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2. A shift in the requirement for CD4+ T cells in the generation of AKR/Gross MuLV-specific CTL in AKR.H-2b:Fv-1b mice occurs prior to the onset of age-dependent CTL nonresponsiveness. Smith PM; Rich RF; Green WR Cell Immunol; 1997 Feb; 175(2):189-98. PubMed ID: 9023425 [TBL] [Abstract][Full Text] [Related]
3. Mechanism of nonresponsiveness to AKR/Gross leukemia virus in AKR.H-2b:Fv-1b mice. An analysis of precursor cytotoxic T lymphocyte frequencies in young versus moderately aged mice. Wegmann KW; McMaster JS; Green WR J Immunol; 1991 Apr; 146(7):2469-77. PubMed ID: 2005406 [TBL] [Abstract][Full Text] [Related]
4. Induction of anti-AKR/gross virus cytolytic T lymphocytes in AKR.H-2b:Fv-1b congenic mice: age-dependent conversion to a nonresponder phenotype. Green WR J Immunol; 1987 Mar; 138(5):1602-6. PubMed ID: 3027182 [TBL] [Abstract][Full Text] [Related]
5. Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. II. Negative control by the Fv-1 locus in AKR mice of responder H-2b haplotype. Green WR J Immunol; 1984 May; 132(5):2665-71. PubMed ID: 6425410 [TBL] [Abstract][Full Text] [Related]
6. Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. I. H-2-encoded dominant gene control. Green WR J Immunol; 1984 May; 132(5):2658-64. PubMed ID: 6425409 [TBL] [Abstract][Full Text] [Related]
7. Generation of anti-AKR/gross murine leukemia virus cytotoxic T lymphocytes (CTL). An analysis of precursor CTL frequencies in the AKR.H-2b and C57BL/6 mouse strains. Wegmann KW; Rich RF; Green WR J Immunol; 1992 Sep; 149(5):1593-8. PubMed ID: 1324278 [TBL] [Abstract][Full Text] [Related]
8. Cell surface expression of cytotoxic T lymphocyte-defined, AKR/Gross leukemia virus-associated tumor antigens by normal AKR.H-2b splenic B cells. Green WR J Immunol; 1983 Dec; 131(6):3078-84. PubMed ID: 6605998 [TBL] [Abstract][Full Text] [Related]
9. Expression of CTL-defined, AKR/Gross retrovirus-associated tumor antigens by normal spleen cells: control by Fv-1, H-2, and proviral genes and effect on antiviral CTL generation. Green WR J Immunol; 1986 Jan; 136(1):308-12. PubMed ID: 2999246 [TBL] [Abstract][Full Text] [Related]
10. Nonresponsiveness of AKR.H-2b congenic mice for anti-AKR/Gross MuLV CTL responses: involvement of inhibitory cells as defined by adoptive transfer experiments. Rich RF; Green WR Cell Immunol; 1995 Jan; 160(1):139-51. PubMed ID: 7842479 [TBL] [Abstract][Full Text] [Related]
11. AKR.H-2b lymphocytes inhibit the secondary in vitro cytotoxic T-lymphocyte response of primed responder cells to AKR/Gross murine leukemia virus-induced tumor cell stimulation. Rich RF; Green WR J Virol; 1996 Jan; 70(1):402-14. PubMed ID: 8523554 [TBL] [Abstract][Full Text] [Related]
12. Induction of anti-MuLV cytotoxic T lymphocytes in the AKR.H-2b and AKR.H-2b:Fv-1b mouse strains. Wegmann KW; Blank KJ; Green WR Cell Immunol; 1988 May; 113(2):308-19. PubMed ID: 2834071 [TBL] [Abstract][Full Text] [Related]
13. Impaired generation of anti-AKR/Gross murine leukemia virus cytotoxic T lymphocytes in mice experimentally infected with MuLV. Coppola MA; Green WR; Rich RF Viral Immunol; 1996; 9(2):107-19. PubMed ID: 8822627 [TBL] [Abstract][Full Text] [Related]
14. Spontaneous in vivo retrovirus-infected T and B cells, but not dendritic cells, mediate antigen-specific Fas ligand/Fas-dependent apoptosis of anti-retroviral CTL. Rich RF; Cook WJ; Green WR Virology; 2006 Mar; 346(2):287-300. PubMed ID: 16337984 [TBL] [Abstract][Full Text] [Related]
15. Clonal heterogeneity of anti-AKR/gross leukemia virus cytotoxic T lymphocytes. Evidence for two distinct antigen systems. Azuma H; Phillips JD; Green WR J Immunol; 1987 Oct; 139(7):2464-73. PubMed ID: 2821116 [TBL] [Abstract][Full Text] [Related]
16. Resistance to cellular immune response in AKR leukemias. Schäfer A; Schmidt W Eur J Immunol; 1986 Jul; 16(7):753-9. PubMed ID: 2424771 [TBL] [Abstract][Full Text] [Related]
17. The specificity of H-2-restricted cytotoxic T lymphocytes directed to AKR/Gross leukemia virus-induced tumors. III. Coordinate alterations in viral gp70 antigen expression and restoration of CTL-susceptibility to insusceptible variant tumors. Manjunath R; Graziano RF; Green WR J Immunol; 1986 Mar; 136(6):2271-9. PubMed ID: 3005407 [TBL] [Abstract][Full Text] [Related]
18. The specificity of H-2-restricted cytotoxic T lymphocytes directed to AKR/Gross leukemia virus-induced tumors. I. Isolation of a selectively resistant variant tumor subclone. Green WR Eur J Immunol; 1983 Nov; 13(11):863-70. PubMed ID: 6196206 [TBL] [Abstract][Full Text] [Related]
19. Cytotoxic T lymphocyte responses to the envelope proteins of endogenous ecotropic and mink cytopathic focus-forming murine leukemia viruses in H-2b mice. Coppola MA; Green WR Virology; 1994 Jul; 202(1):500-5. PubMed ID: 7516600 [TBL] [Abstract][Full Text] [Related]
20. Antiviral protection by CD8+ versus CD4+ T cells. CD8+ T cells correlating with cytotoxic activity in vitro are more efficient in antivaccinia virus protection than CD4-dependent IL. Binder D; Kündig TM J Immunol; 1991 Jun; 146(12):4301-7. PubMed ID: 1710246 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]