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6. On the origin of the transthyretin Val30Met familial amyloid polyneuropathy. Zaros C; Genin E; Hellman U; Saporta MA; Languille L; Wadington-Cruz M; Suhr O; Misrahi M; Planté-Bordeneuve V Ann Hum Genet; 2008 Jul; 72(Pt 4):478-84. PubMed ID: 18460047 [TBL] [Abstract][Full Text] [Related]
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13. Distinct characteristics of amyloid deposits in early- and late-onset transthyretin Val30Met familial amyloid polyneuropathy. Koike H; Ando Y; Ueda M; Kawagashira Y; Iijima M; Fujitake J; Hayashi M; Yamamoto M; Mukai E; Nakamura T; Katsuno M; Hattori N; Sobue G J Neurol Sci; 2009 Dec; 287(1-2):178-84. PubMed ID: 19709674 [TBL] [Abstract][Full Text] [Related]
14. A Trans-acting Factor May Modify Age at Onset in Familial Amyloid Polyneuropathy ATTRV30M in Portugal. Alves-Ferreira M; Coelho T; Santos D; Sequeiros J; Alonso I; Sousa A; Lemos C Mol Neurobiol; 2018 May; 55(5):3676-3683. PubMed ID: 28527106 [TBL] [Abstract][Full Text] [Related]
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19. Cys-10 mixed disulfide modifications exacerbate transthyretin familial variant amyloidogenicity: a likely explanation for variable clinical expression of amyloidosis and the lack of pathology in C10S/V30M transgenic mice? Zhang Q; Kelly JW Biochemistry; 2005 Jun; 44(25):9079-85. PubMed ID: 15966731 [TBL] [Abstract][Full Text] [Related]
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