These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
1703 related articles for article (PubMed ID: 15162010)
1. Mechanisms of DNA double strand break repair and chromosome aberration formation. Iliakis G; Wang H; Perrault AR; Boecker W; Rosidi B; Windhofer F; Wu W; Guan J; Terzoudi G; Pantelias G Cytogenet Genome Res; 2004; 104(1-4):14-20. PubMed ID: 15162010 [TBL] [Abstract][Full Text] [Related]
2. Biochemical evidence for Ku-independent backup pathways of NHEJ. Wang H; Perrault AR; Takeda Y; Qin W; Wang H; Iliakis G Nucleic Acids Res; 2003 Sep; 31(18):5377-88. PubMed ID: 12954774 [TBL] [Abstract][Full Text] [Related]
3. Genetic evidence for the involvement of DNA ligase IV in the DNA-PK-dependent pathway of non-homologous end joining in mammalian cells. Wang H; Zeng ZC; Perrault AR; Cheng X; Qin W; Iliakis G Nucleic Acids Res; 2001 Apr; 29(8):1653-60. PubMed ID: 11292837 [TBL] [Abstract][Full Text] [Related]
4. Radiation-induced genomic rearrangements formed by nonhomologous end-joining of DNA double-strand breaks. Rothkamm K; Kühne M; Jeggo PA; Löbrich M Cancer Res; 2001 May; 61(10):3886-93. PubMed ID: 11358801 [TBL] [Abstract][Full Text] [Related]
5. Backup pathways of NHEJ are suppressed by DNA-PK. Perrault R; Wang H; Wang M; Rosidi B; Iliakis G J Cell Biochem; 2004 Jul; 92(4):781-94. PubMed ID: 15211575 [TBL] [Abstract][Full Text] [Related]
6. DNA-dependent protein kinase stimulates an independently active, nonhomologous, end-joining apparatus. DiBiase SJ; Zeng ZC; Chen R; Hyslop T; Curran WJ; Iliakis G Cancer Res; 2000 Mar; 60(5):1245-53. PubMed ID: 10728683 [TBL] [Abstract][Full Text] [Related]
7. Severe combined immunodeficient cells expressing mutant hRAD54 exhibit a marked DNA double-strand break repair and error-prone chromosome repair defect. Pluth JM; Fried LM; Kirchgessner CU Cancer Res; 2001 Mar; 61(6):2649-55. PubMed ID: 11289143 [TBL] [Abstract][Full Text] [Related]
8. Nonhomologous end-joining of ionizing radiation-induced DNA double-stranded breaks in human tumor cells deficient in BRCA1 or BRCA2. Wang H; Zeng ZC; Bui TA; DiBiase SJ; Qin W; Xia F; Powell SN; Iliakis G Cancer Res; 2001 Jan; 61(1):270-7. PubMed ID: 11196174 [TBL] [Abstract][Full Text] [Related]
10. Efficient rejoining of radiation-induced DNA double-strand breaks in vertebrate cells deficient in genes of the RAD52 epistasis group. Wang H; Zeng ZC; Bui TA; Sonoda E; Takata M; Takeda S; Iliakis G Oncogene; 2001 Apr; 20(18):2212-24. PubMed ID: 11402316 [TBL] [Abstract][Full Text] [Related]
11. Non-homologous end joining is the responsible pathway for the repair of fludarabine-induced DNA double strand breaks in mammalian cells. de Campos-Nebel M; Larripa I; González-Cid M Mutat Res; 2008 Nov; 646(1-2):8-16. PubMed ID: 18812179 [TBL] [Abstract][Full Text] [Related]
12. Marked contribution of alternative end-joining to chromosome-translocation-formation by stochastically induced DNA double-strand-breaks in G2-phase human cells. Soni A; Siemann M; Pantelias GE; Iliakis G Mutat Res Genet Toxicol Environ Mutagen; 2015 Nov; 793():2-8. PubMed ID: 26520366 [TBL] [Abstract][Full Text] [Related]
13. PARP-1 and Ku compete for repair of DNA double strand breaks by distinct NHEJ pathways. Wang M; Wu W; Wu W; Rosidi B; Zhang L; Wang H; Iliakis G Nucleic Acids Res; 2006; 34(21):6170-82. PubMed ID: 17088286 [TBL] [Abstract][Full Text] [Related]
14. Repair of radiation induced DNA double strand breaks by backup NHEJ is enhanced in G2. Wu W; Wang M; Wu W; Singh SK; Mussfeldt T; Iliakis G DNA Repair (Amst); 2008 Feb; 7(2):329-38. PubMed ID: 18155970 [TBL] [Abstract][Full Text] [Related]
15. Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells. Fattah F; Lee EH; Weisensel N; Wang Y; Lichter N; Hendrickson EA PLoS Genet; 2010 Feb; 6(2):e1000855. PubMed ID: 20195511 [TBL] [Abstract][Full Text] [Related]
16. DNA-dependent protein kinase and XRCC4-DNA ligase IV mobilization in the cell in response to DNA double strand breaks. Drouet J; Delteil C; Lefrançois J; Concannon P; Salles B; Calsou P J Biol Chem; 2005 Feb; 280(8):7060-9. PubMed ID: 15520013 [TBL] [Abstract][Full Text] [Related]
17. Different types of V(D)J recombination and end-joining defects in DNA double-strand break repair mutant mammalian cells. Verkaik NS; Esveldt-van Lange RE; van Heemst D; Brüggenwirth HT; Hoeijmakers JH; Zdzienicka MZ; van Gent DC Eur J Immunol; 2002 Mar; 32(3):701-9. PubMed ID: 11870614 [TBL] [Abstract][Full Text] [Related]
18. Induction and repair of DNA double strand breaks: the increasing spectrum of non-homologous end joining pathways. Mladenov E; Iliakis G Mutat Res; 2011 Jun; 711(1-2):61-72. PubMed ID: 21329706 [TBL] [Abstract][Full Text] [Related]
19. The influence of heterochromatin on DNA double strand break repair: Getting the strong, silent type to relax. Goodarzi AA; Jeggo P; Lobrich M DNA Repair (Amst); 2010 Dec; 9(12):1273-82. PubMed ID: 21036673 [TBL] [Abstract][Full Text] [Related]
20. After double-strand break induction by UV-A, homologous recombination and nonhomologous end joining cooperate at the same DSB if both systems are available. Rapp A; Greulich KO J Cell Sci; 2004 Oct; 117(Pt 21):4935-45. PubMed ID: 15367581 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]