183 related articles for article (PubMed ID: 1303308)
1. EGF receptor activity and mitogenic response of Balb/3T3 cells expressing Ras and Myc oncogenes. EGF receptor activity in oncogene transformed cells.
Gopas J; Ono M; Princler G; Smith MR; Tainsky MA; Siddiqui MA; Wishniak O; Segal S; Kuwano M; Kung HF
Cell Mol Biol; 1992 Sep; 38(6):605-14. PubMed ID: 1303308
[TBL] [Abstract][Full Text] [Related]
2. Regulation of epidermal growth factor receptor by activated H-ras and V-myc oncogenes in mouse Balb/3T3 cells: possible roles of AP-1.
Okimoto T; Kohno K; Kuwano M; Gopas J; Kung HF; Ono M
Oncogene; 1996 Apr; 12(8):1625-33. PubMed ID: 8622882
[TBL] [Abstract][Full Text] [Related]
3. Anchorage-independent growth of v-myc-transformed Balb/c 3T3 cells is promoted by platelet-derived growth factor or co-transformation by other oncogenes.
Vennström B; Bravo R
Oncogene; 1987; 1(3):271-6. PubMed ID: 3330775
[TBL] [Abstract][Full Text] [Related]
4. Progressive relaxation of Go-arrest controls and altered responsiveness to insulin, EGF and thrombin in CCL39 lung fibroblasts over-expressing myc and ras oncogenes.
Pichon F; Seuwen K; Pouysségur J; Lagarde AE
Oncogene; 1988 Oct; 3(4):373-81. PubMed ID: 3078948
[TBL] [Abstract][Full Text] [Related]
5. Loss of growth responsiveness to epidermal growth factor and enhanced production of alpha-transforming growth factors in ras-transformed mouse mammary epithelial cells.
Salomon DS; Perroteau I; Kidwell WR; Tam J; Derynck R
J Cell Physiol; 1987 Mar; 130(3):397-409. PubMed ID: 3494020
[TBL] [Abstract][Full Text] [Related]
6. Heparin-treated, v-myc-transformed chicken heart mesenchymal cells assume a normal morphology but are hypersensitive to epidermal growth factor (EGF) and brain fibroblast growth factor (bFGF); cells transformed by the v-Ha-ras oncogene are refractory to EGF and bFGF but are hypersensitive to insulin-like growth factors.
Balk SD; Riley TM; Gunther HS; Morisi A
Proc Natl Acad Sci U S A; 1985 Sep; 82(17):5781-5. PubMed ID: 3898072
[TBL] [Abstract][Full Text] [Related]
7. Transforming growth factor-alpha expression is enhanced in human mammary epithelial cells transformed by an activated c-Ha-ras protooncogene but not by the c-neu protooncogene, and overexpression of the transforming growth factor-alpha complementary DNA leads to transformation.
Ciardiello F; McGeady ML; Kim N; Basolo F; Hynes N; Langton BC; Yokozaki H; Saeki T; Elliott JW; Masui H
Cell Growth Differ; 1990 Sep; 1(9):407-20. PubMed ID: 1981145
[TBL] [Abstract][Full Text] [Related]
8. Transforming growth factor alpha production and epidermal growth factor receptor expression in normal and oncogene transformed human mammary epithelial cells.
Valverius EM; Bates SE; Stampfer MR; Clark R; McCormick F; Salomon DS; Lippman ME; Dickson RB
Mol Endocrinol; 1989 Jan; 3(1):203-14. PubMed ID: 2783757
[TBL] [Abstract][Full Text] [Related]
9. NIH 3T3 cells transfected with a yeast H(+)-ATPase have altered sensitivity to insulin, insulin growth factor-I, and platelet-derived growth factor-AA.
Peterson EP; Martinez GM; Martinez-Zaguilan R; Perona R; Gillies RJ
J Cell Physiol; 1994 Jun; 159(3):551-60. PubMed ID: 8188769
[TBL] [Abstract][Full Text] [Related]
10. Prolonged inhibition by X-rays of DNA synthesis in cells obtained by transformation of primary rat embryo fibroblasts with oncogenes H-ras and v-myc.
Wang Y; Iliakis G
Cancer Res; 1992 Feb; 52(3):508-14. PubMed ID: 1732037
[TBL] [Abstract][Full Text] [Related]
11. The effect of H-ras expression on tumorigenicity and immunogenicity of Balb/c 3T3 fibroblasts.
Ehrlich T; Wishniak O; Isakov N; Cohen O; Segal S; Rager-Zisman B; Gopas J
Immunol Lett; 1993 Dec; 39(1):3-8. PubMed ID: 8144187
[TBL] [Abstract][Full Text] [Related]
12. ras and neu oncogenes reverse serum inhibition and epidermal growth factor dependence of serum-free mouse embryo cells.
Shirahata S; Rawson C; Loo D; Chang YJ; Barnes D
J Cell Physiol; 1990 Jul; 144(1):69-76. PubMed ID: 1973170
[TBL] [Abstract][Full Text] [Related]
13. Interactions of growth factors and retroviral oncogenes with mitogenic signal transduction pathways of Balb/MK keratinocytes.
Falco JP; Taylor WG; Di Fiore PP; Weissman BE; Aaronson SA
Oncogene; 1988 Jun; 2(6):573-8. PubMed ID: 3290805
[TBL] [Abstract][Full Text] [Related]
14. Altered H1 histamine receptor signaling in Balb/3T3 cells transformed by v-K-ras and v-H-ras oncogenes.
Blackmore PF; Oakes SG; Somers KD
Oncogene; 1992 Oct; 7(10):2053-7. PubMed ID: 1408146
[TBL] [Abstract][Full Text] [Related]
15. Oncostatin M-specific receptor mediates inhibition of breast cancer cell growth and down-regulation of the c-myc proto-oncogene.
Liu J; Spence MJ; Wallace PM; Forcier K; Hellström I; Vestal RE
Cell Growth Differ; 1997 Jun; 8(6):667-76. PubMed ID: 9186000
[TBL] [Abstract][Full Text] [Related]
16. Polypeptide growth factors: roles in normal and abnormal cell growth.
Deuel TF
Annu Rev Cell Biol; 1987; 3():443-92. PubMed ID: 3318882
[TBL] [Abstract][Full Text] [Related]
17. The transcription activation domains of v-Myc and VP16 interact with common factors required for cellular transformation and proliferation.
Min S; Crider-Miller SJ; Taparowsky EJ
Cell Growth Differ; 1994 Jun; 5(6):563-73. PubMed ID: 8086335
[TBL] [Abstract][Full Text] [Related]
18. Flat revertants derived from Kirsten murine sarcoma virus-transformed cells produce transforming growth factors.
Salomon DS; Zwiebel JA; Noda M; Bassin RH
J Cell Physiol; 1984 Oct; 121(1):22-30. PubMed ID: 6090475
[TBL] [Abstract][Full Text] [Related]
19. Potent SHC tyrosine phosphorylation by epidermal growth factor at low receptor density or in the absence of receptor autophosphorylation sites.
Soler C; Alvarez CV; Beguinot L; Carpenter G
Oncogene; 1994 Aug; 9(8):2207-15. PubMed ID: 8036006
[TBL] [Abstract][Full Text] [Related]
20. Down-modulation of EGF receptors in cells transformed by the src oncogene.
Wasilenko WJ; Shawver LK; Weber MJ
J Cell Physiol; 1987 Jun; 131(3):450-7. PubMed ID: 3036890
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
