165 related articles for article (PubMed ID: 12401444)
1. Proteasomal inhibition-induced inclusion formation and death in cortical neurons require transcription and ubiquitination.
Rideout HJ; Stefanis L
Mol Cell Neurosci; 2002 Oct; 21(2):223-38. PubMed ID: 12401444
[TBL] [Abstract][Full Text] [Related]
2. Cyclin-dependent kinase activity is required for apoptotic death but not inclusion formation in cortical neurons after proteasomal inhibition.
Rideout HJ; Wang Q; Park DS; Stefanis L
J Neurosci; 2003 Feb; 23(4):1237-45. PubMed ID: 12598612
[TBL] [Abstract][Full Text] [Related]
3. Lack of p53 delays apoptosis, but increases ubiquitinated inclusions, in proteasomal inhibitor-treated cultured cortical neurons.
Dietrich P; Rideout HJ; Wang Q; Stefanis L
Mol Cell Neurosci; 2003 Oct; 24(2):430-41. PubMed ID: 14572464
[TBL] [Abstract][Full Text] [Related]
4. Proteasomal inhibition leads to formation of ubiquitin/alpha-synuclein-immunoreactive inclusions in PC12 cells.
Rideout HJ; Larsen KE; Sulzer D; Stefanis L
J Neurochem; 2001 Aug; 78(4):899-908. PubMed ID: 11520910
[TBL] [Abstract][Full Text] [Related]
5. Alpha-synuclein-enhanced green fluorescent protein fusion proteins form proteasome sensitive inclusions in primary neurons.
McLean PJ; Kawamata H; Hyman BT
Neuroscience; 2001; 104(3):901-12. PubMed ID: 11440819
[TBL] [Abstract][Full Text] [Related]
6. alpha-synuclein is required for the fibrillar nature of ubiquitinated inclusions induced by proteasomal inhibition in primary neurons.
Rideout HJ; Dietrich P; Wang Q; Dauer WT; Stefanis L
J Biol Chem; 2004 Nov; 279(45):46915-20. PubMed ID: 15322100
[TBL] [Abstract][Full Text] [Related]
7. Synphilin-1 degradation by the ubiquitin-proteasome pathway and effects on cell survival.
Lee G; Junn E; Tanaka M; Kim YM; Mouradian MM
J Neurochem; 2002 Oct; 83(2):346-52. PubMed ID: 12423244
[TBL] [Abstract][Full Text] [Related]
8. Impairment of the ubiquitin-proteasome system causes dopaminergic cell death and inclusion body formation in ventral mesencephalic cultures.
McNaught KS; Mytilineou C; Jnobaptiste R; Yabut J; Shashidharan P; Jennert P; Olanow CW
J Neurochem; 2002 Apr; 81(2):301-6. PubMed ID: 12064477
[TBL] [Abstract][Full Text] [Related]
9. Expression of A53T mutant but not wild-type alpha-synuclein in PC12 cells induces alterations of the ubiquitin-dependent degradation system, loss of dopamine release, and autophagic cell death.
Stefanis L; Larsen KE; Rideout HJ; Sulzer D; Greene LA
J Neurosci; 2001 Dec; 21(24):9549-60. PubMed ID: 11739566
[TBL] [Abstract][Full Text] [Related]
10. Dopaminergic neurons in rat ventral midbrain cultures undergo selective apoptosis and form inclusions, but do not up-regulate iHSP70, following proteasomal inhibition.
Rideout HJ; Lang-Rollin IC; Savalle M; Stefanis L
J Neurochem; 2005 Jun; 93(5):1304-13. PubMed ID: 15934949
[TBL] [Abstract][Full Text] [Related]
11. Inhibition of proteasomal activity causes inclusion formation in neuronal and non-neuronal cells overexpressing Parkin.
Ardley HC; Scott GB; Rose SA; Tan NG; Markham AF; Robinson PA
Mol Biol Cell; 2003 Nov; 14(11):4541-56. PubMed ID: 12937272
[TBL] [Abstract][Full Text] [Related]
12. Proteasome mediates dopaminergic neuronal degeneration, and its inhibition causes alpha-synuclein inclusions.
Sawada H; Kohno R; Kihara T; Izumi Y; Sakka N; Ibi M; Nakanishi M; Nakamizo T; Yamakawa K; Shibasaki H; Yamamoto N; Akaike A; Inden M; Kitamura Y; Taniguchi T; Shimohama S
J Biol Chem; 2004 Mar; 279(11):10710-9. PubMed ID: 14672949
[TBL] [Abstract][Full Text] [Related]
13. UCH-L1 aggresome formation in response to proteasome impairment indicates a role in inclusion formation in Parkinson's disease.
Ardley HC; Scott GB; Rose SA; Tan NG; Robinson PA
J Neurochem; 2004 Jul; 90(2):379-91. PubMed ID: 15228595
[TBL] [Abstract][Full Text] [Related]
14. Aggregated and monomeric alpha-synuclein bind to the S6' proteasomal protein and inhibit proteasomal function.
Snyder H; Mensah K; Theisler C; Lee J; Matouschek A; Wolozin B
J Biol Chem; 2003 Apr; 278(14):11753-9. PubMed ID: 12551928
[TBL] [Abstract][Full Text] [Related]
15. Accumulation of mutant huntingtin fragments in aggresome-like inclusion bodies as a result of insufficient protein degradation.
Waelter S; Boeddrich A; Lurz R; Scherzinger E; Lueder G; Lehrach H; Wanker EE
Mol Biol Cell; 2001 May; 12(5):1393-407. PubMed ID: 11359930
[TBL] [Abstract][Full Text] [Related]
16. Application of proteasomal inhibitors to mouse sympathetic neurons activates the intrinsic apoptotic pathway.
Lang-Rollin I; Vekrellis K; Wang Q; Rideout HJ; Stefanis L
J Neurochem; 2004 Sep; 90(6):1511-20. PubMed ID: 15341534
[TBL] [Abstract][Full Text] [Related]
17. Identification and functional characterization of a novel R621C mutation in the synphilin-1 gene in Parkinson's disease.
Marx FP; Holzmann C; Strauss KM; Li L; Eberhardt O; Gerhardt E; Cookson MR; Hernandez D; Farrer MJ; Kachergus J; Engelender S; Ross CA; Berger K; Schöls L; Schulz JB; Riess O; Krüger R
Hum Mol Genet; 2003 Jun; 12(11):1223-31. PubMed ID: 12761037
[TBL] [Abstract][Full Text] [Related]
18. Aggresomes formed by alpha-synuclein and synphilin-1 are cytoprotective.
Tanaka M; Kim YM; Lee G; Junn E; Iwatsubo T; Mouradian MM
J Biol Chem; 2004 Feb; 279(6):4625-31. PubMed ID: 14627698
[TBL] [Abstract][Full Text] [Related]
19. alpha-synuclein metabolism and aggregation is linked to ubiquitin-independent degradation by the proteasome.
Tofaris GK; Layfield R; Spillantini MG
FEBS Lett; 2001 Nov; 509(1):22-6. PubMed ID: 11734199
[TBL] [Abstract][Full Text] [Related]
20. Involvement of macroautophagy in the dissolution of neuronal inclusions.
Rideout HJ; Lang-Rollin I; Stefanis L
Int J Biochem Cell Biol; 2004 Dec; 36(12):2551-62. PubMed ID: 15325592
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
