BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

631 related articles for article (PubMed ID: 15044322)

  • 1. Cardiomyocyte-specific overexpression of nitric oxide synthase 3 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction.
    Janssens S; Pokreisz P; Schoonjans L; Pellens M; Vermeersch P; Tjwa M; Jans P; Scherrer-Crosbie M; Picard MH; Szelid Z; Gillijns H; Van de Werf F; Collen D; Bloch KD
    Circ Res; 2004 May; 94(9):1256-62. PubMed ID: 15044322
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Endothelial nitric oxide synthase limits left ventricular remodeling after myocardial infarction in mice.
    Scherrer-Crosbie M; Ullrich R; Bloch KD; Nakajima H; Nasseri B; Aretz HT; Lindsey ML; Vançon AC; Huang PL; Lee RT; Zapol WM; Picard MH
    Circulation; 2001 Sep; 104(11):1286-91. PubMed ID: 11551881
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Cardiomyocyte-restricted restoration of nitric oxide synthase 3 attenuates left ventricular remodeling after chronic pressure overload.
    Buys ES; Raher MJ; Blake SL; Neilan TG; Graveline AR; Passeri JJ; Llano M; Perez-Sanz TM; Ichinose F; Janssens S; Zapol WM; Picard MH; Bloch KD; Scherrer-Crosbie M
    Am J Physiol Heart Circ Physiol; 2007 Jul; 293(1):H620-7. PubMed ID: 17416602
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Disruption of nitric oxide synthase 3 protects against the cardiac injury, dysfunction, and mortality induced by doxorubicin.
    Neilan TG; Blake SL; Ichinose F; Raher MJ; Buys ES; Jassal DS; Furutani E; Perez-Sanz TM; Graveline A; Janssens SP; Picard MH; Scherrer-Crosbie M; Bloch KD
    Circulation; 2007 Jul; 116(5):506-14. PubMed ID: 17638931
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Cardiomyocyte-restricted overexpression of endothelial nitric oxide synthase (NOS3) attenuates beta-adrenergic stimulation and reinforces vagal inhibition of cardiac contraction.
    Massion PB; Dessy C; Desjardins F; Pelat M; Havaux X; Belge C; Moulin P; Guiot Y; Feron O; Janssens S; Balligand JL
    Circulation; 2004 Oct; 110(17):2666-72. PubMed ID: 15492314
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Pressure overload-induced LV hypertrophy and dysfunction in mice are exacerbated by congenital NOS3 deficiency.
    Ichinose F; Bloch KD; Wu JC; Hataishi R; Aretz HT; Picard MH; Scherrer-Crosbie M
    Am J Physiol Heart Circ Physiol; 2004 Mar; 286(3):H1070-5. PubMed ID: 14644766
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Cardiomyocyte-specific overexpression of nitric oxide synthase 3: impact on left ventricular function and myocardial infarction.
    Bloch KD; Janssens S
    Trends Cardiovasc Med; 2005 Oct; 15(7):249-53. PubMed ID: 16226679
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Modulation of in vivo cardiac function by myocyte-specific nitric oxide synthase-3.
    Champion HC; Georgakopoulos D; Takimoto E; Isoda T; Wang Y; Kass DA
    Circ Res; 2004 Mar; 94(5):657-63. PubMed ID: 14752030
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice.
    Matsushima S; Ide T; Yamato M; Matsusaka H; Hattori F; Ikeuchi M; Kubota T; Sunagawa K; Hasegawa Y; Kurihara T; Oikawa S; Kinugawa S; Tsutsui H
    Circulation; 2006 Apr; 113(14):1779-86. PubMed ID: 16585391
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Statin-induced improvement of endothelial progenitor cell mobilization, myocardial neovascularization, left ventricular function, and survival after experimental myocardial infarction requires endothelial nitric oxide synthase.
    Landmesser U; Engberding N; Bahlmann FH; Schaefer A; Wiencke A; Heineke A; Spiekermann S; Hilfiker-Kleiner D; Templin C; Kotlarz D; Mueller M; Fuchs M; Hornig B; Haller H; Drexler H
    Circulation; 2004 Oct; 110(14):1933-9. PubMed ID: 15466656
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Inhibition of endogenous Mst1 prevents apoptosis and cardiac dysfunction without affecting cardiac hypertrophy after myocardial infarction.
    Odashima M; Usui S; Takagi H; Hong C; Liu J; Yokota M; Sadoshima J
    Circ Res; 2007 May; 100(9):1344-52. PubMed ID: 17395874
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Overexpression of glutathione peroxidase prevents left ventricular remodeling and failure after myocardial infarction in mice.
    Shiomi T; Tsutsui H; Matsusaka H; Murakami K; Hayashidani S; Ikeuchi M; Wen J; Kubota T; Utsumi H; Takeshita A
    Circulation; 2004 Feb; 109(4):544-9. PubMed ID: 14744974
    [TBL] [Abstract][Full Text] [Related]  

