232 related articles for article (PubMed ID: 14975238)
1. SOCS3 is a critical physiological negative regulator of G-CSF signaling and emergency granulopoiesis.
Croker BA; Metcalf D; Robb L; Wei W; Mifsud S; DiRago L; Cluse LA; Sutherland KD; Hartley L; Williams E; Zhang JG; Hilton DJ; Nicola NA; Alexander WS; Roberts AW
Immunity; 2004 Feb; 20(2):153-65. PubMed ID: 14975238
[TBL] [Abstract][Full Text] [Related]
2. STAT3 is a negative regulator of granulopoiesis but is not required for G-CSF-dependent differentiation.
Lee CK; Raz R; Gimeno R; Gertner R; Wistinghausen B; Takeshita K; DePinho RA; Levy DE
Immunity; 2002 Jul; 17(1):63-72. PubMed ID: 12150892
[TBL] [Abstract][Full Text] [Related]
3. SOCS3 is a physiological negative regulator for granulopoiesis and granulocyte colony-stimulating factor receptor signaling.
Kimura A; Kinjyo I; Matsumura Y; Mori H; Mashima R; Harada M; Chien KR; Yasukawa H; Yoshimura A
J Biol Chem; 2004 Feb; 279(8):6905-10. PubMed ID: 14699146
[TBL] [Abstract][Full Text] [Related]
4. Distinct activities of suppressor of cytokine signaling (SOCS) proteins and involvement of the SOCS box in controlling G-CSF signaling.
van de Geijn GJ; Gits J; Touw IP
J Leukoc Biol; 2004 Jul; 76(1):237-44. PubMed ID: 15107455
[TBL] [Abstract][Full Text] [Related]
5. Socs3 maintains the specificity of biological responses to cytokine signals during granulocyte and macrophage differentiation.
Croker BA; Mielke LA; Wormald S; Metcalf D; Kiu H; Alexander WS; Hilton DJ; Roberts AW
Exp Hematol; 2008 Jul; 36(7):786-98. PubMed ID: 18400361
[TBL] [Abstract][Full Text] [Related]
6. Roles of Stat3 and ERK in G-CSF signaling.
Kamezaki K; Shimoda K; Numata A; Haro T; Kakumitsu H; Yoshie M; Yamamoto M; Takeda K; Matsuda T; Akira S; Ogawa K; Harada M
Stem Cells; 2005 Feb; 23(2):252-63. PubMed ID: 15671148
[TBL] [Abstract][Full Text] [Related]
7. Involvement of casein kinase Iepsilon in cytokine-induced granulocytic differentiation.
Okamura A; Iwata N; Nagata A; Tamekane A; Shimoyama M; Gomyo H; Yakushijin K; Urahama N; Hamaguchi M; Fukui C; Chihara K; Ito M; Matsui T
Blood; 2004 Apr; 103(8):2997-3004. PubMed ID: 15070676
[TBL] [Abstract][Full Text] [Related]
8. Identification of CCR2, flotillin, and gp49B genes as new G-CSF targets during neutrophilic differentiation.
Iida S; Kohro T; Kodama T; Nagata S; Fukunaga R
J Leukoc Biol; 2005 Aug; 78(2):481-90. PubMed ID: 15894583
[TBL] [Abstract][Full Text] [Related]
9. Signaling mechanisms coupled to tyrosines in the granulocyte colony-stimulating factor receptor orchestrate G-CSF-induced expansion of myeloid progenitor cells.
