139 related articles for article (PubMed ID: 12670879)
1. Overexpression of the stress protein Grp94 reduces cardiomyocyte necrosis due to calcium overload and simulated ischemia.
Vitadello M; Penzo D; Petronilli V; Michieli G; Gomirato S; Menabò R; Di Lisa F; Gorza L
FASEB J; 2003 May; 17(8):923-5. PubMed ID: 12670879
[TBL] [Abstract][Full Text] [Related]
2. GRP94 reduces cell death in SH-SY5Y cells perturbated calcium homeostasis.
Bando Y; Katayama T; Aleshin AN; Manabe T; Tohyama M
Apoptosis; 2004 Jul; 9(4):501-8. PubMed ID: 15192333
[TBL] [Abstract][Full Text] [Related]
3. Effects of heat shock protein 70 activation by metabolic inhibition preconditioning or kappa-opioid receptor stimulation on Ca2+ homeostasis in rat ventricular myocytes subjected to ischemic insults.
Liu J; Kam KW; Zhou JJ; Yan WY; Chen M; Wu S; Wong TM
J Pharmacol Exp Ther; 2004 Aug; 310(2):606-13. PubMed ID: 15051801
[TBL] [Abstract][Full Text] [Related]
4. Overexpression of sorcin enhances cardiac contractility in vivo and in vitro.
Frank KF; Bölck B; Ding Z; Krause D; Hattebuhr N; Malik A; Brixius K; Hajjar RJ; Schrader J; Schwinger RH
J Mol Cell Cardiol; 2005 Apr; 38(4):607-15. PubMed ID: 15808837
[TBL] [Abstract][Full Text] [Related]
5. Effects of mouse recombinant bone morphogenetic protein-7 transfection on cell apoptosis, NF-kappaB, and downstream genes in cultured primary cardiomyocytes after simulated ischemia and reperfusion injury.
Xu JH; Zhao YY; Wang JK; Yuan ZG; Zhang TZ
J Cardiovasc Pharmacol; 2010 Jul; 56(1):69-77. PubMed ID: 20631551
[TBL] [Abstract][Full Text] [Related]
6. Heat shock protein 70 gene transfection protects rat myocardium cell against anoxia-reoxygeneration injury.
Liu JC; He M; Wan L; Cheng XS
Chin Med J (Engl); 2007 Apr; 120(7):578-83. PubMed ID: 17442206
[TBL] [Abstract][Full Text] [Related]
7. Heat shock proteins in myocardial stress.
Dillmann WH; Mestril R
Z Kardiol; 1995; 84 Suppl 4():87-90. PubMed ID: 8585278
[TBL] [Abstract][Full Text] [Related]
8. [Effects of sarcoplasmic reticulum calcium ATPase 2a overexpression on endoplasmic reticulum stress in cardiomyocytes].
Dong JJ; Lu XC; Liu XH; Zhang ZY; Li XY
Zhonghua Yi Xue Za Zhi; 2009 Feb; 89(6):415-8. PubMed ID: 19567124
[TBL] [Abstract][Full Text] [Related]
9. Endogenous glycogen prevents Ca2+ overload and hypercontracture in harp seal myocardial cells during simulated ischemia.
Henden T; Aasum E; Folkow L; Mjøs OD; Lathrop DA; Larsen TS
J Mol Cell Cardiol; 2004 Jul; 37(1):43-50. PubMed ID: 15242734
[TBL] [Abstract][Full Text] [Related]
10. Grp94 acts as a mediator of curcumin-induced antioxidant defence in myogenic cells.
Pizzo P; Scapin C; Vitadello M; Florean C; Gorza L
J Cell Mol Med; 2010 Apr; 14(4):970-81. PubMed ID: 20569277
[TBL] [Abstract][Full Text] [Related]
11. Allopurinol modulates reactive oxygen species generation and Ca2+ overload in ischemia-reperfused heart and hypoxia-reoxygenated cardiomyocytes.
Kang SM; Lim S; Song H; Chang W; Lee S; Bae SM; Chung JH; Lee H; Kim HG; Yoon DH; Kim TW; Jang Y; Sung JM; Chung NS; Hwang KC
Eur J Pharmacol; 2006 Mar; 535(1-3):212-9. PubMed ID: 16516885
[TBL] [Abstract][Full Text] [Related]
12. Endoplasmic reticulum stress-inducible protein GRP94 is associated with an Mg2+-dependent serine kinase activity modulated by Ca2+ and GRP78/BiP.
Ramakrishnan M; Schönthal AH; Lee AS
J Cell Physiol; 1997 Feb; 170(2):115-29. PubMed ID: 9009140
[TBL] [Abstract][Full Text] [Related]
13. Identification and physiological activity of survival factor released from cardiomyocytes during ischaemia and reperfusion.
Mizukami Y; Ono K; Du CK; Aki T; Hatano N; Okamoto Y; Ikeda Y; Ito H; Hamano K; Morimoto S
Cardiovasc Res; 2008 Sep; 79(4):589-99. PubMed ID: 18534967
[TBL] [Abstract][Full Text] [Related]
14. Overexpression of histidine-rich Ca-binding protein protects against ischemia/reperfusion-induced cardiac injury.
Zhou X; Fan GC; Ren X; Waggoner JR; Gregory KN; Chen G; Jones WK; Kranias EG
Cardiovasc Res; 2007 Aug; 75(3):487-97. PubMed ID: 17499229
[TBL] [Abstract][Full Text] [Related]
15. adapt78, a stress-inducible mRNA, is related to the glucose-regulated protein family of genes.
Leahy KP; Davies KJ; Dull M; Kort JJ; Lawrence KW; Crawford DR
Arch Biochem Biophys; 1999 Aug; 368(1):67-74. PubMed ID: 10415113
[TBL] [Abstract][Full Text] [Related]
16. Adenovirus-mediated FKBP12.6 overexpression induces hypertrophy and apoptosis in cultured neonatal cardiomyocytes.
Zhong J; Chen J; Cao T; Wang L; Zhang W; Liu D; Zhu Z
Clin Exp Pharmacol Physiol; 2009 Feb; 36(2):135-40. PubMed ID: 18759859
[TBL] [Abstract][Full Text] [Related]
17. Deletion of the inducible 70-kDa heat shock protein genes in mice impairs cardiac contractile function and calcium handling associated with hypertrophy.
Kim YK; Suarez J; Hu Y; McDonough PM; Boer C; Dix DJ; Dillmann WH
Circulation; 2006 Jun; 113(22):2589-97. PubMed ID: 16735677
[TBL] [Abstract][Full Text] [Related]
18. Mechanism of cGMP-mediated protection in a cellular model of myocardial reperfusion injury.
Abdallah Y; Gkatzoflia A; Pieper H; Zoga E; Walther S; Kasseckert S; Schäfer M; Schlüter KD; Piper HM; Schäfer C
Cardiovasc Res; 2005 Apr; 66(1):123-31. PubMed ID: 15769455
[TBL] [Abstract][Full Text] [Related]
19. [Protection of myocardiocytes against anoxia-reoxygeneration injury by heat shock protein 70 gene transfection: experiment with rats].
Liu JC; Wan L; He M; Cheng XS
Zhonghua Yi Xue Za Zhi; 2007 Dec; 87(48):3436-9. PubMed ID: 18476547
[TBL] [Abstract][Full Text] [Related]
20. Reduced amount of the glucose-regulated protein GRP94 in skeletal myoblasts results in loss of fusion competence.
Gorza L; Vitadello M
FASEB J; 2000 Mar; 14(3):461-75. PubMed ID: 10698961
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]