292 related articles for article (PubMed ID: 14729970)
1. E2a/Pbx1 induces the rapid proliferation of stem cell factor-dependent murine pro-T cells that cause acute T-lymphoid or myeloid leukemias in mice.
Sykes DB; Kamps MP
Mol Cell Biol; 2004 Feb; 24(3):1256-69. PubMed ID: 14729970
[TBL] [Abstract][Full Text] [Related]
2. Retrovirus-Mediated Expression of E2A-PBX1 Blocks Lymphoid Fate but Permits Retention of Myeloid Potential in Early Hematopoietic Progenitors.
Woodcroft MW; Nanan K; Thompson P; Tyryshkin K; Smith SP; Slany RK; LeBrun DP
PLoS One; 2015; 10(6):e0130495. PubMed ID: 26098938
[TBL] [Abstract][Full Text] [Related]
3. An inhibitory switch derepressed by pbx, hox, and Meis/Prep1 partners regulates DNA-binding by pbx1 and E2a-pbx1 and is dispensable for myeloid immortalization by E2a-pbx1.
Calvo KR; Knoepfler P; McGrath S; Kamps MP
Oncogene; 1999 Dec; 18(56):8033-43. PubMed ID: 10637514
[TBL] [Abstract][Full Text] [Related]
4. E2a/Pbx1 oncogene inhibits terminal differentiation but not myeloid potential of pro-T cells.
Bourette RP; Grasset MF; Mouchiroud G
Oncogene; 2007 Jan; 26(2):234-47. PubMed ID: 16819510
[TBL] [Abstract][Full Text] [Related]
5. Meis1 and pKnox1 bind DNA cooperatively with Pbx1 utilizing an interaction surface disrupted in oncoprotein E2a-Pbx1.
Knoepfler PS; Calvo KR; Chen H; Antonarakis SE; Kamps MP
Proc Natl Acad Sci U S A; 1997 Dec; 94(26):14553-8. PubMed ID: 9405651
[TBL] [Abstract][Full Text] [Related]
6. [Identification of proteins associated with transcription factors HOXA9 and E2A-PBX1 by tandem affinity purification].
Shestakova EA; Boutin M; Bourassa S; Bonneil E; Bijl JJ
Mol Biol (Mosk); 2017; 51(3):490-501. PubMed ID: 28707666
[TBL] [Abstract][Full Text] [Related]
7. Both Pbx1 and E2A-Pbx1 bind the DNA motif ATCAATCAA cooperatively with the products of multiple murine Hox genes, some of which are themselves oncogenes.
Lu Q; Knoepfler PS; Scheele J; Wright DD; Kamps MP
Mol Cell Biol; 1995 Jul; 15(7):3786-95. PubMed ID: 7791786
[TBL] [Abstract][Full Text] [Related]
8. Transformation properties of the E2a-Pbx1 chimeric oncoprotein: fusion with E2a is essential, but the Pbx1 homeodomain is dispensable.
Monica K; LeBrun DP; Dedera DA; Brown R; Cleary ML
Mol Cell Biol; 1994 Dec; 14(12):8304-14. PubMed ID: 7969166
[TBL] [Abstract][Full Text] [Related]
9. E2A-Pbx1, the t(1;19) translocation protein of human pre-B-cell acute lymphocytic leukemia, causes acute myeloid leukemia in mice.
Kamps MP; Baltimore D
Mol Cell Biol; 1993 Jan; 13(1):351-7. PubMed ID: 8093327
[TBL] [Abstract][Full Text] [Related]
10. Oncoprotein E2A-Pbx1 immortalizes a myeloid progenitor in primary marrow cultures without abrogating its factor-dependence.
Kamps MP; Wright DD
Oncogene; 1994 Nov; 9(11):3159-66. PubMed ID: 7936638
[TBL] [Abstract][Full Text] [Related]
11. Identification of cooperative genes for E2A-PBX1 to develop acute lymphoblastic leukemia.
Sera Y; Yamasaki N; Oda H; Nagamachi A; Wolff L; Inukai T; Inaba T; Honda H
Cancer Sci; 2016 Jul; 107(7):890-8. PubMed ID: 27088431
[TBL] [Abstract][Full Text] [Related]
12. DNA-binding by oncoprotein E2a-Pbx1 is important for blocking differentiation but dispensable for fibroblast transformation.
Kamps MP; Wright DD; Lu Q
Oncogene; 1996 Jan; 12(1):19-30. PubMed ID: 8552391
[TBL] [Abstract][Full Text] [Related]
13. Comparative genomics reveals multistep pathogenesis of E2A-PBX1 acute lymphoblastic leukemia.
Duque-Afonso J; Feng J; Scherer F; Lin CH; Wong SH; Wang Z; Iwasaki M; Cleary ML
J Clin Invest; 2015 Sep; 125(9):3667-80. PubMed ID: 26301816
[TBL] [Abstract][Full Text] [Related]
14. Fusion with E2A converts the Pbx1 homeodomain protein into a constitutive transcriptional activator in human leukemias carrying the t(1;19) translocation.
Lu Q; Wright DD; Kamps MP
Mol Cell Biol; 1994 Jun; 14(6):3938-48. PubMed ID: 7910944
[TBL] [Abstract][Full Text] [Related]
15. Bmi-1 regulation of INK4A-ARF is a downstream requirement for transformation of hematopoietic progenitors by E2a-Pbx1.
Smith KS; Chanda SK; Lingbeek M; Ross DT; Botstein D; van Lohuizen M; Cleary ML
Mol Cell; 2003 Aug; 12(2):393-400. PubMed ID: 14536079
[TBL] [Abstract][Full Text] [Related]
16. Selective repression of transcriptional activators by Pbx1 does not require the homeodomain.
Lu Q; Kamps MP
Proc Natl Acad Sci U S A; 1996 Jan; 93(1):470-4. PubMed ID: 8552663
[TBL] [Abstract][Full Text] [Related]
17. Different molecular consequences of the 1;19 chromosomal translocation in childhood B-cell precursor acute lymphoblastic leukemia.
Privitera E; Kamps MP; Hayashi Y; Inaba T; Shapiro LH; Raimondi SC; Behm F; Hendershot L; Carroll AJ; Baltimore D
Blood; 1992 Apr; 79(7):1781-8. PubMed ID: 1348433
[TBL] [Abstract][Full Text] [Related]
18. The highest affinity DNA element bound by Pbx complexes in t(1;19) leukemic cells fails to mediate cooperative DNA-binding or cooperative transactivation by E2a-Pbx1 and class I Hox proteins - evidence for selective targetting of E2a-Pbx1 to a subset of Pbx-recognition elements.
Knoepfler PS; Kamps MP
Oncogene; 1997 May; 14(21):2521-31. PubMed ID: 9191052
[TBL] [Abstract][Full Text] [Related]
19. Oncogenic homeodomain transcription factor E2A-Pbx1 activates a novel WNT gene in pre-B acute lymphoblastoid leukemia.
McWhirter JR; Neuteboom ST; Wancewicz EV; Monia BP; Downing JR; Murre C
Proc Natl Acad Sci U S A; 1999 Sep; 96(20):11464-9. PubMed ID: 10500199
[TBL] [Abstract][Full Text] [Related]
20. Chimeric oncoprotein E2a-Pbx1 induces apoptosis of hematopoietic cells by a p53-independent mechanism that is suppressed by Bcl-2.
Smith KS; Jacobs Y; Chang CP; Cleary ML
Oncogene; 1997 Jun; 14(24):2917-26. PubMed ID: 9205098
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]