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2. Transplantation of bone marrow genetically engineered to express proinsulin II protects against autoimmune insulitis in NOD mice. Chan J; Clements W; Field J; Nasa Z; Lock P; Yap F; Toh BH; Alderuccio F J Gene Med; 2006 Nov; 8(11):1281-90. PubMed ID: 16989008 [TBL] [Abstract][Full Text] [Related]
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10. Transient insulin autoantibody expression independent of development of diabetes: comparison of NOD and NOR strains. Abiru N; Yu L; Miao D; Maniatis AK; Liu E; Moriyama H; Eisenbarth GS J Autoimmun; 2001 Aug; 17(1):1-6. PubMed ID: 11488632 [TBL] [Abstract][Full Text] [Related]
11. Normal incidence of diabetes in NOD mice tolerant to glutamic acid decarboxylase. Jaeckel E; Klein L; Martin-Orozco N; von Boehmer H J Exp Med; 2003 Jun; 197(12):1635-44. PubMed ID: 12796471 [TBL] [Abstract][Full Text] [Related]
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18. Gene-gun biolistic immunization encoding glutamic acid decarboxylase: a model for studying Langerhans cell abnormalities and mimicry in the nonobese diabetic mouse. Joussemet B; Vu AT; Sai P; Bach JM Ann N Y Acad Sci; 2005 Jun; 1051():613-25. PubMed ID: 16127002 [TBL] [Abstract][Full Text] [Related]
19. Disabling an integral CTL epitope allows suppression of autoimmune diabetes by intranasal proinsulin peptide. Martinez NR; Augstein P; Moustakas AK; Papadopoulos GK; Gregori S; Adorini L; Jackson DC; Harrison LC J Clin Invest; 2003 May; 111(9):1365-71. PubMed ID: 12727928 [TBL] [Abstract][Full Text] [Related]
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