These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

90 related articles for article (PubMed ID: 12028057)

  • 1. Two new molecular bases for the Dombrock null phenotype.
    Rios M; Storry JR; Hue-Roye K; Chung A; Reid ME
    Br J Haematol; 2002 Jun; 117(3):765-7. PubMed ID: 12028057
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Molecular basis of the Dombrock null phenotype.
    Rios M; Hue-Roye K; Storry JR; Lee T; Miller JL; Reid ME
    Transfusion; 2001 Nov; 41(11):1405-7. PubMed ID: 11724986
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Insights into the Holley- and Joseph- phenotypes.
    Rios M; Hue-Roye K; Øyen R; Miller J; Reid ME
    Transfusion; 2002 Jan; 42(1):52-8. PubMed ID: 11896313
    [TBL] [Abstract][Full Text] [Related]  

  • 4. A novel DO null allele with a c.268C>T (p.Gln90Stop) mutation in Japanese.
    Onodera T; Tsuneyama H; Ogasawara K; Isa K; Satake M; Tadokoro K; Uchikawa M
    Vox Sang; 2015 Aug; 109(2):191-3. PubMed ID: 25865759
    [TBL] [Abstract][Full Text] [Related]  

  • 5. New antigen in the Dombrock blood group system, DOYA, ablates expression of Do(a) and weakens expression of Hy, Jo(a), and Gy(a) antigens.
    Mayer B; Thornton N; Yürek S; Wylie D; Hue-Roye K; Poole J; Bartolmäs T; Salama A; Lomas-Francis C; Velliquette RW; Yazdanbakhsh K; Reid ME
    Transfusion; 2010 Jun; 50(6):1295-302. PubMed ID: 20088839
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Short deletion within the blood group Dombrock locus causing a Do(null) phenotype.
    Lucien N; Celton JL; Le Pennec PY; Cartron JP; Bailly P
    Blood; 2002 Aug; 100(3):1063-4. PubMed ID: 12130524
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Complexities of the Dombrock blood group system revealed.
    Reid ME
    Transfusion; 2005 Aug; 45(2 Suppl):92S-9S. PubMed ID: 16086795
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Evidence that the Gya, Hy and Joa antigens belong to the Dombrock blood group system.
    Banks JA; Hemming N; Poole J
    Vox Sang; 1995; 68(3):177-82. PubMed ID: 7625075
    [TBL] [Abstract][Full Text] [Related]  

  • 9. A DOB allele encoding an amino acid substitution (Phe62Ser) resulting in a Dombrock null phenotype.
    Westhoff C; Vege S; Yazdanbakhsh K; Wylie D; Razib M; Hue-Roye K; Halverson G; Read S; Whiteoak E; Nickle P; Maurer J; Kavitsky D; Nance S; Reid ME
    Transfusion; 2007 Aug; 47(8):1356-62. PubMed ID: 17655578
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Absence of DOMR, a new antigen in the Dombrock blood group system that weakens expression of Do(b) , Gy(a) , Hy, Jo(a) , and DOYA antigens.
    Costa FP; Hue-Roye K; Sausais L; Velliquette RW; Da Costa Ferreira E; Lomas-Francis C; Reid ME
    Transfusion; 2010 Sep; 50(9):2026-31. PubMed ID: 20412531
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Molecular studies of DO alleles reveal that JO is more prevalent than HY in Brazil, whereas HY is more prevalent in New York.
    Castilho L; Baleotti W; Tossas E; Hue-Roye K; Ribeiro KR; Lomas-Francis C; Charles-Pierre D; Reid ME
    Immunohematology; 2008; 24(4):135-7. PubMed ID: 19856715
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Novel molecular basis of an Inab phenotype.
    Hue-Roye K; Powell VI; Patel G; Lane D; Maguire M; Chung A; Reid ME
    Immunohematology; 2005; 21(2):53-5. PubMed ID: 15954804
    [TBL] [Abstract][Full Text] [Related]  

  • 13. A novel nonsense mutation in CEP290 induces exon skipping and leads to a relatively mild retinal phenotype.
    Littink KW; Pott JW; Collin RW; Kroes HY; Verheij JB; Blokland EA; de Castro Miró M; Hoyng CB; Klaver CC; Koenekoop RK; Rohrschneider K; Cremers FP; van den Born LI; den Hollander AI
    Invest Ophthalmol Vis Sci; 2010 Jul; 51(7):3646-52. PubMed ID: 20130272
    [TBL] [Abstract][Full Text] [Related]  

  • 14. The Dombrock blood group system: a review.
    Lomas-Francis C; Reid ME
    Immunohematology; 2010; 26(2):71-8. PubMed ID: 20932078
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Two novel null alleles of the KEL gene detected in two Chinese women with the K(null) phenotype.
    Yang Y; Wang L; Wang C; Chen H; Guo Z; Zhang Y; Zhu Z
    Transfus Med; 2009 Oct; 19(5):235-44. PubMed ID: 19747286
    [TBL] [Abstract][Full Text] [Related]  

  • 16. DNA analysis for the Dombrock polymorphism.
    Rios M; Hue-Roye K; Lee AH; Chiofolo JT; Miller JL; Reid ME
    Transfusion; 2001 Sep; 41(9):1143-6. PubMed ID: 11552072
    [TBL] [Abstract][Full Text] [Related]  

  • 17. An AQP1 null allele in an Indian woman with Co(a-b-) phenotype and high-titer anti-Co3 associated with mild HDN.
    Joshi SR; Wagner FF; Vasantha K; Panjwani SR; Flegel WA
    Transfusion; 2001 Oct; 41(10):1273-8. PubMed ID: 11606828
    [TBL] [Abstract][Full Text] [Related]  

  • 18. A new ABCG2 null allele with a 27-kb deletion including the promoter region causing the Jr(a-) phenotype.
    Ogasawara K; Osabe T; Suzuki Y; Tsuneyama H; Isa K; Kawai M; Obara K; Ogiyama Y; Ito S; Uchikawa M; Satake M; Tadokoro K
    Transfusion; 2015 Jun; 55(6 Pt 2):1467-71. PubMed ID: 25522810
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Three new high-prevalence antigens in the Cromer blood group system.
    Hue-Roye K; Lomas-Francis C; Belaygorod L; Lublin DM; Barnes J; Chung A; Fung-Kee-Fung K; Kinney J; Goldman-Lavi R; Yahalom V; Poole J; Ivankovic Z; Alcantara D; Bekavac M; Cepulic BG; Velliquette RW; Mason R; Reid ME
    Transfusion; 2007 Sep; 47(9):1621-9. PubMed ID: 17725726
    [TBL] [Abstract][Full Text] [Related]  

  • 20. SCER and SCAN: two novel high-prevalence antigens in the Scianna blood group system.
    Flegel WA; Chen Q; Reid ME; Martin J; Orsini LA; Poole J; Moulds MK; Wagner FF
    Transfusion; 2005 Dec; 45(12):1940-4. PubMed ID: 16371048
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 5.