  • 13. MCIP1 overexpression suppresses left ventricular remodeling and sustains cardiac function after myocardial infarction.
    van Rooij E; Doevendans PA; Crijns HJ; Heeneman S; Lips DJ; van Bilsen M; Williams RS; Olson EN; Bassel-Duby R; Rothermel BA; De Windt LJ
    Circ Res; 2004 Feb; 94(3):e18-26. PubMed ID: 14739160
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Circulating NOS3 modulates left ventricular remodeling following reperfused myocardial infarction.
    Gorressen S; Stern M; van de Sandt AM; Cortese-Krott MM; Ohlig J; Rassaf T; Gödecke A; Fischer JW; Heusch G; Merx MW; Kelm M
    PLoS One; 2015; 10(4):e0120961. PubMed ID: 25875863
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Mice lacking inducible nitric oxide synthase have improved left ventricular contractile function and reduced apoptotic cell death late after myocardial infarction.
    Sam F; Sawyer DB; Xie Z; Chang DL; Ngoy S; Brenner DA; Siwik DA; Singh K; Apstein CS; Colucci WS
    Circ Res; 2001 Aug; 89(4):351-6. PubMed ID: 11509452
    [TBL] [Abstract][Full Text] [Related]  

  • 16. nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction.
    Dawson D; Lygate CA; Zhang MH; Hulbert K; Neubauer S; Casadei B
    Circulation; 2005 Dec; 112(24):3729-37. PubMed ID: 16344403
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Cardiomyocyte-specific overexpression of oestrogen receptor β improves survival and cardiac function after myocardial infarction in female and male mice.
    Schuster I; Mahmoodzadeh S; Dworatzek E; Jaisser F; Messaoudi S; Morano I; Regitz-Zagrosek V
    Clin Sci (Lond); 2016 Mar; 130(5):365-76. PubMed ID: 26608078
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Enhanced expression of β3-adrenoceptors in cardiac myocytes attenuates neurohormone-induced hypertrophic remodeling through nitric oxide synthase.
    Belge C; Hammond J; Dubois-Deruy E; Manoury B; Hamelet J; Beauloye C; Markl A; Pouleur AC; Bertrand L; Esfahani H; Jnaoui K; Götz KR; Nikolaev VO; Vanderper A; Herijgers P; Lobysheva I; Iaccarino G; Hilfiker-Kleiner D; Tavernier G; Langin D; Dessy C; Balligand JL
    Circulation; 2014 Jan; 129(4):451-62. PubMed ID: 24190960
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Nitric oxide mediates benefits of angiotensin II type 2 receptor overexpression during post-infarct remodeling.
    Bove CM; Yang Z; Gilson WD; Epstein FH; French BA; Berr SS; Bishop SP; Matsubara H; Carey RM; Kramer CM
    Hypertension; 2004 Mar; 43(3):680-5. PubMed ID: 14732725
    [TBL] [Abstract][Full Text] [Related]  

  • 20. The angiotensin II type 2 receptor and improved adjacent region function post-MI.
    Bove CM; Gilson WD; Scott CD; Epstein FH; Yang Z; Dimaria JM; Berr SS; French BA; Bishop SP; Kramer CM
    J Cardiovasc Magn Reson; 2005; 7(2):459-64. PubMed ID: 15881529
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 32.