306 related articles for article (PubMed ID: 12913204)
1. Biomagnification of cycad neurotoxins in flying foxes: implications for ALS-PDC in Guam.
Banack SA; Cox PA
Neurology; 2003 Aug; 61(3):387-9. PubMed ID: 12913204
[TBL] [Abstract][Full Text] [Related]
2. Western Pacific ALS/PDC and flying foxes: What's next?
Armon C
Neurology; 2003 Aug; 61(3):291-2. PubMed ID: 12913185
[No Abstract] [Full Text] [Related]
3. Biomagnification of cyanobacterial neurotoxins and neurodegenerative disease among the Chamorro people of Guam.
Cox PA; Banack SA; Murch SJ
Proc Natl Acad Sci U S A; 2003 Nov; 100(23):13380-3. PubMed ID: 14612559
[TBL] [Abstract][Full Text] [Related]
4. Cycad neurotoxins, consumption of flying foxes, and ALS-PDC disease in Guam.
Cox PA; Sacks OW
Neurology; 2002 Mar; 58(6):956-9. PubMed ID: 11914415
[TBL] [Abstract][Full Text] [Related]
5. Beyond Guam: the cyanobacteria/BMAA hypothesis of the cause of ALS and other neurodegenerative diseases.
Bradley WG; Mash DC
Amyotroph Lateral Scler; 2009; 10 Suppl 2():7-20. PubMed ID: 19929726
[TBL] [Abstract][Full Text] [Related]
6. Cycad neurotoxin, consumption of flying foxes, and ALS/PDC disease in Guam.
Chen KM; Craig UK; Lee CT; Haddock R
Neurology; 2002 Nov; 59(10):1664; author reply 1664-5. PubMed ID: 12451227
[No Abstract] [Full Text] [Related]
7. Neurotoxic flying foxes as dietary items for the Chamorro people, Marianas Islands.
Banack SA; Murch SJ; Cox PA
J Ethnopharmacol; 2006 Jun; 106(1):97-104. PubMed ID: 16457975
[TBL] [Abstract][Full Text] [Related]
8. A mechanism for slow release of biomagnified cyanobacterial neurotoxins and neurodegenerative disease in Guam.
Murch SJ; Cox PA; Banack SA
Proc Natl Acad Sci U S A; 2004 Aug; 101(33):12228-31. PubMed ID: 15295100
[TBL] [Abstract][Full Text] [Related]
9. A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.
Chernoff N; Hill DJ; Diggs DL; Faison BD; Francis BM; Lang JR; Larue MM; Le TT; Loftin KA; Lugo JN; Schmid JE; Winnik WM
J Toxicol Environ Health B Crit Rev; 2017; 20(4):1-47. PubMed ID: 28598725
[TBL] [Abstract][Full Text] [Related]
10. Animal models of BMAA neurotoxicity: a critical review.
Karamyan VT; Speth RC
Life Sci; 2008 Jan; 82(5-6):233-46. PubMed ID: 18191417
[TBL] [Abstract][Full Text] [Related]
11. 2-Amino-3-(methylamino)-propanoic acid (BMAA) in cycad flour: an unlikely cause of amyotrophic lateral sclerosis and parkinsonism-dementia of Guam.
Duncan MW; Steele JC; Kopin IJ; Markey SP
Neurology; 1990 May; 40(5):767-72. PubMed ID: 2330104
[TBL] [Abstract][Full Text] [Related]
12. Neurodegenerative disease. Guam's deadly stalker: on the loose worldwide?
Miller G
Science; 2006 Jul; 313(5786):428-31. PubMed ID: 16873621
[No Abstract] [Full Text] [Related]
13. Creating a Simian Model of Guam ALS/PDC Which Reflects Chamorro Lifetime BMAA Exposures.
Banack SA; Cox PA
Neurotox Res; 2018 Jan; 33(1):24-32. PubMed ID: 28478528
[TBL] [Abstract][Full Text] [Related]
14. Return of the cycad hypothesis - does the amyotrophic lateral sclerosis/parkinsonism dementia complex (ALS/PDC) of Guam have new implications for global health?
Ince PG; Codd GA
Neuropathol Appl Neurobiol; 2005 Aug; 31(4):345-53. PubMed ID: 16008818
[TBL] [Abstract][Full Text] [Related]
15. Content of the neurotoxins cycasin (methylazoxymethanol beta-D-glucoside) and BMAA (beta-N-methylamino-L-alanine) in cycad flour prepared by Guam Chamorros.
Kisby GE; Ellison M; Spencer PS
Neurology; 1992 Jul; 42(7):1336-40. PubMed ID: 1620343
[TBL] [Abstract][Full Text] [Related]
16. The analysis of underivatized β-Methylamino-L-alanine (BMAA), BAMA, AEG & 2,4-DAB in Pteropus mariannus mariannus specimens using HILIC-LC-MS/MS.
Foss AJ; Chernoff N; Aubel MT
Toxicon; 2018 Sep; 152():150-159. PubMed ID: 30102919
[TBL] [Abstract][Full Text] [Related]
17. Previous studies underestimate BMAA concentrations in cycad flour.
Cheng R; Banack SA
Amyotroph Lateral Scler; 2009; 10 Suppl 2():41-3. PubMed ID: 19929730
[TBL] [Abstract][Full Text] [Related]
18. BMAA selectively injures motor neurons via AMPA/kainate receptor activation.
Rao SD; Banack SA; Cox PA; Weiss JH
Exp Neurol; 2006 Sep; 201(1):244-52. PubMed ID: 16764863
[TBL] [Abstract][Full Text] [Related]
19. The cycad neurotoxic amino acid, beta-N-methylamino-L-alanine (BMAA), elevates intracellular calcium levels in dissociated rat brain cells.
Brownson DM; Mabry TJ; Leslie SW
J Ethnopharmacol; 2002 Oct; 82(2-3):159-67. PubMed ID: 12241991
[TBL] [Abstract][Full Text] [Related]
20. The ALS/PDC syndrome of Guam and the cycad hypothesis.
Cruz-Aguado R; Shaw CA
Neurology; 2009 Feb; 72(5):474, 476; author reply 475-6. PubMed ID: 19195085
[No Abstract] [Full Text] [Related]
[Next] [New Search]