These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
339 related articles for article (PubMed ID: 14642275)
1. FAK deficiency in cells contributing to the basal lamina results in cortical abnormalities resembling congenital muscular dystrophies. Beggs HE; Schahin-Reed D; Zang K; Goebbels S; Nave KA; Gorski J; Jones KR; Sretavan D; Reichardt LF Neuron; 2003 Oct; 40(3):501-14. PubMed ID: 14642275 [TBL] [Abstract][Full Text] [Related]
2. Increase in reelin-positive cells in the marginal zone of Pax6 mutant mouse cortex. Stoykova A; Hatano O; Gruss P; Götz M Cereb Cortex; 2003 Jun; 13(6):560-71. PubMed ID: 12764029 [TBL] [Abstract][Full Text] [Related]
3. Ectopia of meningeal fibroblasts and reactive gliosis in the cerebral cortex of the mouse model of muscle-eye-brain disease. Yang Y; Zhang P; Xiong Y; Li X; Qi Y; Hu H J Comp Neurol; 2007 Dec; 505(5):459-77. PubMed ID: 17924568 [TBL] [Abstract][Full Text] [Related]
4. Breaches of the pial basement membrane and disappearance of the glia limitans during development underlie the cortical lamination defect in the mouse model of muscle-eye-brain disease. Hu H; Yang Y; Eade A; Xiong Y; Qi Y J Comp Neurol; 2007 Mar; 501(1):168-83. PubMed ID: 17206611 [TBL] [Abstract][Full Text] [Related]
5. Downregulation of functional Reelin receptors in projection neurons implies that primary Reelin action occurs at early/premigratory stages. Uchida T; Baba A; Pérez-Martínez FJ; Hibi T; Miyata T; Luque JM; Nakajima K; Hattori M J Neurosci; 2009 Aug; 29(34):10653-62. PubMed ID: 19710317 [TBL] [Abstract][Full Text] [Related]
7. Cajal-Retzius cells in the mouse: transcription factors, neurotransmitters, and birthdays suggest a pallial origin. Hevner RF; Neogi T; Englund C; Daza RA; Fink A Brain Res Dev Brain Res; 2003 Mar; 141(1-2):39-53. PubMed ID: 12644247 [TBL] [Abstract][Full Text] [Related]
8. Zic deficiency in the cortical marginal zone and meninges results in cortical lamination defects resembling those in type II lissencephaly. Inoue T; Ogawa M; Mikoshiba K; Aruga J J Neurosci; 2008 Apr; 28(18):4712-25. PubMed ID: 18448648 [TBL] [Abstract][Full Text] [Related]
10. Breaches of the pial basement membrane and disappearance of the glia limitans during development underlie the cortical lamination defect in the mouse model of muscle-eye-brain disease. Hu H; Yang Y; Eade A; Xiong Y; Qi Y J Comp Neurol; 2007 May; 502(2):168-83. PubMed ID: 17479518 [TBL] [Abstract][Full Text] [Related]
11. Rapid tyrosine phosphorylation of neuronal proteins including tau and focal adhesion kinase in response to amyloid-beta peptide exposure: involvement of Src family protein kinases. Williamson R; Scales T; Clark BR; Gibb G; Reynolds CH; Kellie S; Bird IN; Varndell IM; Sheppard PW; Everall I; Anderton BH J Neurosci; 2002 Jan; 22(1):10-20. PubMed ID: 11756483 [TBL] [Abstract][Full Text] [Related]
12. Origin of the cortical layer I in rodents. Jiménez D; Rivera R; López-Mascaraque L; De Carlos JA Dev Neurosci; 2003; 25(2-4):105-15. PubMed ID: 12966209 [TBL] [Abstract][Full Text] [Related]
13. Birth-date-dependent segregation of the mouse cerebral cortical neurons in reaggregation cultures. Ajioka I; Nakajima K Eur J Neurosci; 2005 Jul; 22(2):331-42. PubMed ID: 16045486 [TBL] [Abstract][Full Text] [Related]
14. Immature astrocytes in Fukuyama congenital muscular dystrophy: an immunohistochemical study. Yamamoto T; Armstrong D; Shibata N; Kanazawa M; Kobayashi M Pediatr Neurol; 1999 Jan; 20(1):31-7. PubMed ID: 10029257 [TBL] [Abstract][Full Text] [Related]