760 related articles for article (PubMed ID: 12891712)
1. The non-homologous end-joining pathway is not involved in the radiosensitization of mammalian cells by heat shock.
Dynlacht JR; Bittner ME; Bethel JA; Beck BD
J Cell Physiol; 2003 Sep; 196(3):557-64. PubMed ID: 12891712
[TBL] [Abstract][Full Text] [Related]
2. Heat inactivation of Ku autoantigen: possible role in hyperthermic radiosensitization.
Burgman P; Ouyang H; Peterson S; Chen DJ; Li GC
Cancer Res; 1997 Jul; 57(14):2847-50. PubMed ID: 9230187
[TBL] [Abstract][Full Text] [Related]
3. Mechanisms of DNA double strand break repair and chromosome aberration formation.
Iliakis G; Wang H; Perrault AR; Boecker W; Rosidi B; Windhofer F; Wu W; Guan J; Terzoudi G; Pantelias G
Cytogenet Genome Res; 2004; 104(1-4):14-20. PubMed ID: 15162010
[TBL] [Abstract][Full Text] [Related]
4. DNA double strand break repair inhibition as a cause of heat radiosensitization: re-evaluation considering backup pathways of NHEJ.
Iliakis G; Wu W; Wang M
Int J Hyperthermia; 2008 Feb; 24(1):17-29. PubMed ID: 18214766
[TBL] [Abstract][Full Text] [Related]
5. Biochemical evidence for Ku-independent backup pathways of NHEJ.
Wang H; Perrault AR; Takeda Y; Qin W; Wang H; Iliakis G
Nucleic Acids Res; 2003 Sep; 31(18):5377-88. PubMed ID: 12954774
[TBL] [Abstract][Full Text] [Related]
6. Involvement of Ku80 in microhomology-mediated end joining for DNA double-strand breaks in vivo.
Katsura Y; Sasaki S; Sato M; Yamaoka K; Suzukawa K; Nagasawa T; Yokota J; Kohno T
DNA Repair (Amst); 2007 May; 6(5):639-48. PubMed ID: 17236818
[TBL] [Abstract][Full Text] [Related]
7. Radiation-induced genomic rearrangements formed by nonhomologous end-joining of DNA double-strand breaks.
Rothkamm K; Kühne M; Jeggo PA; Löbrich M
Cancer Res; 2001 May; 61(10):3886-93. PubMed ID: 11358801
[TBL] [Abstract][Full Text] [Related]
8. Repair of radiation induced DNA double strand breaks by backup NHEJ is enhanced in G2.
Wu W; Wang M; Wu W; Singh SK; Mussfeldt T; Iliakis G
DNA Repair (Amst); 2008 Feb; 7(2):329-38. PubMed ID: 18155970
[TBL] [Abstract][Full Text] [Related]
9. Examining the non-homologous repair process following cisplatin and radiation treatments.
Myint WK; Ng C; Raaphorst GP
Int J Radiat Biol; 2002 May; 78(5):417-24. PubMed ID: 12020431
[TBL] [Abstract][Full Text] [Related]
10. Collaboration of homologous recombination and nonhomologous end-joining factors for the survival and integrity of mice and cells.
Couëdel C; Mills KD; Barchi M; Shen L; Olshen A; Johnson RD; Nussenzweig A; Essers J; Kanaar R; Li GC; Alt FW; Jasin M
Genes Dev; 2004 Jun; 18(11):1293-304. PubMed ID: 15175261
[TBL] [Abstract][Full Text] [Related]
11. The Ku-dependent non-homologous end-joining but not other repair pathway is inhibited by high linear energy transfer ionizing radiation.
Wang H; Wang X; Zhang P; Wang Y
DNA Repair (Amst); 2008 May; 7(5):725-33. PubMed ID: 18325854
[TBL] [Abstract][Full Text] [Related]
12. DNA ligase IV-deficient cells are more resistant to ionizing radiation in the absence of Ku70: Implications for DNA double-strand break repair.
Adachi N; Ishino T; Ishii Y; Takeda S; Koyama H
Proc Natl Acad Sci U S A; 2001 Oct; 98(21):12109-13. PubMed ID: 11593023
[TBL] [Abstract][Full Text] [Related]
13. Nuclear localization of mouse Ku70 in interphase cells and focus formation of mouse Ku70 at DNA damage sites immediately after irradiation.
Koike M; Yutoku Y; Koike A
J Vet Med Sci; 2015 Sep; 77(9):1137-42. PubMed ID: 25947323
[TBL] [Abstract][Full Text] [Related]
14. DNA-PK-dependent phosphorylation of Ku70/80 is not required for non-homologous end joining.
Douglas P; Gupta S; Morrice N; Meek K; Lees-Miller SP
DNA Repair (Amst); 2005 Aug; 4(9):1006-18. PubMed ID: 15941674
[TBL] [Abstract][Full Text] [Related]
15. Genetic evidence for the involvement of DNA ligase IV in the DNA-PK-dependent pathway of non-homologous end joining in mammalian cells.
Wang H; Zeng ZC; Perrault AR; Cheng X; Qin W; Iliakis G
Nucleic Acids Res; 2001 Apr; 29(8):1653-60. PubMed ID: 11292837
[TBL] [Abstract][Full Text] [Related]
16. Breast cancer risk associated with genotypic polymorphism of the nonhomologous end-joining genes: a multigenic study on cancer susceptibility.
Fu YP; Yu JC; Cheng TC; Lou MA; Hsu GC; Wu CY; Chen ST; Wu HS; Wu PE; Shen CY
Cancer Res; 2003 May; 63(10):2440-6. PubMed ID: 12750264
[TBL] [Abstract][Full Text] [Related]
17. Ku80-deleted cells are defective at base excision repair.
Li H; Marple T; Hasty P
Mutat Res; 2013; 745-746():16-25. PubMed ID: 23567907
[TBL] [Abstract][Full Text] [Related]
18. KARP-1 works as a heterodimer with Ku70, but the function of KARP-1 cannot perfectly replace that of Ku80 in DSB repair.
Koike M; Yutoku Y; Koike A
Exp Cell Res; 2011 Oct; 317(16):2267-75. PubMed ID: 21756904
[TBL] [Abstract][Full Text] [Related]
19. Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells.
Fattah F; Lee EH; Weisensel N; Wang Y; Lichter N; Hendrickson EA
PLoS Genet; 2010 Feb; 6(2):e1000855. PubMed ID: 20195511
[TBL] [Abstract][Full Text] [Related]
20. Double-strand break repair by Ku70 requires heterodimerization with Ku80 and DNA binding functions.
Jin S; Weaver DT
EMBO J; 1997 Nov; 16(22):6874-85. PubMed ID: 9362500
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]