These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

144 related articles for article (PubMed ID: 14973064)

  • 1. Normal cells control the growth of neighboring transformed cells independent of gap junctional communication and SRC activity.
    Alexander DB; Ichikawa H; Bechberger JF; Valiunas V; Ohki M; Naus CC; Kunimoto T; Tsuda H; Miller WT; Goldberg GS
    Cancer Res; 2004 Feb; 64(4):1347-58. PubMed ID: 14973064
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Nontransformed cells can normalize gap junctional communication with transformed cells.
    Valiunas V; Bechberger JF; Naus CC; Brink PR; Goldberg GS
    Biochem Biophys Res Commun; 2005 Jul; 333(1):174-9. PubMed ID: 15936725
    [TBL] [Abstract][Full Text] [Related]  

  • 3. SRC utilizes Cas to block gap junctional communication mediated by connexin43.
    Shen Y; Khusial PR; Li X; Ichikawa H; Moreno AP; Goldberg GS
    J Biol Chem; 2007 Jun; 282(26):18914-21. PubMed ID: 17488714
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Phosphorylation of connexin43 induced by Src: regulation of gap junctional communication between transformed cells.
    Pahujaa M; Anikin M; Goldberg GS
    Exp Cell Res; 2007 Dec; 313(20):4083-90. PubMed ID: 17956757
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Mind the gap; regulation of gap junctional, intercellular communication by the SRC oncogene product and its effectors.
    Geletu M; Trotman-Grant A; Raptis L
    Anticancer Res; 2012 Oct; 32(10):4245-50. PubMed ID: 23060544
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Heterocellular N-cadherin junctions enable nontransformed cells to inhibit the growth of adjacent transformed cells.
    Sheehan SA; Retzbach EP; Shen Y; Krishnan H; Goldberg GS
    Cell Commun Signal; 2022 Feb; 20(1):19. PubMed ID: 35177067
    [TBL] [Abstract][Full Text] [Related]  

  • 7. In vitro growth inhibition of neoplastically transformed cells by non-transformed cells: requirement for gap junctional intercellular communication.
    Esinduy CB; Chang CC; Trosko JE; Ruch RJ
    Carcinogenesis; 1995 Apr; 16(4):915-21. PubMed ID: 7728974
    [TBL] [Abstract][Full Text] [Related]  

  • 8. PI3K/Akt signaling is involved in the disruption of gap junctional communication caused by v-Src and TNF-α.
    Ito S; Hyodo T; Hasegawa H; Yuan H; Hamaguchi M; Senga T
    Biochem Biophys Res Commun; 2010 Sep; 400(2):230-5. PubMed ID: 20727856
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Maintaining connexin43 gap junctional communication in v-Src cells does not alter growth properties associated with the transformed phenotype.
    Warn-Cramer BJ; Lin R; Martyn K; Guyette CV; Lau AF
    Cell Commun Adhes; 2003; 10(4-6):299-303. PubMed ID: 14681032
    [TBL] [Abstract][Full Text] [Related]  

  • 10. A connexin 43 antisense vector reduces the ability of normal cells to inhibit the foci formation of transformed cells.
    Goldberg GS; Martyn KD; Lau AF
    Mol Carcinog; 1994 Oct; 11(2):106-14. PubMed ID: 7916992
    [TBL] [Abstract][Full Text] [Related]  

  • 11. v-Src requires Ras signaling for the suppression of gap junctional intercellular communication.
    Ito S; Ito Y; Senga T; Hattori S; Matsuo S; Hamaguchi M
    Oncogene; 2006 Apr; 25(16):2420-4. PubMed ID: 16301992
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Gap junctional communication and neoplastic transformation.
    Hotz-Wagenblatt A; Shalloway D
    Crit Rev Oncog; 1993; 4(5):541-58. PubMed ID: 8241323
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Effects of cadherin mediated contact normalization on oncogenic Src kinase mediated gene expression and protein phosphorylation.
    Nicoletto RE; Holdcraft CJ; Yin AC; Retzbach EP; Sheehan SA; Greenspan AA; Laugier CM; Trama J; Zhao C; Zheng H; Goldberg GS
    Sci Rep; 2024 Oct; 14(1):23942. PubMed ID: 39397108
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Lack of intercellular communication between chemically transformed and surrounding nontransformed BALB/c 3T3 cells.
    Enomoto T; Yamasaki H
    Cancer Res; 1984 Nov; 44(11):5200-3. PubMed ID: 6488180
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Effects of SRC and STAT3 upon gap junctional, intercellular communication in lung cancer lines.
    Geletu M; Guy S; Raptis L
    Anticancer Res; 2013 Oct; 33(10):4401-10. PubMed ID: 24123009
    [TBL] [Abstract][Full Text] [Related]  

  • 16. p130gag-fps disrupts gap junctional communication and induces phosphorylation of connexin43 in a manner similar to that of pp60v-src.
    Kurata WE; Lau AF
    Oncogene; 1994 Jan; 9(1):329-35. PubMed ID: 8302599
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Increased connexin-43 and gap junctional communication correlate with altered phenotypic characteristics of cells overexpressing the receptor for hyaluronic acid-mediated motility.
    Nagy JI; Hossain MZ; Lynn BD; Curpen GE; Yang S; Turley EA
    Cell Growth Differ; 1996 Jun; 7(6):745-51. PubMed ID: 8780888
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Selective lack of intercellular communication between transformed and nontransformed cells as a common property of chemical and oncogene transformation of BALB/c 3T3 cells.
    Yamasaki H; Hollstein M; Mesnil M; Martel N; Aguelon AM
    Cancer Res; 1987 Nov; 47(21):5658-64. PubMed ID: 3311356
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Regulation of Cx43 gap junctions: the gatekeeper and the password.
    Hossain MZ; Boynton AL
    Sci STKE; 2000 Oct; 2000(54):pe1. PubMed ID: 11752614
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Further evidence for the involvement of gap-junctional intercellular communication in induction and maintenance of transformed foci in BALB/c 3T3 cells.
    Yamasaki H; Katoh F
    Cancer Res; 1988 Jun; 48(12):3490-5. PubMed ID: 2836057
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 8.