514 related articles for article (PubMed ID: 12682257)
1. Genetically resistant mice lacking MyD88-adapter protein display a high susceptibility to Leishmania major infection associated with a polarized Th2 response.
Muraille E; De Trez C; Brait M; De Baetselier P; Leo O; Carlier Y
J Immunol; 2003 Apr; 170(8):4237-41. PubMed ID: 12682257
[TBL] [Abstract][Full Text] [Related]
2. High levels of susceptibility and T helper 2 response in MyD88-deficient mice infected with Leishmania major are interleukin-4 dependent.
Debus A; Gläsner J; Röllinghoff M; Gessner A
Infect Immun; 2003 Dec; 71(12):7215-8. PubMed ID: 14638820
[TBL] [Abstract][Full Text] [Related]
3. Genetically resistant mice lacking interleukin-12 are susceptible to infection with Leishmania major and mount a polarized Th2 cell response.
Mattner F; Magram J; Ferrante J; Launois P; Di Padova K; Behin R; Gately MK; Louis JA; Alber G
Eur J Immunol; 1996 Jul; 26(7):1553-9. PubMed ID: 8766560
[TBL] [Abstract][Full Text] [Related]
4. In BALB/c mice, IL-4 production during the initial phase of infection with Leishmania major is necessary and sufficient to instruct Th2 cell development resulting in progressive disease.
Himmelrich H; Launois P; Maillard I; Biedermann T; Tacchini-Cottier F; Locksley RM; Röcken M; Louis JA
J Immunol; 2000 May; 164(9):4819-25. PubMed ID: 10779790
[TBL] [Abstract][Full Text] [Related]
5. Mice lacking myeloid differentiation factor 88 display profound defects in host resistance and immune responses to Mycobacterium avium infection not exhibited by Toll-like receptor 2 (TLR2)- and TLR4-deficient animals.
Feng CG; Scanga CA; Collazo-Custodio CM; Cheever AW; Hieny S; Caspar P; Sher A
J Immunol; 2003 Nov; 171(9):4758-64. PubMed ID: 14568952
[TBL] [Abstract][Full Text] [Related]
6. KM(+), a lectin from Artocarpus integrifolia, induces IL-12 p40 production by macrophages and switches from type 2 to type 1 cell-mediated immunity against Leishmania major antigens, resulting in BALB/c mice resistance to infection.
Panunto-Castelo A; Souza MA; Roque-Barreira MC; Silva JS
Glycobiology; 2001 Dec; 11(12):1035-42. PubMed ID: 11805076
[TBL] [Abstract][Full Text] [Related]
7. Cutting edge: MyD88 is required for resistance to Toxoplasma gondii infection and regulates parasite-induced IL-12 production by dendritic cells.
Scanga CA; Aliberti J; Jankovic D; Tilloy F; Bennouna S; Denkers EY; Medzhitov R; Sher A
J Immunol; 2002 Jun; 168(12):5997-6001. PubMed ID: 12055206
[TBL] [Abstract][Full Text] [Related]
8. BALB/c mice bearing a transgenic IL-12 receptor beta 2 gene exhibit a nonhealing phenotype to Leishmania major infection despite intact IL-12 signaling.
Nishikomori R; Gurunathan S; Nishikomori K; Strober W
J Immunol; 2001 Jun; 166(11):6776-83. PubMed ID: 11359836
[TBL] [Abstract][Full Text] [Related]
9. TLR ligands can activate dendritic cells to provide a MyD88-dependent negative signal for Th2 cell development.
Sun J; Walsh M; Villarino AV; Cervi L; Hunter CA; Choi Y; Pearce EJ
J Immunol; 2005 Jan; 174(2):742-51. PubMed ID: 15634894
[TBL] [Abstract][Full Text] [Related]
10. An immunomodulatory function for neutrophils during the induction of a CD4+ Th2 response in BALB/c mice infected with Leishmania major.
