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2. [The effect of tri-o-cresyl phosphate on the endocrine system]. HEGYVARY C; CSALAY L Kiserl Orvostud; 1963 Jun; 15():275-9. PubMed ID: 13953324 [No Abstract] [Full Text] [Related]
3. THE INFLUENCE OF THE ANTIDIURETIC HORMONE ON THE RELEASE OF ADRENOCORTICOTROPHIN. CALIGARIS L; CAMINO R; TALEISNIK S Acta Physiol Lat Am; 1963; 13():119-23. PubMed ID: 14098040 [No Abstract] [Full Text] [Related]
4. THE MECHANISM OF INCREASED SODIUM EXCRETION DURING WATER LOADING WITH 2-5 PERCENT DEXTROSE AND VASOPRESSIN. JONES NF; BARRACLOUGH MA; MILLS IH Clin Sci; 1963 Dec; 25():449-57. PubMed ID: 14101494 [No Abstract] [Full Text] [Related]
5. ADRENAL-NEUROHYPOPHYSIAL RELATIONSHIPS IN THE DEHYDRATED RAT. MOSES AM Endocrinology; 1963 Aug; 73():230-6. PubMed ID: 14076203 [No Abstract] [Full Text] [Related]
6. [Effects of the corticotropic hormone and vasopressin on the in vitro secretion of the rat adrenal]. MIALHE-VOLOSS C; KOCH B Ann Endocrinol (Paris); 1962; 23():737-40. PubMed ID: 13935401 [No Abstract] [Full Text] [Related]
7. FERROUS SULFATE POISONING: A REVIEW, CASE SUMMARIES, AND THERAPEUTIC REGIMEN. COVEY TJ J Pediatr; 1964 Feb; 64():218-26. PubMed ID: 14119521 [No Abstract] [Full Text] [Related]
8. ADRENOCORTICAL HYPERFUNCTION AND POTASSIUM METABOLISM IN PATIENTS WITH "NON- ENDOCRINE" TUMORS AND CUSHING'S SYNDROME. PRUNTY FT; BROOKS RV; DUPRE J; GIMLETTE TM; HUTCHINSON JS; MCSWINEY RR; MILLS IH J Clin Endocrinol Metab; 1963 Aug; 23():737-46. PubMed ID: 14059550 [No Abstract] [Full Text] [Related]
9. CHANGES IN THE CENTRAL NERVOUS SYSTEM IN THE CAT AS THE RESULT OF TRI-O-CRESYL PHOSPHATE POISONING. CAVANAGH JB; PATANGIA GN Brain; 1965 Mar; 88():165-80. PubMed ID: 14280272 [No Abstract] [Full Text] [Related]
10. [INDUCED HYPERHYDRATION AS AN ACTIVATING METHOD FOR ENCEPHALOGRAPHY]. VAZQUEZDELMERCADO J; GONZALEZCALDERON JX; NAVASEGURA J Rev Med Hosp Gen (Mex); 1963 Dec; 26():889-98. PubMed ID: 14112593 [No Abstract] [Full Text] [Related]
11. [Aspects of liver damage in human and experimental poisoning with tri-orthocresylphosphate]. D'Antuono G; Coccheri S Minerva Med; 1968 Sep; 59(78):4079-80. PubMed ID: 5686140 [No Abstract] [Full Text] [Related]
12. [Electroencephalographic observations during toxic polyneuritis (caused by tri-o-cresylphosphate)]. PRETOLANI E; SCOTTI M Riv Infort Mal Prof; 1963; 50():357-66. PubMed ID: 13986079 [No Abstract] [Full Text] [Related]
13. THE CONCENTRATION OF VASOPRESSIN IN THE BLOOD AND THE RATE OF URINARY EXCRETION OF THE HORMONE BY PATIENTS WITH CIRRHOSIS OF THE LIVER. LEE J; KERR DN Clin Sci; 1963 Dec; 25():375-84. PubMed ID: 14101486 [No Abstract] [Full Text] [Related]
14. [Effect of vasopressin on the elimination of water load, sodium and potassium in weanling rats]. KRECEK J; DLOUHA H; KRECKOVA J Cesk Fysiol; 1958 Feb; 7(1):30-1. PubMed ID: 13561335 [No Abstract] [Full Text] [Related]
15. EFFECTS OF WALTERINESSIA AEGYPTA VENOM ON BLOOD SODIUM, POTASSIUM AND CATECHOLAMINES, AND URINE 17-KETOSTEROIDS. MOHAMED AH; EL-SEROUGI M; KAMEL A Toxicon; 1964 Oct; 2():103-7. PubMed ID: 14301285 [No Abstract] [Full Text] [Related]
16. PLASMA SODIUM, CALCIUM AND POTASSIUM LEVELS IN RABBITS DURING EXPERIMENTAL ACUTE LEAD POISONING. KOSMIDER S; GRABSKI J; STRADOWSKI J Arch Immunol Ther Exp (Warsz); 1963; 11():303-6. PubMed ID: 14062976 [No Abstract] [Full Text] [Related]
17. [Chronic poisoning the tri-orthocresylphosphate in albino rats with special reference to dental and periodontal tissues]. Montanari G; Rossi A; Scotti S Mondo Odontostomatol; 1968; 10(1):56-70. PubMed ID: 5239723 [No Abstract] [Full Text] [Related]
18. Potentiation by vasopressin of corticotropin release induced by corticotropin-releasing factor. Yates FE; Russell SM; Dallman MF; Hodge GA; McCann SM; Dhariwal AP Endocrinology; 1971 Jan; 88(1):3-15. PubMed ID: 4320769 [No Abstract] [Full Text] [Related]
19. Techniques for the Evaluation of Adrenal Cortical Function by the Use of Adrenocorticotrophin: A Review. Prunty FT J Clin Pathol; 1950 May; 3(2):87-105. PubMed ID: 16810876 [No Abstract] [Full Text] [Related]
20. [RESPONSE TO THE APPLICATION OF CORTICOTROPIN IN HEALTHY PREMATURE INFANTS DURING THE NEONATAL STAGE. 2. MODIFICATIONS IN CHLORIDE, SODIUM AND POTASSIUM LEVELS IN CAPILLARY BLOOD]. JURADOGARCIA E; DULANTOGUTIERREZ E; ALVAREZDELOSCOBOS J; D IAZBENSUSSEN S; AGUADOFLORES C Bol Med Hosp Infant Mex; 1963; 20():693-711. PubMed ID: 14095189 [No Abstract] [Full Text] [Related] [Next] [New Search]