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6. Cerebral blood flow is reduced by N omega-nitro-L-arginine methyl ester during delayed hypoperfusion in cats. Clavier N; Kirsch JR; Hurn PD; Traystman RJ Am J Physiol; 1994 Jul; 267(1 Pt 2):H174-81. PubMed ID: 8048583 [TBL] [Abstract][Full Text] [Related]
7. Cerebral pressure-flow and metabolic responses to sustained hypoxia: effect of CO2. Yang SP; Bergö GW; Krasney E; Krasney JA J Appl Physiol (1985); 1994 Jan; 76(1):303-13. PubMed ID: 8175522 [TBL] [Abstract][Full Text] [Related]
8. Contribution of nitric oxide to coronary vasodilation during hypercapnic acidosis. Gurevicius J; Salem MR; Metwally AA; Silver JM; Crystal GJ Am J Physiol; 1995 Jan; 268(1 Pt 2):H39-47. PubMed ID: 7530920 [TBL] [Abstract][Full Text] [Related]
9. Nitro-L-arginine attenuates hypercapnic cerebrovasodilation without affecting cerebral metabolism. Iadecola C; Xu X Am J Physiol; 1994 Feb; 266(2 Pt 2):R518-25. PubMed ID: 8141411 [TBL] [Abstract][Full Text] [Related]
10. [The effect of sufentanil on cerebral blood flow, cerebral metabolism and the CO2 reactivity of the cerebral vessels in man]. Stephan H; Gröger P; Weyland A; Hoeft A; Sonntag H Anaesthesist; 1991 Mar; 40(3):153-60. PubMed ID: 1827962 [TBL] [Abstract][Full Text] [Related]
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13. Hypoxic vasodilation does not require nitric oxide (EDRF/NO) synthesis. Vallet B; Curtis SE; Winn MJ; King CE; Chapler CK; Cain SM J Appl Physiol (1985); 1994 Mar; 76(3):1256-61. PubMed ID: 8005870 [TBL] [Abstract][Full Text] [Related]
14. Nitric oxide-dependent and -independent components of cerebrovasodilation elicited by hypercapnia. Iadecola C; Zhang F Am J Physiol; 1994 Feb; 266(2 Pt 2):R546-52. PubMed ID: 7511352 [TBL] [Abstract][Full Text] [Related]
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