221 related articles for article (PubMed ID: 12435411)
1. NMDA-evoked excitotoxicity increases tissue transglutaminase in cerebellar granule cells.
Ientile R; Caccamo D; Macaione V; Torre V; Macaione S
Neuroscience; 2002; 115(3):723-9. PubMed ID: 12435411
[TBL] [Abstract][Full Text] [Related]
2. Excitotoxin-induced changes in transglutaminase during differentiation of cerebellar granule cells.
Caccamo D; Currò M; Cusumano G; Crisafulli G; Ientile R
Amino Acids; 2004 Mar; 26(2):197-201. PubMed ID: 15042450
[TBL] [Abstract][Full Text] [Related]
3. N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.
Urushitani M; Nakamizo T; Inoue R; Sawada H; Kihara T; Honda K; Akaike A; Shimohama S
J Neurosci Res; 2001 Mar; 63(5):377-87. PubMed ID: 11223912
[TBL] [Abstract][Full Text] [Related]
4. Excitotoxic and post-ischemic neurodegeneration: Involvement of transglutaminases.
Caccamo D; Campisi A; Currò M; Li Volti G; Vanella A; Ientile R
Amino Acids; 2004 Dec; 27(3-4):373-9. PubMed ID: 15365907
[TBL] [Abstract][Full Text] [Related]
5. Protection by cholesterol-extracting cyclodextrins: a role for N-methyl-D-aspartate receptor redistribution.
Abulrob A; Tauskela JS; Mealing G; Brunette E; Faid K; Stanimirovic D
J Neurochem; 2005 Mar; 92(6):1477-86. PubMed ID: 15748165
[TBL] [Abstract][Full Text] [Related]
6. Excitotoxic death induced by released glutamate in depolarized primary cultures of mouse cerebellar granule cells is dependent on GABAA receptors and niflumic acid-sensitive chloride channels.
Babot Z; Cristòfol R; Suñol C
Eur J Neurosci; 2005 Jan; 21(1):103-12. PubMed ID: 15654847
[TBL] [Abstract][Full Text] [Related]
7. Domoic acid neurotoxicity in cultured cerebellar granule neurons is controlled preferentially by the NMDA receptor Ca(2+) influx pathway.
Berman FW; LePage KT; Murray TF
Brain Res; 2002 Jan; 924(1):20-9. PubMed ID: 11743991
[TBL] [Abstract][Full Text] [Related]
8. NMDA receptor-dependent CREB activation in survival of cerebellar granule cells during in vivo and in vitro development.
Monti B; Marri L; Contestabile A
Eur J Neurosci; 2002 Oct; 16(8):1490-8. PubMed ID: 12405962
[TBL] [Abstract][Full Text] [Related]
9. Long-term exposure to dieldrin reduces gamma-aminobutyric acid type A and N-methyl-D-aspartate receptor function in primary cultures of mouse cerebellar granule cells.
Babot Z; Vilaró MT; Suñol C
J Neurosci Res; 2007 Dec; 85(16):3687-95. PubMed ID: 17663462
[TBL] [Abstract][Full Text] [Related]
10. Leptin enhances NR2B-mediated N-methyl-D-aspartate responses via a mitogen-activated protein kinase-dependent process in cerebellar granule cells.
Irving AJ; Wallace L; Durakoglugil D; Harvey J
Neuroscience; 2006; 138(4):1137-48. PubMed ID: 16413128
[TBL] [Abstract][Full Text] [Related]
11. Intracellular survival pathways against glutamate receptor agonist excitotoxicity in cultured neurons. Intracellular calcium responses.
Marini AM; Ueda Y; June CH
Ann N Y Acad Sci; 1999; 890():421-37. PubMed ID: 10668447
[TBL] [Abstract][Full Text] [Related]
12. Regulation of Kv4.2 channels by glutamate in cultured hippocampal neurons.
Lei Z; Deng P; Xu ZC
J Neurochem; 2008 Jul; 106(1):182-92. PubMed ID: 18363830
[TBL] [Abstract][Full Text] [Related]
13. Evidence that the early loss of membrane protein kinase C is a necessary step in the excitatory amino acid-induced death of primary cortical neurons.
Durkin JP; Tremblay R; Chakravarthy B; Mealing G; Morley P; Small D; Song D
J Neurochem; 1997 Apr; 68(4):1400-12. PubMed ID: 9084410
[TBL] [Abstract][Full Text] [Related]
14. NMDA and non-NMDA receptor-mediated excitotoxicity are potentiated in cultured striatal neurons by prior chronic depolarization.
Chen Q; Surmeier DJ; Reiner A
Exp Neurol; 1999 Sep; 159(1):283-96. PubMed ID: 10486197
[TBL] [Abstract][Full Text] [Related]
15. N-methyl-D-aspartate and TrkB receptors protect neurons against glutamate excitotoxicity through an extracellular signal-regulated kinase pathway.
Zhu D; Wu X; Strauss KI; Lipsky RH; Qureshi Z; Terhakopian A; Novelli A; Banaudha K; Marini AM
J Neurosci Res; 2005 Apr; 80(1):104-13. PubMed ID: 15744743
[TBL] [Abstract][Full Text] [Related]
16. Brevetoxins cause acute excitotoxicity in primary cultures of rat cerebellar granule neurons.
Berman FW; Murray TF
J Pharmacol Exp Ther; 1999 Jul; 290(1):439-44. PubMed ID: 10381810
[TBL] [Abstract][Full Text] [Related]
17. Increased sensitivity to N-methyl-D-aspartate receptor-induced excitotoxicity in cerebellar granule cells from interleukin-1 receptor type I-deficient mice.
Pelidou SH; Schultzberg M; Iverfeldt K
J Neuroimmunol; 2002 Dec; 133(1-2):108-15. PubMed ID: 12446013
[TBL] [Abstract][Full Text] [Related]
18. Knockdown of the aryl hydrocarbon receptor attenuates excitotoxicity and enhances NMDA-induced BDNF expression in cortical neurons.
Lin CH; Chen CC; Chou CM; Wang CY; Hung CC; Chen JY; Chang HW; Chen YC; Yeh GC; Lee YH
J Neurochem; 2009 Nov; 111(3):777-89. PubMed ID: 19712055
[TBL] [Abstract][Full Text] [Related]
19. Methamphetamine exposure antagonizes N-methyl-D-aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures.
Smith KJ; Self RL; Butler TR; Mullins MM; Ghayoumi L; Holley RC; Littleton JM; Prendergast MA
Brain Res; 2007 Jul; 1157():74-80. PubMed ID: 17524372
[TBL] [Abstract][Full Text] [Related]
20. Adenosine A2A receptor blockade differentially influences excitotoxic mechanisms at pre- and postsynaptic sites in the rat striatum.
Tebano MT; Pintor A; Frank C; Domenici MR; Martire A; Pepponi R; Potenza RL; Grieco R; Popoli P
J Neurosci Res; 2004 Jul; 77(1):100-7. PubMed ID: 15197743
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]