These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
97 related articles for article (PubMed ID: 12738236)
1. Na+ channel mutation leading to loss of function and non-progressive cardiac conduction defects. Herfst LJ; Potet F; Bezzina CR; Groenewegen WA; Le Marec H; Hoorntje TM; Demolombe S; Baró I; Escande D; Jongsma HJ; Wilde AA; Rook MB J Mol Cell Cardiol; 2003 May; 35(5):549-57. PubMed ID: 12738236 [TBL] [Abstract][Full Text] [Related]
2. Congenital long-QT syndrome caused by a novel mutation in a conserved acidic domain of the cardiac Na+ channel. Wei J; Wang DW; Alings M; Fish F; Wathen M; Roden DM; George AL Circulation; 1999 Jun; 99(24):3165-71. PubMed ID: 10377081 [TBL] [Abstract][Full Text] [Related]
3. Correlations between clinical and physiological consequences of the novel mutation R878C in a highly conserved pore residue in the cardiac Na+ channel. Zhang Y; Wang T; Ma A; Zhou X; Gui J; Wan H; Shi R; Huang C; Grace AA; Huang CL; Trump D; Zhang H; Zimmer T; Lei M Acta Physiol (Oxf); 2008 Dec; 194(4):311-23. PubMed ID: 18616619 [TBL] [Abstract][Full Text] [Related]
4. Novel SCN5A mutation leading either to isolated cardiac conduction defect or Brugada syndrome in a large French family. Kyndt F; Probst V; Potet F; Demolombe S; Chevallier JC; Baro I; Moisan JP; Boisseau P; Schott JJ; Escande D; Le Marec H Circulation; 2001 Dec; 104(25):3081-6. PubMed ID: 11748104 [TBL] [Abstract][Full Text] [Related]
5. A mutant cardiac sodium channel with multiple biophysical defects associated with overlapping clinical features of Brugada syndrome and cardiac conduction disease. Shirai N; Makita N; Sasaki K; Yokoi H; Sakuma I; Sakurada H; Akai J; Kimura A; Hiraoka M; Kitabatake A Cardiovasc Res; 2002 Feb; 53(2):348-54. PubMed ID: 11827685 [TBL] [Abstract][Full Text] [Related]
6. A novel mutation in SCN5A, delQKP 1507-1509, causing long QT syndrome: role of Q1507 residue in sodium channel inactivation. Keller DI; Acharfi S; Delacrétaz E; Benammar N; Rotter M; Pfammatter JP; Fressart V; Guicheney P; Chahine M J Mol Cell Cardiol; 2003 Dec; 35(12):1513-21. PubMed ID: 14654377 [TBL] [Abstract][Full Text] [Related]
7. Novel LQT-3 mutation affects Na+ channel activity through interactions between alpha- and beta1-subunits. An RH; Wang XL; Kerem B; Benhorin J; Medina A; Goldmit M; Kass RS Circ Res; 1998 Jul; 83(2):141-6. PubMed ID: 9686753 [TBL] [Abstract][Full Text] [Related]
8. A novel mutation in the SCN5A gene is associated with Brugada syndrome. Shin DJ; Kim E; Park SB; Jang WC; Bae Y; Han J; Jang Y; Joung B; Lee MH; Kim SS; Huang H; Chahine M; Yoon SK Life Sci; 2007 Jan; 80(8):716-24. PubMed ID: 17141278 [TBL] [Abstract][Full Text] [Related]
9. Compound heterozygosity for mutations (W156X and R225W) in SCN5A associated with severe cardiac conduction disturbances and degenerative changes in the conduction system. Bezzina CR; Rook MB; Groenewegen WA; Herfst LJ; van der Wal AC; Lam J; Jongsma HJ; Wilde AA; Mannens MM Circ Res; 2003 Feb; 92(2):159-68. PubMed ID: 12574143 [TBL] [Abstract][Full Text] [Related]
10. Human SCN5A gene mutations alter cardiac sodium channel kinetics and are associated with the Brugada syndrome. Rook MB; Bezzina Alshinawi C; Groenewegen WA; van Gelder IC; van Ginneken AC; Jongsma HJ; Mannens MM; Wilde AA Cardiovasc Res; 1999 Dec; 44(3):507-17. PubMed ID: 10690282 [TBL] [Abstract][Full Text] [Related]
11. Clinical, genetic, and biophysical characterization of SCN5A mutations associated with atrioventricular conduction block. Wang DW; Viswanathan PC; Balser JR; George AL; Benson DW Circulation; 2002 Jan; 105(3):341-6. PubMed ID: 11804990 [TBL] [Abstract][Full Text] [Related]
12. Brugada syndrome and fever: genetic and molecular characterization of patients carrying SCN5A mutations. Keller DI; Rougier JS; Kucera JP; Benammar N; Fressart V; Guicheney P; Madle A; Fromer M; Schläpfer J; Abriel H Cardiovasc Res; 2005 Aug; 67(3):510-9. PubMed ID: 15890323 [TBL] [Abstract][Full Text] [Related]
18. A1152D mutation of the Na+ channel causes paramyotonia congenita and emphasizes the role of DIII/S4-S5 linker in fast inactivation. Bouhours M; Luce S; Sternberg D; Willer JC; Fontaine B; Tabti N J Physiol; 2005 Jun; 565(Pt 2):415-27. PubMed ID: 15790667 [TBL] [Abstract][Full Text] [Related]
19. Molecular mechanism for an inherited cardiac arrhythmia. Bennett PB; Yazawa K; Makita N; George AL Nature; 1995 Aug; 376(6542):683-5. PubMed ID: 7651517 [TBL] [Abstract][Full Text] [Related]
20. Febrile seizures and generalized epilepsy associated with a mutation in the Na+-channel beta1 subunit gene SCN1B. Wallace RH; Wang DW; Singh R; Scheffer IE; George AL; Phillips HA; Saar K; Reis A; Johnson EW; Sutherland GR; Berkovic SF; Mulley JC Nat Genet; 1998 Aug; 19(4):366-70. PubMed ID: 9697698 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]