Tacchini-Cottier F; Zweifel C; Belkaid Y; Mukankundiye C; Vasei M; Launois P; Milon G; Louis JA
J Immunol; 2000 Sep; 165(5):2628-36. PubMed ID: 10946291
[TBL] [Abstract][Full Text] [Related]
11. IL-12Rbeta2-deficient mice of a genetically resistant background are susceptible to Leishmania major infection and develop a parasite-specific Th2 immune response.
Chakir H; Campos-Neto A; Mojibian M; Webb JR
Microbes Infect; 2003 Apr; 5(4):241-9. PubMed ID: 12706437
[TBL] [Abstract][Full Text] [Related]
12. CD28 costimulation and parasite dose combine to influence the susceptibility of BALB/c mice to infection with Leishmania major.
Compton HL; Farrell JP
J Immunol; 2002 Feb; 168(3):1302-8. PubMed ID: 11801669
[TBL] [Abstract][Full Text] [Related]
13. The MyD88 protein 88 pathway is differently involved in immune responses induced by distinct substrains of Leishmania major.
Revaz-Breton M; Ronet C; Ives A; Torre YH; Masina S; Tacchini-Cottier F; Launois P
Eur J Immunol; 2010 Jun; 40(6):1697-707. PubMed ID: 20333623
[TBL] [Abstract][Full Text] [Related]
14. Endotoxin can induce MyD88-deficient dendritic cells to support T(h)2 cell differentiation.
Kaisho T; Hoshino K; Iwabe T; Takeuchi O; Yasui T; Akira S
Int Immunol; 2002 Jul; 14(7):695-700. PubMed ID: 12096028
[TBL] [Abstract][Full Text] [Related]
15. Heat-killed Brucella abortus induces TNF and IL-12p40 by distinct MyD88-dependent pathways: TNF, unlike IL-12p40 secretion, is Toll-like receptor 2 dependent.
Huang LY; Aliberti J; Leifer CA; Segal DM; Sher A; Golenbock DT; Golding B
J Immunol; 2003 Aug; 171(3):1441-6. PubMed ID: 12874236
[TBL] [Abstract][Full Text] [Related]
16. Induction of a Th2 population from a polarized Leishmania-specific Th1 population by in vitro culture with IL-4.
Mocci S; Coffman RL
J Immunol; 1995 Apr; 154(8):3779-87. PubMed ID: 7706719
[TBL] [Abstract][Full Text] [Related]
17. Dendritic cell-derived IL-12p40 homodimer contributes to susceptibility in cutaneous leishmaniasis in BALB/c mice.
Nigg AP; Zahn S; Rückerl D; Hölscher C; Yoshimoto T; Ehrchen JM; Wölbing F; Udey MC; von Stebut E
J Immunol; 2007 Jun; 178(11):7251-8. PubMed ID: 17513774
[TBL] [Abstract][Full Text] [Related]
18. Setting in motion the immune mechanisms underlying genetically determined resistance and susceptibility to infection with Leishmania major.
Launois P; Conceiçao-Silva F; Himmerlich H; Parra-Lopez C; Tacchini-Cottier F; Louis JA
Parasite Immunol; 1998 May; 20(5):223-30. PubMed ID: 9651923
[TBL] [Abstract][Full Text] [Related]
19. Control of infection with Leishmania major in susceptible BALB/c mice lacking the common gamma-chain for FcR is associated with reduced production of IL-10 and TGF-beta by parasitized cells.
Padigel UM; Farrell JP
J Immunol; 2005 May; 174(10):6340-5. PubMed ID: 15879134
[TBL] [Abstract][Full Text] [Related]
20. Intracellular bacterial infection-induced IFN-gamma is critically but not solely dependent on Toll-like receptor 4-myeloid differentiation factor 88-IFN-alpha beta-STAT1 signaling.
Rothfuchs AG; Trumstedt C; Wigzell H; Rottenberg ME
J Immunol; 2004 May; 172(10):6345-53. PubMed ID: 15128825
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
