BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

123 related articles for article (PubMed ID: 12706231)

  • 1. Protein kinase C activation generates superoxide and contributes to impairment of cerebrovasodilation induced by G protein activation after brain injury.
    Armstead WM
    Brain Res; 2003 May; 971(2):153-60. PubMed ID: 12706231
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Cyclooxygenase-2-dependent superoxide generation contributes to age-dependent impairment of G protein-mediated cerebrovasodilation.
    Armstead WM
    Anesthesiology; 2003 Jun; 98(6):1378-83. PubMed ID: 12766646
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Vasopressin-induced protein kinase C-dependent superoxide generation contributes to atp-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injury.
    Armstead WM
    Stroke; 2001 Jun; 32(6):1408-14. PubMed ID: 11387506
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Influence of polyethylene glycol superoxide dismutase/catalase on altered opioid-induced pial artery dilation after brain injury.
    Thorogood MC; Armstead WM
    Anesthesiology; 1996 Mar; 84(3):614-25. PubMed ID: 8659790
    [TBL] [Abstract][Full Text] [Related]  

  • 5. ET-1 contributes to age-dependent G protein impairment after brain injury.
    Armstead WM
    J Neurotrauma; 2003 Jan; 20(1):105-10. PubMed ID: 12614592
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Superoxide generation links protein kinase C activation to impaired ATP-sensitive K+ channel function after brain injury.
    Armstead WM
    Stroke; 1999 Jan; 30(1):153-9. PubMed ID: 9880404
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Superoxide generation links nociceptin/orphanin FQ (NOC/oFQ) release to impaired N-methyl-D-aspartate cerebrovasodilation after brain injury.
    Kulkarni M; Armstead WM
    Stroke; 2000 Aug; 31(8):1990-6. PubMed ID: 10926968
    [TBL] [Abstract][Full Text] [Related]  

  • 8. NOC/oFQ activates PKC and generates superoxide to impair hypotensive cerebrovasodilation after hypoxia/ischemia.
    Armstead W
    Med Sci Monit; 2002 Jan; 8(1):BR8-BR14. PubMed ID: 11782666
    [TBL] [Abstract][Full Text] [Related]  

  • 9. NOC/oFQ PKC-dependent superoxide generation contributes to hypoxic-ischemic impairment of NMDA cerebrovasodilation.
    Armstead WM
    Am J Physiol Heart Circ Physiol; 2000 Dec; 279(6):H2678-84. PubMed ID: 11087221
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Influence of brain injury on vasopressin-induced pial artery vasodilation: role of superoxide anion.
    Armstead WM
    Am J Physiol; 1996 Apr; 270(4 Pt 2):H1272-8. PubMed ID: 8967366
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K(+) channel function impairment after brain injury.
    Armstead WM
    Brain Res; 2001 Aug; 910(1-2):19-28. PubMed ID: 11489250
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Role of altered cyclooxygenase metabolism in impaired cerebrovasodilation to nociceptin/orphanin FQ following brain injury.
    Armstead WM
    Brain Res Bull; 2000 Dec; 53(6):807-12. PubMed ID: 11179847
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Relationship between NOC/oFQ, dynorphin, and COX-2 activation in impaired NMDA cerebrovasodilation after brain injury.
    Kulkarni M; Armstead WM
    J Neurotrauma; 2002 Aug; 19(8):965-73. PubMed ID: 12225656
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Endothelin production links superoxide generation to altered opioid-induced pial artery vasodilation after brain injury in pigs.
    Kasemsri T; Armstead WM
    Stroke; 1997 Jan; 28(1):190-6; discussion 197. PubMed ID: 8996511
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Endothelin-Induced cyclooxygenase-dependent superoxide generation contributes to K+ channel functional impairment after brain injury.
    Armstead WM
    J Neurotrauma; 2001 Oct; 18(10):1039-48. PubMed ID: 11686491
    [TBL] [Abstract][Full Text] [Related]  

  • 16. G protein activation elicits cerebrovasodilation through interaction with K(ATP) and K(Ca) channels.
    Armstead WM
    Brain Res; 2002 Dec; 957(2):369-72. PubMed ID: 12445981
    [TBL] [Abstract][Full Text] [Related]  

  • 17. NOC/oFQ activates ERK and JNK but not p38 MAPK to impair prostaglandin cerebrovasodilation after brain injury.
    Ross J; Armstead WM
    Brain Res; 2005 Aug; 1054(1):95-102. PubMed ID: 16099438
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Plasminogen activators contribute to age-dependent impairment of NMDA cerebrovasodilation after brain injury.
    Armstead WM; Cines DB; Higazie AA
    Brain Res Dev Brain Res; 2005 May; 156(2):139-46. PubMed ID: 16099300
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to K(ATP) and K(ca) channel impairment after brain injury.
    Armstead WM
    Brain Res; 2002 Jul; 943(2):276-82. PubMed ID: 12101050
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Age dependent endothelin contribution to NOC/oFQ induced impairment of NMDA cerebrovasodilation after brain injury.
    Armstead WM
    Peptides; 2001 Jan; 22(1):39-46. PubMed ID: 11179596
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 7.