1897 related articles for article (PubMed ID: 12543789)
1. Insulin-like growth factor I-mediated protection from rapamycin-induced apoptosis is independent of Ras-Erk1-Erk2 and phosphatidylinositol 3'-kinase-Akt signaling pathways.
Thimmaiah KN; Easton J; Huang S; Veverka KA; Germain GS; Harwood FC; Houghton PJ
Cancer Res; 2003 Jan; 63(2):364-74. PubMed ID: 12543789
[TBL] [Abstract][Full Text] [Related]
2. Protection from rapamycin-induced apoptosis by insulin-like growth factor-I is partially dependent on protein kinase C signaling.
Thimmaiah KN; Easton JB; Houghton PJ
Cancer Res; 2010 Mar; 70(5):2000-9. PubMed ID: 20179209
[TBL] [Abstract][Full Text] [Related]
3. In cardiomyocyte hypoxia, insulin-like growth factor-I-induced antiapoptotic signaling requires phosphatidylinositol-3-OH-kinase-dependent and mitogen-activated protein kinase-dependent activation of the transcription factor cAMP response element-binding protein.
Mehrhof FB; Müller FU; Bergmann MW; Li P; Wang Y; Schmitz W; Dietz R; von Harsdorf R
Circulation; 2001 Oct; 104(17):2088-94. PubMed ID: 11673351
[TBL] [Abstract][Full Text] [Related]
4. Stimulation of insulin-like growth factor (IGF) binding protein-3 synthesis by IGF-I and transforming growth factor-alpha is mediated by both phosphatidylinositol-3 kinase and mitogen-activated protein kinase pathways in mammary epithelial cells.
Sivaprasad U; Fleming J; Verma PS; Hogan KA; Desury G; Cohick WS
Endocrinology; 2004 Sep; 145(9):4213-21. PubMed ID: 15192040
[TBL] [Abstract][Full Text] [Related]
5. The lethal effects of pharmacological cyclin-dependent kinase inhibitors in human leukemia cells proceed through a phosphatidylinositol 3-kinase/Akt-dependent process.
Yu C; Rahmani M; Dai Y; Conrad D; Krystal G; Dent P; Grant S
Cancer Res; 2003 Apr; 63(8):1822-33. PubMed ID: 12702569
[TBL] [Abstract][Full Text] [Related]
6. The cell survival signal Akt is differentially activated by PDGF-BB, EGF, and FGF-2 in osteoblastic cells.
Chaudhary LR; Hruska KA
J Cell Biochem; 2001 Mar; 81(2):304-11. PubMed ID: 11241670
[TBL] [Abstract][Full Text] [Related]
7. Differential insulin-like growth factor I receptor signaling and function in estrogen receptor (ER)-positive MCF-7 and ER-negative MDA-MB-231 breast cancer cells.
Bartucci M; Morelli C; Mauro L; Andò S; Surmacz E
Cancer Res; 2001 Sep; 61(18):6747-54. PubMed ID: 11559546
[TBL] [Abstract][Full Text] [Related]
8. Anchorage-independent activation of mitogen-activated protein kinase through phosphatidylinositol-3 kinase by insulin-like growth factor I.
Suzuki K; Takahashi K
Biochem Biophys Res Commun; 2000 May; 272(1):111-5. PubMed ID: 10872812
[TBL] [Abstract][Full Text] [Related]
9. Insulin, insulin-like growth factor-I, and platelet-derived growth factor activate extracellular signal-regulated kinase by distinct pathways in muscle cells.
Tsakiridis T; Tsiani E; Lekas P; Bergman A; Cherepanov V; Whiteside C; Downey GP
Biochem Biophys Res Commun; 2001 Oct; 288(1):205-11. PubMed ID: 11594774
[TBL] [Abstract][Full Text] [Related]
10. Insulin-like growth factor-I-dependent signal transduction pathways leading to the induction of cell growth and differentiation of human neuroblastoma cell line SH-SY5Y: the roles of MAP kinase pathway and PI 3-kinase pathway.