Hermans MH; van de Geijn GJ; Antonissen C; Gits J; van Leeuwen D; Ward AC; Touw IP
Blood; 2003 Apr; 101(7):2584-90. PubMed ID: 12468431
[TBL] [Abstract][Full Text] [Related]
10. G-CSF receptor truncations found in SCN/AML relieve SOCS3-controlled inhibition of STAT5 but leave suppression of STAT3 intact.
van de Geijn GJ; Gits J; Aarts LH; Heijmans-Antonissen C; Touw IP
Blood; 2004 Aug; 104(3):667-74. PubMed ID: 15069015
[TBL] [Abstract][Full Text] [Related]
11. The SOCS box of suppressor of cytokine signaling-3 contributes to the control of G-CSF responsiveness in vivo.
Boyle K; Egan P; Rakar S; Willson TA; Wicks IP; Metcalf D; Hilton DJ; Nicola NA; Alexander WS; Roberts AW; Robb L
Blood; 2007 Sep; 110(5):1466-74. PubMed ID: 17510322
[TBL] [Abstract][Full Text] [Related]
12. STAT3 governs distinct pathways in emergency granulopoiesis and mature neutrophils.
Panopoulos AD; Zhang L; Snow JW; Jones DM; Smith AM; El Kasmi KC; Liu F; Goldsmith MA; Link DC; Murray PJ; Watowich SS
Blood; 2006 Dec; 108(12):3682-90. PubMed ID: 16888100
[TBL] [Abstract][Full Text] [Related]
13. Granulocyte colony-stimulating factor activation of Stat3 alpha and Stat3 beta in immature normal and leukemic human myeloid cells.
Chakraborty A; White SM; Schaefer TS; Ball ED; Dyer KF; Tweardy DJ
Blood; 1996 Oct; 88(7):2442-9. PubMed ID: 8839834
[TBL] [Abstract][Full Text] [Related]
14. STAT-3 activation is required for normal G-CSF-dependent proliferation and granulocytic differentiation.
McLemore ML; Grewal S; Liu F; Archambault A; Poursine-Laurent J; Haug J; Link DC
Immunity; 2001 Feb; 14(2):193-204. PubMed ID: 11239451
[TBL] [Abstract][Full Text] [Related]
15. IFNγ induces monopoiesis and inhibits neutrophil development during inflammation.
de Bruin AM; Libregts SF; Valkhof M; Boon L; Touw IP; Nolte MA
Blood; 2012 Feb; 119(6):1543-54. PubMed ID: 22117048
[TBL] [Abstract][Full Text] [Related]
16. STAT3 controls myeloid progenitor growth during emergency granulopoiesis.
Zhang H; Nguyen-Jackson H; Panopoulos AD; Li HS; Murray PJ; Watowich SS
Blood; 2010 Oct; 116(14):2462-71. PubMed ID: 20581311
[TBL] [Abstract][Full Text] [Related]
17. PLCγ2 and PKC are important to myeloid lineage commitment triggered by M-SCF and G-CSF.
Barbosa CM; Bincoletto C; Barros CC; Ferreira AT; Paredes-Gamero EJ
J Cell Biochem; 2014 Jan; 115(1):42-51. PubMed ID: 24038146
[TBL] [Abstract][Full Text] [Related]
18. Inflammation triggers emergency granulopoiesis through a density-dependent feedback mechanism.
Cain DW; Snowden PB; Sempowski GD; Kelsoe G
PLoS One; 2011; 6(5):e19957. PubMed ID: 21655273
[TBL] [Abstract][Full Text] [Related]
19. STAT3-mediated differentiation and survival and of myeloid cells in response to granulocyte colony-stimulating factor: role for the cyclin-dependent kinase inhibitor p27(Kip1).
de Koning JP; Soede-Bobok AA; Ward AC; Schelen AM; Antonissen C; van Leeuwen D; Löwenberg B; Touw IP
Oncogene; 2000 Jul; 19(29):3290-8. PubMed ID: 10918585
[TBL] [Abstract][Full Text] [Related]
20. Increased G-CSF responsiveness of bone marrow cells from hematopoietic cell phosphatase deficient viable motheaten mice.
Tapley P; Shevde NK; Schweitzer PA; Gallina M; Christianson SW; Lin IL; Stein RB; Shultz LD; Rosen J; Lamb P
Exp Hematol; 1997 Feb; 25(2):122-31. PubMed ID: 9015212
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
