235 related articles for article (PubMed ID: 15220418)
1. Characterization of the amino acid residues of sendai virus C protein that are critically involved in its interferon antagonism and RNA synthesis down-regulation.
Kato A; Cortese-Grogan C; Moyer SA; Sugahara F; Sakaguchi T; Kubota T; Otsuki N; Kohase M; Tashiro M; Nagai Y
J Virol; 2004 Jul; 78(14):7443-54. PubMed ID: 15220418
[TBL] [Abstract][Full Text] [Related]
2. Y2, the smallest of the Sendai virus C proteins, is fully capable of both counteracting the antiviral action of interferons and inhibiting viral RNA synthesis.
Kato A; Ohnishi Y; Kohase M; Saito S; Tashiro M; Nagai Y
J Virol; 2001 Apr; 75(8):3802-10. PubMed ID: 11264369
[TBL] [Abstract][Full Text] [Related]
3. The amino-terminal half of Sendai virus C protein is not responsible for either counteracting the antiviral action of interferons or down-regulating viral RNA synthesis.
Kato A; Ohnishi Y; Hishiyama M; Kohase M; Saito S; Tashiro M; Nagai Y
J Virol; 2002 Jul; 76(14):7114-24. PubMed ID: 12072511
[TBL] [Abstract][Full Text] [Related]
4. Importance of the anti-interferon capacity of Sendai virus C protein for pathogenicity in mice.
Kato A; Kiyotani K; Kubota T; Yoshida T; Tashiro M; Nagai Y
J Virol; 2007 Apr; 81(7):3264-71. PubMed ID: 17215288
[TBL] [Abstract][Full Text] [Related]
5. All four Sendai Virus C proteins bind Stat1, but only the larger forms also induce its mono-ubiquitination and degradation.
Garcin D; Marq JB; Strahle L; le Mercier P; Kolakofsky D
Virology; 2002 Apr; 295(2):256-65. PubMed ID: 12033784
[TBL] [Abstract][Full Text] [Related]
6. Nipah virus V and W proteins have a common STAT1-binding domain yet inhibit STAT1 activation from the cytoplasmic and nuclear compartments, respectively.
Shaw ML; García-Sastre A; Palese P; Basler CF
J Virol; 2004 Jun; 78(11):5633-41. PubMed ID: 15140960
[TBL] [Abstract][Full Text] [Related]
7. Dephosphorylation failure of tyrosine-phosphorylated STAT1 in IFN-stimulated Sendai virus C protein-expressing cells.
Saito S; Ogino T; Miyajima N; Kato A; Kohase M
Virology; 2002 Feb; 293(2):205-9. PubMed ID: 11886239
[TBL] [Abstract][Full Text] [Related]
8. Sendai virus wild-type and mutant C proteins show a direct correlation between L polymerase binding and inhibition of viral RNA synthesis.
Grogan CC; Moyer SA
Virology; 2001 Sep; 288(1):96-108. PubMed ID: 11543662
[TBL] [Abstract][Full Text] [Related]
9. The STAT2 activation process is a crucial target of Sendai virus C protein for the blockade of alpha interferon signaling.
Gotoh B; Takeuchi K; Komatsu T; Yokoo J
J Virol; 2003 Mar; 77(6):3360-70. PubMed ID: 12610111
[TBL] [Abstract][Full Text] [Related]
10. Structural Basis of the Inhibition of STAT1 Activity by Sendai Virus C Protein.
Oda K; Matoba Y; Irie T; Kawabata R; Fukushi M; Sugiyama M; Sakaguchi T
J Virol; 2015 Nov; 89(22):11487-99. PubMed ID: 26339056
[TBL] [Abstract][Full Text] [Related]
11. Inhibition of the gamma interferon response by a Sendai virus C protein mutant with no STAT1-binding ability.
Gotoh B; Takeuchi K; Komatsu T
FEBS Lett; 2004 Jun; 567(2-3):291-6. PubMed ID: 15178339
[TBL] [Abstract][Full Text] [Related]
12. C and V proteins of Sendai virus target signaling pathways leading to IRF-3 activation for the negative regulation of interferon-beta production.
Komatsu T; Takeuchi K; Yokoo J; Gotoh B
Virology; 2004 Jul; 325(1):137-48. PubMed ID: 15231393
[TBL] [Abstract][Full Text] [Related]
13. A short peptide at the amino terminus of the Sendai virus C protein acts as an independent element that induces STAT1 instability.
Garcin D; Marq JB; Iseni F; Martin S; Kolakofsky D
J Virol; 2004 Aug; 78(16):8799-811. PubMed ID: 15280488
[TBL] [Abstract][Full Text] [Related]
14. The amino-terminal extensions of the longer Sendai virus C proteins modulate pY701-Stat1 and bulk Stat1 levels independently of interferon signaling.
Garcin D; Marq JB; Goodbourn S; Kolakofsky D
J Virol; 2003 Feb; 77(4):2321-9. PubMed ID: 12551969
[TBL] [Abstract][Full Text] [Related]
15. Longer and shorter forms of Sendai virus C proteins play different roles in modulating the cellular antiviral response.
Garcin D; Curran J; Itoh M; Kolakofsky D
J Virol; 2001 Aug; 75(15):6800-7. PubMed ID: 11435558
[TBL] [Abstract][Full Text] [Related]
16. Inhibition of IFN-alpha/beta signaling by two discrete peptides within measles virus V protein that specifically bind STAT1 and STAT2.
Caignard G; Bouraï M; Jacob Y; ; Tangy F; Vidalain PO
Virology; 2009 Jan; 383(1):112-20. PubMed ID: 19007958
[TBL] [Abstract][Full Text] [Related]
17. High resistance of human parainfluenza type 2 virus protein-expressing cells to the antiviral and anti-cell proliferative activities of alpha/beta interferons: cysteine-rich V-specific domain is required for high resistance to the interferons.
Nishio M; Tsurudome M; Ito M; Kawano M; Komada H; Ito Y
J Virol; 2001 Oct; 75(19):9165-76. PubMed ID: 11533180
[TBL] [Abstract][Full Text] [Related]
18. Measles virus V protein blocks interferon (IFN)-alpha/beta but not IFN-gamma signaling by inhibiting STAT1 and STAT2 phosphorylation.
Takeuchi K; Kadota SI; Takeda M; Miyajima N; Nagata K
FEBS Lett; 2003 Jun; 545(2-3):177-82. PubMed ID: 12804771
[TBL] [Abstract][Full Text] [Related]
19. Prolactin activates Stat1 but does not antagonize Stat1 activation and growth inhibition by type I interferons in human breast cancer cells.
Schaber JD; Fang H; Xu J; Grimley PM; Rui H
Cancer Res; 1998 May; 58(9):1914-9. PubMed ID: 9581833
[TBL] [Abstract][Full Text] [Related]
20. Identification of paramyxovirus V protein residues essential for STAT protein degradation and promotion of virus replication.
Nishio M; Tsurudome M; Ito M; Garcin D; Kolakofsky D; Ito Y
J Virol; 2005 Jul; 79(13):8591-601. PubMed ID: 15956600
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]