Kurihara S; Hakuno F; Takahashi S
Endocr J; 2000 Dec; 47(6):739-51. PubMed ID: 11228049
[TBL] [Abstract][Full Text] [Related]
11. Cell cycle progression and activation of Akt kinase are required for insulin-like growth factor I-mediated suppression of apoptosis in granulosa cells.
Hu CL; Cowan RG; Harman RM; Quirk SM
Mol Endocrinol; 2004 Feb; 18(2):326-38. PubMed ID: 14593075
[TBL] [Abstract][Full Text] [Related]
12. The chimeric protein tyrosine kinase ETV6-NTRK3 requires both Ras-Erk1/2 and PI3-kinase-Akt signaling for fibroblast transformation.
Tognon C; Garnett M; Kenward E; Kay R; Morrison K; Sorensen PH
Cancer Res; 2001 Dec; 61(24):8909-16. PubMed ID: 11751416
[TBL] [Abstract][Full Text] [Related]
13. Enhancement of survival by LPA via Erk1/Erk2 and PI 3-kinase/Akt pathways in a murine hepatocyte cell line.
Sautin YY; Crawford JM; Svetlov SI
Am J Physiol Cell Physiol; 2001 Dec; 281(6):C2010-9. PubMed ID: 11698260
[TBL] [Abstract][Full Text] [Related]
14. Activation of phosphatidylinositol 3-kinase contributes to insulin-like growth factor I-mediated inhibition of pancreatic beta-cell death.
Liu W; Chin-Chance C; Lee EJ; Lowe WL
Endocrinology; 2002 Oct; 143(10):3802-12. PubMed ID: 12239091
[TBL] [Abstract][Full Text] [Related]
15. Downstream effectors of oncogenic ras in multiple myeloma cells.
Hu L; Shi Y; Hsu JH; Gera J; Van Ness B; Lichtenstein A
Blood; 2003 Apr; 101(8):3126-35. PubMed ID: 12515720
[TBL] [Abstract][Full Text] [Related]
16. Glucose-potentiated chemotaxis in human vascular smooth muscle is dependent on cross-talk between the PI3K and MAPK signaling pathways.
Campbell M; Allen WE; Sawyer C; Vanhaesebroeck B; Trimble ER
Circ Res; 2004 Aug; 95(4):380-8. PubMed ID: 15242975
[TBL] [Abstract][Full Text] [Related]
17. Concomitant activation of the PI3K-Akt and the Ras-ERK signaling pathways is essential for transformation by the V-SEA tyrosine kinase oncogene.
Agazie Y; Ischenko I; Hayman M
Oncogene; 2002 Jan; 21(5):697-707. PubMed ID: 11850798
[TBL] [Abstract][Full Text] [Related]
18. Differential regulation of the phosphoinositide 3-kinase and MAP kinase pathways by hepatocyte growth factor vs. insulin-like growth factor-I in myogenic cells.
Halevy O; Cantley LC
Exp Cell Res; 2004 Jul; 297(1):224-34. PubMed ID: 15194438
[TBL] [Abstract][Full Text] [Related]
19. Synthetic lethal interaction between PI3K/Akt/mTOR and Ras/MEK/ERK pathway inhibition in rhabdomyosarcoma.
Guenther MK; Graab U; Fulda S
Cancer Lett; 2013 Sep; 337(2):200-9. PubMed ID: 23684925
[TBL] [Abstract][Full Text] [Related]
20. Up-regulation of urokinase-type plasminogen activator by insulin-like growth factor-I depends upon phosphatidylinositol-3 kinase and mitogen-activated protein kinase kinase.
Dunn SE; Torres JV; Oh JS; Cykert DM; Barrett JC
Cancer Res; 2001 Feb; 61(4):1367-74. PubMed ID: 11245436
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]