; Gabriel HD; Nelles E; Schwarz M; Willecke K
Curr Biol; 1997 Sep; 7(9):713-6. PubMed ID: 9285723
[TBL] [Abstract][Full Text] [Related]
3. Ablation of gap junctional communication in hepatocytes of transgenic mice does not lead to disrupted cellular homeostasis or increased spontaneous tumourigenesis.
Ott T; Jokwitz M; Lenhard D; Romualdi A; Dombrowski F; Ittrich C; Schwarz M; Willecke K
Eur J Cell Biol; 2006 Aug; 85(8):717-28. PubMed ID: 16740338
[TBL] [Abstract][Full Text] [Related]
4. Lack of phenobarbital-mediated promotion of hepatocarcinogenesis in connexin32-null mice.
Moennikes O; Buchmann A; Romualdi A; Ott T; Werringloer J; Willecke K; Schwarz M
Cancer Res; 2000 Sep; 60(18):5087-91. PubMed ID: 11016633
[TBL] [Abstract][Full Text] [Related]
5. Multiple mechanisms are responsible for altered expression of gap junction genes during oncogenesis in rat liver.
Neveu MJ; Hully JR; Babcock KL; Hertzberg EL; Nicholson BJ; Paul DL; Pitot HC
J Cell Sci; 1994 Jan; 107 ( Pt 1)():83-95. PubMed ID: 8175925
[TBL] [Abstract][Full Text] [Related]
6. Transgenic disruption of gap junctional intercellular communication enhances early but not late stage hepatocarcinogenesis in the rat.
Hokaiwado N; Asamoto M; Ogawa K; Shirai T
Toxicol Pathol; 2005; 33(6):695-701. PubMed ID: 16243774
[TBL] [Abstract][Full Text] [Related]
7. The effect of connexin32 null mutation on hepatocarcinogenesis in different mouse strains.
Moennikes O; Buchmann A; Ott T; Willecke1 K; Schwarz M
Carcinogenesis; 1999 Jul; 20(7):1379-82. PubMed ID: 10383916
[TBL] [Abstract][Full Text] [Related]
8. Rapid development of hepatic tumors in transforming growth factor alpha transgenic mice associated with increased cell proliferation in precancerous hepatocellular lesions initiated by N-nitrosodiethylamine and promoted by phenobarbital.
Tamano S; Merlino GT; Ward JM
Carcinogenesis; 1994 Sep; 15(9):1791-8. PubMed ID: 7923571
[TBL] [Abstract][Full Text] [Related]
9. Mice deficient for the gap junction protein Connexin32 exhibit increased radiation-induced tumorigenesis associated with elevated mitogen-activated protein kinase (p44/Erk1, p42/Erk2) activation.
King TJ; Lampe PD
Carcinogenesis; 2004 May; 25(5):669-80. PubMed ID: 14742325
[TBL] [Abstract][Full Text] [Related]
10. WY-14,643-mediated promotion of hepatocarcinogenesis in connexin32-wild-type and connexin32-null mice.
Moennikes O; Stahl S; Bannasch P; Buchmann A; Schwarz M
Carcinogenesis; 2003 Sep; 24(9):1561-5. PubMed ID: 12819187
[TBL] [Abstract][Full Text] [Related]
11. Age-dependent carcinogenic susceptibility in rat liver is related to potential of gap junctional intercellular communication.
Naiki-Ito A; Kato H; Asamoto M; Naiki T; Shirai T
Toxicol Pathol; 2012 Jul; 40(5):715-21. PubMed ID: 22569583
[TBL] [Abstract][Full Text] [Related]
12. Sustained hepatic expression of FoxM1B in transgenic mice has minimal effects on hepatocellular carcinoma development but increases cell proliferation rates in preneoplastic and early neoplastic lesions.
Kalinina OA; Kalinin SA; Polack EW; Mikaelian I; Panda S; Costa RH; Adami GR
Oncogene; 2003 Sep; 22(40):6266-76. PubMed ID: 13679865
[TBL] [Abstract][Full Text] [Related]
13. Preneoplastic and neoplastic progression during hepatocarcinogenesis in mice injected with diethylnitrosamine in infancy.
Goldfarb S; Pugh TD; Koen H; He YZ
Environ Health Perspect; 1983 Apr; 50():149-61. PubMed ID: 6873010
[TBL] [Abstract][Full Text] [Related]
14. Cell proliferation and regulation of negative growth factors in mouse liver foci.
Moser GJ; Wolf DC; Harden R; Standeven AM; Mills J; Jirtle RL; Goldsworthy TL
Carcinogenesis; 1996 Sep; 17(9):1835-40. PubMed ID: 8824503
[TBL] [Abstract][Full Text] [Related]
15. Dissimilar patterns of promotion by di(2-ethylhexyl)phthalate and phenobarbital of hepatocellular neoplasia initiated by diethylnitrosamine in B6C3F1 mice.
Ward JM; Rice JM; Creasia D; Lynch P; Riggs C
Carcinogenesis; 1983 Aug; 4(8):1021-9. PubMed ID: 6872148
[TBL] [Abstract][Full Text] [Related]
16. Both early and late stages of hepatocarcinogenesis are enhanced in Cx32 dominant negative mutant transgenic rats with disrupted gap junctional intercellular communication.
Hokaiwado N; Asamoto M; Futakuchi M; Ogawa K; Takahashi S; Shirai T
J Membr Biol; 2007 Aug; 218(1-3):101-6. PubMed ID: 17978847
[TBL] [Abstract][Full Text] [Related]
17. Dose-related induction of hepatic preneoplastic lesions by diethylnitrosamine in C57BL/6 mice.
Kushida M; Kamendulis LM; Peat TJ; Klaunig JE
Toxicol Pathol; 2011 Aug; 39(5):776-86. PubMed ID: 21628716
[TBL] [Abstract][Full Text] [Related]
18. Colocalized alterations in connexin32 and cytochrome P450IIB1/2 by phenobarbital and related liver tumor promoters.
Neveu MJ; Babcock KL; Hertzberg EL; Paul DL; Nicholson BJ; Pitot HC
Cancer Res; 1994 Jun; 54(12):3145-52. PubMed ID: 8205533
[TBL] [Abstract][Full Text] [Related]
19. Production of liver preneoplasia and gallbladder agenesis in turkey fetuses administered diethylnitrosamine.
Williams GM; Brunnemann KD; Iatropoulos MJ; Smart DJ; Enzmann HG
Arch Toxicol; 2011 Jun; 85(6):681-7. PubMed ID: 20981410
[TBL] [Abstract][Full Text] [Related]
20. Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver.
Dagli ML; Yamasaki H; Krutovskikh V; Omori Y
Carcinogenesis; 2004 Apr; 25(4):483-92. PubMed ID: 14688024
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
